Categories
Animal Hide, Shell & Others

Arca shell

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Other Names: Arca noae or the Noah’s Ark shell
Family: Arcidae
Genus: Arca
Species: A. noae
Kingdom: Animalia
Phylum: Mollusca
Class: Bivalvia
Order: Arcoida

Common Names: Cockle shell or Wa Leng Zi in mandarin,

Distribution & availablity: Arca noae or Ark shell is found in the Mediterranean and Adriatic Seas. It used to be common in the Adriatic but in 1949/50 there was a sudden unexplained, catastrophic decline in numbers. Since then populations have been creeping back upwards and in 2002, densities of up to 13 individuals per square metre (11 square feet) were recorded but, because of lack of records, it is unclear whether a return to prior population levels had been reached.
Description:
The shell of Arca noae grows to about 10 cm (4 in) in length. It is shortened at the anterior end and elongated posteriorly. It is irregularly striped in brown and white and has fine sculptured ribs running from the umbones to the margin. The hinge is long and straight and the shell is attached strongly to the substrate by byssal threads. There are pallial eyes on the edges of the mantle, especially at the posterior end. There are 42 to 48 radial ribs outside.

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They are characterized by boat-shaped shells with long, straight hinge lines bearing many small, interlocking teeth. The shells are usually coated with a thick, sometimes hairy periostracum (outer organic shell layer). Many of these clams have rows of simple eyes along the mantle margins. Most of the 200 or so known species are found in tropical seas, with only a few species occurring in temperate areas. Ark shells are slow-moving or sedentary.It lives shallowly buried in sands and silts.

Biology:
In the lower part of the intertidal zone in the Adriatic, Arca noae often grows in association with Modiolus barbatus.The shells are often heavily encrusted with epibionts. Water is drawn into the shell mainly at the posterior end. Plankton and fine organic particles are filtered out as the water passes over the gills and inedible particles are rejected at the same time. Its shell contains a large amount of calcium carbonate and a small amount of calcium phosphate. Besides, it also contains aluminum silicate and inorganic elements, such as chlorine, chromium, copper, iron, potassium, manganese, sodium, nickel, phosphorus, sulfur, silicon, strontium, and zinc. And it has a specific enrichment capacity of nuclide manganese.

Collection & Uses:
Arca noae is fished commercially in the Adriatic Sea, either by divers gathering individual shells by hand or from boats using specially adapted rakes which are pulled along the seabed. The shellfish are then sold in local markets. In China it is produced around the coastal areas and captured all year round for medicinal purpose. And the subsequent steps are to clean, slightly boil in boiling water, remove the meat, and finally dried in sun. Medicinally it is used broken and raw or calcined.

Medicinal Uses:
Modern pharmacology reveals that its ability of reducing gastric ulcer pain comes from calcium carbonate, which can neutralize stomach acid.

Ark shell, clam shells (Hai Ge Ke), and pumice (Fu Hai Shi) have quite similar medicinal uses in traditional Chinese medicine. However, it doesn’t mean that they are interchangeable. On the contrary, they need to be well distinguished clinically for proper uses.

All of the three are of salty in flavor and have the common function of eliminating phlegm, softening hardness and dissipating binds. As a result, all of them can be used to treat phlegm-fire stagnation induced scrofula, subcutaneous nodule, goiter and tumor; clam shell and pumice stone can also treat cough and asthma accompanied with thick yellow sputum that is caused by lung heat and phlegm-fire since both of them is capable of clearing lung and eliminating phlegm; clam shell and ark shell are also good at treating stomach discomfort and acid reflux since they can neutralize acidity and relieve pain.

And they do have their own advantage respectively on healing properties. Clam shell is still capable of inducing diuresis to alleviate edema. So it is often used for the treatment of edema and difficult urination; pumice stone can treat bloody stranguria and urolithiasis by inducing diuresis; ark shell removes blood stasis and disperses phlegm. Hence, it treats mass in the abdomen and hepatosplenomegaly.

Sample ark shell recipes on herbal remedies:   The Chinese Pharmacopoeia says that it is salty in flavor and neutral in nature. It covers meridians of lung, stomach, and liver. Crucial functions are dissolving phlegm, dispersing blood stasis, resolving hard lump, relieving hyperacidity, and stopping pain. Prime ark shell uses and indications include substantial amounts of lingering phlegm, difficulty coughing up thick, sticky mucus, goiter and tumor, scrofula, abdominal mass, stomachache, and acid regurgitation. Recommended dosage is from 9 to 15 grams in decoction. And please keep in mind to decoct it before other ingredients.

1) Han Hua Wan from Zheng Zhi Zhun Sheng (The Level-line of Patterns and Treatment). It is formulated with Hai Zao (Sargassum Seaweed), Kun Bu (Kombu), etc. to treat scrofula, goiter and tumor;

2) Wa Long Zi Wan from Wan Shi Jia Chao Fang (Wan’s Heirloom Prescriptions). It is fried, processed with vinegar, and used alone to cure abdominal mass and eliminate phlegm;

3) Wa Leng Zi Wan from Nu Ke Zhi Zhang (Full Knowledge of Gynecology). It is formulated with Xiang Fu (Cyperus), Tao Ren (Peach Seed), Mu Dan Pi (Tree Peony), Chuan Xiong (lovage), Da Huang (rhubarb), and Hong Hua (Safflower) to heal pain and no blood flow during menstruation. Lower abdomen is hard and full when pressed and it is kind of excess pain.

Clinical research of ark shells:  50 cases of burns and scalds, including second degree, have been treated with the combination of ark shell and vegetable oil at the ratio of 1:1. The oil was directly applied to the wounds and wall of them were cured. – Si Chuan Yi Xue (Sichuan Medicine), 1982; 1:44.

Ark shell side effects and contraindications:
Generally ark shell causes no adverse reaction when it is used in the treatments a variety of diseases like gastric and duodenal ulcers. But there were individual cases reported with facial swelling, blood in the urine, cloudy urine, recurrent urinary tract infections and others. Ben Cao Yong Fa Yan Jiu (Studies of The Uses of Drugs in Chinese Materia Medica) says that it shouldn’t be used in the patients with no blood stasis and sputum retention.

Disclaimer : The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplement, it is always advisable to consult with your own health care provider.
Resources:
http://en.wikipedia.org/wiki/Arca_noae
http://www.britannica.com/EBchecked/topic/34878/ark-shell

Ark Shell (Wa Leng Zi)

Categories
Ayurvedic

Siddha

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Introduction:
Siddha  is usually considered as the oldest medical system known to mankind.  Contemporary Tamil literature holds that the system of Siddha medicine originated in Southern India, in the state of Tamil Nadu. Siddha is reported to have surfaced more than 10,000 years ago.

“Siddhargal” or Siddhars were the premier scientists of ancient days.  Siddhars, mainly from Southern India laid the foundation for this system of medication. Siddhars were spiritual adepts who possessed the ashta siddhis, or the eight supernatural powers. Sage Agathiyar is considered the guru of all Sidhars, and the Siddha system is believed to have been handed over to him by Lord . “Agathiyar” was the first Siddhar,   and his disciples and Siddhars from other schools produced thousands of texts on Siddha, including medicine, and form the propounders of the system to the world.

The Siddha science is the oldest traditional treatment system generated from Dravidian culture. The Siddha flourished in the period of Indus Valley civilization.  Palm leaf manuscripts says that the Siddha system was first described by Lord Shiva to his wife Parvati. Parvati explained all this knowledge to her son Lord Muruga. He taught all these knowledge to his disciple sage Agasthya. Agasthya taught 18 Siddhars and they spread this knowledge to human beings.

The word Siddha comes from the Sanskrit word Siddhi which means an object to be attained perfection or heavenly bliss.  Siddha focused to “Ashtamahasiddhi,” the eight supernatural power. Those who attained or achieved the above said powers are known as Siddhars. There were 18 important Siddhars in olden days and they developed this system of medicine. Hence, it is called Siddha medicine. The Siddhars wrote their knowledge in palm leaf manuscripts, fragments of which were found in parts of South India. It is believed that some families may possess more fragments but keep them solely for their own use. There is a huge collection of Siddha manuscripts kept by traditional Siddha families.

Generally the basic concepts of the Siddha medicine are almost similar to Ayurveda. The only difference appears to be that the siddha medicine recognizes predominance of Vaadham, Pitham and Kabam in childhood, adulthood and old age, respectively, whereas in Ayurveda, it is totally reversed: Kabam is dominant in childhood, Vaatham in old age and Pitham in adults.

According to the Siddha medicine, various psychological and physiological functions of the body are attributed to the combination of seven elements: first is ooneer (plasma) responsible for growth, development and nourishment; second is cheneer (blood) responsible for nourishing muscles, imparting colour and improving intellect; the third is oon (muscle) responsible for shape of the body; fourth is koluppu/Kozhuppu (fatty tissue) responsible for oil balance and lubricating joints; fifth is elumbu (bone) responsible for body structure and posture and movement; sixth is elumbu majjai (bone marrow) responsible for formation of blood corpuscles; and the last is sukkilam (semen) responsible for reproduction. Like in Ayurveda, in Siddha medicine also, the physiological components of the human beings are classified as Vaadham (air), Pitham (fire) and Kabam(earth and water).

Concept of disease and cause:
It is assumed that when the normal equilibrium of the three humors — Vaadham, Pittham and Kabam — is disturbed, disease is caused. The factors assumed to affect this equilibrium are environment, climatic conditions, diet, physical activities, and stress. Under normal conditions, the ratio between Vaadham, Pittham, and Kabam are 4:2:1, respectively.

According to the Siddha medicine system, diet and lifestyle play a major role in health and in curing diseases. This concept of the Siddha medicine is termed as pathiyam and apathiyam, which is essentially a list of “do’s and don’ts”

Diagnosis:
In diagnosis, examination of eight items is required which is commonly known as “enn vakaith thervu”. These are:

1.Na (tongue): black in Vaatham, yellow or red in pitham, white in kabam, ulcerated in anaemia.
2.Varnam (colour): dark in Vaatham, yellow or red in pitham, pale in kabam.
3.Kural (voice): normal in Vaatham, high-pitched in pitham, low-pitched in kabam, slurred in alcoholism.
4.Kan (eyes): muddy conjunctiva, yellowish or red in pitham, pale in kabam.
5.Thodal (touch): dry in Vaatham, warm in pitham, chill in kapha, sweating in different parts of the body.
6.Malam (stool): black stools indicate Vaatham, yellow pitham, pale in kabam, dark red in ulcer and shiny in terminal illness.
7.Neer (urine): early morning urine is examined; straw color indicates indigestion, reddish-yellow color in excessive heat, rose in blood pressure, saffron color in jaundice, and looks like meat washed water in renal disease.
8.Naadi (pulse): the confirmatory method recorded on the radial art.

Drugs:
The drugs used by the Siddhars could be classified into three groups: thavaram (herbal product), thadhu (inorganic substances) and jangamam (animal products). The Thadhu drugs are further classified as: uppu (water-soluble inorganic substances or drugs that give out vapour when put into fire), pashanam (drugs not dissolved in water but emit vapour when fired), uparasam (similar to pashanam but differ in action), loham (not dissolved in water but melt when fired), rasam (drugs which are soft), and ghandhagam (drugs which are insoluble in water, like sulphur).

The drugs used in siddha medicine were classified on the basis of five properties: suvai (taste), gunam (character), veeryam (potency), pirivu (class) and mahimai (action).

According to their mode of application, the siddha medicines could be categorized into two classes:

Internal medicine was used through the oral route and further classified into 32 categories based on their form, methods of preparation, shelf-life, etc.
External medicine includes certain forms of drugs and also certain applications (such as nasal, eye and ear drops), and also certain procedures (such as leech application). It also classified into 32 categories.

Treatment:
The treatment in siddha medicine is aimed at keeping the three humors in equilibrium and maintenance of seven elements. So proper diet, medicine and a disciplined regimen of life are advised for a healthy living and to restore equilibrium of humors in diseased condition. Saint Thiruvalluvar explains four requisites of successful treatment. These are the patient, the attendant, physician and medicine. When the physician is well-qualified and the other agents possess the necessary qualities, even severe diseases can be cured easily, according to these concepts.

The treatment should be commenced as early as possible after assessing the course and cause of the disease. Treatment is classified into three categories: devamaruthuvum (Divine method); manuda maruthuvum (rational method); and asura maruthuvum (surgical method). In Divine method, medicines like parpam, Chendooragyhtyjm, guru, kuligai made of mercury, sulfur and pashanams are used. In the rational method, medicines made of herbs like churanam, kudineer, or vadagam are used. In surgical method, incision, excision, heat application, blood letting, or leech application are used.

According to therapies the treatments of siddha medicines could be further categorized into following categories such as purgative therapy, emetic therapy, fasting therapy, steam therapy, oleation therapy, physical therapy, solar therapy, blood-letting therapy, yoga therapy, etc.

Sidda education:
Siddha has lost its popularity after modern medicine was introduced, as a scientific medical system, even in Tamil Nadu. Still, there are a few ardent followers of the system who prefer Siddha for only a few diseases like jaundice. After some modern doctors, such as Dr. Ramalingam, IMPCOPS, president, Chennai, C.N. Deivanayagam, tried to popularize the Siddha system, a few modern doctors have started suggesting Siddha. In 2012, VA Shiva Ayyadurai, a Tamilian and MIT systems scientist, launched an educational program for medical doctors through the Chopra Center with Deepak Chopra which integrates concepts from traditional systems medicine such as Siddha, Ayurveda, and traditional Chinese medicine, with systems science and systems biology.

The Tamil Nadu state runs a 5.5-year course in Siddha medicine (BSMS: Bachelor in Siddha Medicine and Surgery). The Indian Government also gives its focus on Siddha, by starting up medical colleges and research centers like National Institute of Siddha  and Central Council for Research in Siddha. There has been renewed interest in Siddha, as many started feeling modern medicine is not complete and changing its stands/theories frequently. The health minister of Tamil Nadu in 2007 claimed that Siddha medicine is effective for chikungunya

Educational institutions:
Government of Tamil Nadu runs two Siddha medical colleges:

Government Siddha Medical College, Palayamkottai, Tirunelveli district
Government Siddha Medical College, Anna Hospital Campus, Arumbakkam, Chennai – 600106

.
Government of India runs a Siddha medical college:

National Institute of Siddha, Grand Southern Trunk Road, Tambaram Sanatorium, Chennai – 600047

.
Colleges available in Kerala:

*Santhigiri Siddha Medical College, Thiruvananthapuram
*Private Siddha colleges (approved by Dept. of AYUSH, Govt. of India and affiliated to TN Dr. MGR Medical University, Chennai):

*Velumailu Siddha Medical College and Hospital, No. 48, G.W.T. Road, Opp. Rajiv Gandhi Memorial, Sriperumbudur – 602 105
*Sri Sai Ram Siddha Medical College & Research Centre, Sai Leo Nagar, Poonthandalam, West Tambaram, Chennai – 600 044
*R.V.S. Siddha Medical College & Hospital, Kumaran Kottam, Kannampalayam, Coimbatore – 641042
*A.T.S.V.S. Siddha Medical College, Munchirai, Pudukkadai Post, Kanyakumari – 629171
*Sivaraj Siddha Medical College, Siddhar Kovil Road, Thumbathulipatty, Salem – 636307

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Government of Sri Lanka runs three… Siddha medical colleges offering BSMS degrees:

*Department of Siddha Medicine, University of Jaffna, Kaithady, Jaffna, Sri Lanka
*Unit of Siddha Medicine, Trincomalee Campus, Eastern University, Trincomalee, Sri Lanka

Resources: http://en.wikipedia.org/wiki/Siddha_medicine

Categories
Herbs & Plants

Rosemary

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Botanical Name: Rosmarinus officinalis
Family: Lamiaceae
Genus: Rosmarinus
Species: R. officinalis
Kingdom: Plantae
Order: Lamiales

Common Name: Rosemary

Habitat: Rosemary is native to Mediterranean region. Now it is growing in most places of the world.
Description:
Rosemary, is a woody, perennial herb with fragrant, evergreen, needle-like leaves and white, pink, purple, or blue flowers, native to the Mediterranean region. It is a member of the mint family Lamiaceae, which includes many other herbs. The name “rosemary” derives from the Latin for “dew” (ros) and “sea” (marinus), or “dew of the sea”. The plant is also sometimes called anthos, from the ancient Greek world, meaning “flower”. Rosemary has a fibrous root system.

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Rosemary leaves are similar to hemlock needles. The leaves are used as a flavoring in foods such as stuffings and roast lamb, pork, chicken and turkey. It is native to the Mediterranean and Asia, but is reasonably hardy in cool climates. It can withstand droughts, surviving a severe lack of water for lengthy periods. Forms range from upright to trailing; the upright forms can reach 1.5 m (5 ft) tall, rarely 2 m (6 ft 7 in). The leaves are evergreen, 2–4 cm (0.8–1.6 in) long and 2–5 mm broad, green above, and white below, with dense, short, woolly hair. The plant flowers in spring and summer in temperate climates, but the plants can be in constant bloom in warm climates; flowers are white, pink, purple or deep blue. Rosemary also has a tendency to flower outside its normal flowering season; it has been known to flower as late as early December, and as early as mid-February.

Cultivation:
Since it is attractive and drought-tolerant, rosemary is used as an ornamental plant in gardens and for xeriscape landscaping, especially in regions of Mediterranean climate. It is considered easy to grow and pest-resistant. Rosemary can grow quite large and retain attractiveness for many years, can be pruned into formal shapes and low hedges, and has been used for topiary. It is easily grown in pots. The groundcover cultivars spread widely, with a dense and durable texture.

Rosemary grows on friable loam soil with good drainage in an open, sunny position. It will not withstand waterlogging and some varieties are susceptible to frost. It grows best in neutral to alkaline conditions (pH 7–7.8) with average fertility. It can be propagated from an existing plant by clipping a shoot (from a soft new growth) 10–15 cm (4–6 in) long, stripping a few leaves from the bottom, and planting it directly into soil

Edible Uses:
The leaves are used to flavor various foods, such as stuffings and roast meats. Fresh or dried leaves are used in traditional Italian cuisine. They have a bitter, astringent taste and a characteristic aroma which complements many cooked foods. Herbal tea can be made from the leaves. When roasted with meats or vegetables, the leaves impart a mustard-like aroma with an additional fragrance of charred wood compatible with barbecued foods.

In amounts typically used to flavor foods, such as one teaspoon (1 gram), rosemary provides no nutritional value. Rosemary extract has been shown to improve the shelf life and heat stability of omega 3-rich oils which are prone to rancidity.

Medicinal Uses:
Rosemary contains substances that are useful for stimulating the immune system, increasing circulation, and improving digestion. Rosemary also contains anti-inflammatory compounds that may make it useful for reducing the severity of asthma attacks. In addition, rosemary has been shown to increase the blood flow to the head and brain, improving concentration.

Phytochemicals and traditional medicine:
Rosemary contains a number of phytochemicals, including rosmarinic acid, camphor, caffeic acid, ursolic acid, betulinic acid, and the antioxidants carnosic acid and carnosol.

In traditional medicine of India, extracts and essential oil from flowers and leaves are used to treat a variety of disorders.

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Other Uses:
Rosemary is used as a decorative plant in gardens where it may have pest control effects.

Folklore and customs:
In the Middle Ages, rosemary was associated with wedding ceremonies. The bride would wear a rosemary headpiece and the groom and wedding guests would all wear a sprig of rosemary. From this association with weddings, rosemary was thought to be a love charm.

In myths, rosemary has a reputation for improving memory and has been used as a symbol for remembrance during war commemorations and funerals in Europe and Australia. Mourners would throw it into graves as a symbol of remembrance for the dead. In Shakespeare’s Hamlet, Ophelia says, “There’s rosemary, that’s for remembrance.” (Hamlet, iv. 5.) In Australia, sprigs of rosemary are worn on ANZAC Day and sometimes Remembrance Day to signify remembrance; the herb grows wild on the Gallipoli Peninsula.

Hungary water was first prepared for the Queen of Hungary Elisabeth of Poland to ” … renovate vitality of paralyzed limbs … ” and to treat gout. It was used externally and prepared by mixing fresh rosemary tops into spirits of wine. Don Quixote (Part One, Chapter XVII) mixes it in his recipe of the miraculous balm of Fierabras.

Mythology:
According to legend, it was draped around the Greek goddess Aphrodite when she rose from the sea, born of Uranus’s semen. The Virgin Mary is said to have spread her blue cloak over a white-blossomed rosemary bush when she was resting, and the flowers turned blue. The shrub then became known as the “Rose of Mary”

Known hazards:
The essential oil of rosemary is potent and should be avoided by pregnant and breastfeeding women. The oil may cause severe adverse effects including seizures when taken internally and may irritate the skin when applied externally.
Disclaimer : The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplement, it is always advisable to consult with your own health care provider.

Resources:
http://en.wikipedia.org/wiki/Rosemary
http://www.whfoods.com/genpage.php?tname=foodspice&dbid=75
http://www.kew.org/science-conservation/plants-fungi/rosmarinus-officinalis-rosemary

Categories
Herbs & Plants

Thaumatococcus daniellii

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Botanical Name: Thaumatococcus daniellii
Family:    Marantaceae
Genus:    Thaumatococcus
Kingdom:    Plantae
Order: Zingiberales

Synonyms:  Phrynium daniellii

Common names: Miracle fruit (but the unrelated species Synsepalum dulcificum is better known by that name) and miracle berry, Katamfe or Katempfe, Yoruba soft cane, and African serendipity berry.

Habitat: Thaumatococcus daniellii  is  native to the rainforests of western Africa from Sierra Leone to Zaire. It is also an introduced species in Australia and Singapore.

Description:
Thaumatococcus daniellii is a rhizomatous, perennial herb, up to 3-3.5 m high.  It  has large, papery leaves up to 46 centimeters long and 40 cm wide, arise singly from each node of the rhizome. Inflorescences are single or simply branched spikes’ and emerge from the lowest node.  It bears pale purple flowers and a soft fruit containing a few shiny black seeds. In its native range,the fruit is fleshy, trigonal in shape and matures to a dark red/brown colour when fully ripe. At maturity each fruit contains three black, extremely hard seeds. The seeds are enveloped by a sticky thin, pale yellow basal aril, which contains the sweetening protein, thaumatin.
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Varieties:
Thaumatococcus daniellii var. daniellii – western + central Africa from Sierra Leone to Zaire
Thaumatococcus daniellii var. puberulifolius Dhetchuvi & Diafouka – central Africa (Zaire, Gabon, Congo-Brazzaville, Cameroon, Central African Republic)

Edible Uses:
Fruit: The most popular use of T. daniellii is as sweetener. The aril contains a non-toxic, intensely sweet protein named thaumatin, which is at least 3000 times as sweet as sucrose. In West Africa, the aril is traditionally used for sweetening bread, over-fermented palm-wine and sour food. When the seeds are chewed, for up to an hour afterwards they cause sour materials eaten or drunk to taste very sweet. Since the mid-1990s, thaumatin is used as sweetener and flavour enhancer by the food and confectionary industry. Substituting synthetic sweeteners, it is used as a non-caloric natural sweetener. Thaumatin is not a carbohydrate thus it is an ideal sweetener for diabetics.

The seeds of T. daniellii also produce a jelly that swells to 10 times its own weight and hence provides a substitute for agar.

Medicinal Uses:
Thaumatococcus  daniellii is also used in traditional medicinal uses in the Ivory Coast and Congo. The fruit is used as a laxative and the seed as an emetic and for pulmonary problems.
In traditional medicinal use the leaf sap is used as antidote against venoms, stings and bites. Leaf and root sap are used as sedative and for treating insanity.

Other Uses:
In West Africa, T. daniellii is mostly cultivated for the leaves. The lamina of the leaves is used for wrapping foods. The petiole is used to weave mats and as tools and building materials. The entire leaf is also used for roofing.
Disclaimer : The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplement, it is always advisable to consult with your own health care provider.

Resources:
http://en.wikipedia.org/wiki/Thaumatococcus_daniellii

Categories
Health Alert Health Problems & Solutions

Glycaemic Index

Definition:
The glycemic index or glycaemic index (GI) is a number associated with a particular type of food that indicates the food’s effect on a person’s blood glucose (also called blood sugar) level. The number typically ranges between 50 and 100, where 100 represents the standard, an equivalent amount of pure glucose.

The GI represents the total rise in a person’s blood sugar level following consumption of the food; it may or may not represent the rapidity of the rise in blood sugar. The steepness of the rise can be influenced by a number of other factors, such as the quantity of fat eaten with the food. The GI is useful for understanding how the body breaks down carbohydrates  and only takes into account the available carbohydrate (total carbohydrate minus fiber) in a food. Although the food may contain fats and other components that contribute to the total rise in blood sugar, these effects are not reflected in the GI.

The glycemic index is usually applied in the context of the quantity of the food and the amount of carbohydrate in the food that is actually consumed. A related measure, the glycemic load (GL), factors this in by multiplying the glycemic index of the food in question by the carbohydrate content of the actual serving. Watermelon has a high glycemic index, but a low glycemic load for the quantity typically consumed. Fructose, by contrast, has a low glycemic index, but can have a high glycemic load if a large quantity is consumed.

GI tables are available that list many types of foods and their GIs. Some tables also include the serving size and the glycemic load of the food per serving.

A practical limitation of the glycemic index is that it does not measure insulin production due to rises in blood sugar. As a result, two foods could have the same glycemic index, but produce different amounts of insulin. Likewise, two foods could have the same glycemic load, but cause different insulin responses. Furthermore, both the glycemic index and glycemic load measurements are defined by the carbohydrate content of food. For example when eating steak, which has no carbohydrate content but provides a high protein intake, up to 50% of that protein can be converted to glucose when there is little to no carbohydrate consumed with it.  But because it contains no carbohydrate itself, steak cannot have a glycemic index. For some food comparisons, the “insulin index” may be more useful.

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Glycemic index charts often give only one value per food, but variations are possible due to variety, ripeness (riper fruits contain more sugars increasing GI), cooking methods (the more cooked, or over cooked, a food the more its cellular structure is broken with a tendency for it to digest quickly and raise GI more), processing (e.g., flour has a higher GI than the whole grain from which it is ground as grinding breaks the grain’s protective layers) and the length of storage. Potatoes are a notable example, ranging from moderate to very high GI even within the same variety.

The glycemic response is different from one person to another, and also in the same person from day to day, depending on blood glucose levels, insulin resistance, and other factors.

Most of the values on the glycemic index do not show the impact on glucose levels after two hours. Some people with diabetes may have elevated levels after four hours.

Why  GI is so Important?
Over the past 15 years, low-GI diets have been associated with decreased risk of cardiovascular disease, type 2 diabetes, metabolic syndrome, stroke, depression, chronic kidney disease, formation of gall stones, neural tube defects, formation of uterine fibroids, and cancers of the breast, colon, prostate, and pancreas. Taking advantage of these potential health benefits can be as simple as sticking with whole, natural foods that are either low or very low in their GI value.

Determination of GI of a food:
Foods with carbohydrates that break down quickly during digestion and release glucose rapidly into the bloodstream tend to have a high GI; foods with carbohydrates that break down more slowly, releasing glucose more gradually into the bloodstream, tend to have a low GI. The concept was developed by Dr. David J. Jenkins and colleagues  in 1980–1981 at the University of Toronto in their research to find out which foods were best for people with diabetes. A lower glycemic index suggests slower rates of digestion and absorption of the foods’ carbohydrates and may also indicate greater extraction from the liver and periphery of the products of carbohydrate digestion. A lower glycemic response usually equates to a lower insulin demand but not always, and may improve long-term blood glucose control   and blood lipids. The insulin index is also useful for providing a direct measure of the insulin response to a food.

The glycemic index of a food is defined as the incremental area under the two-hour blood glucose response curve (AUC) following a 12-hour fast and ingestion of a food with a certain quantity of available carbohydrate (usually 50 g). The AUC of the test food is divided by the AUC of the standard (either glucose or white bread, giving two different definitions) and multiplied by 100. The average GI value is calculated from data collected in 10 human subjects. Both the standard and test food must contain an equal amount of available carbohydrate. The result gives a relative ranking for each tested food.

The current validated methods use glucose as the reference food, giving it a glycemic index value of 100 by definition. This has the advantages of being universal and producing maximum GI values of approximately 100. White bread can also be used as a reference food, giving a different set of GI values (if white bread = 100, then glucose ? 140). For people whose staple carbohydrate source is white bread, this has the advantage of conveying directly whether replacement of the dietary staple with a different food would result in faster or slower blood glucose response. A disadvantage with this system is that the reference food is not well-defined.

Classification:
GI values can be interpreted intuitively as percentages on an absolute scale and are commonly interpreted as follows:

Low GI…..(55 or less fructose;) …….Examples:beans (white, black, pink, kidney, lentil, soy, almond, peanut, walnut, chickpea); small seeds (sunflower, flax, pumpkin, poppy, sesame, hemp); most whole intact grains (durum/spelt/kamut wheat, millet, oat, rye, rice, barley); most vegetables, most sweet fruits (peaches, strawberries, mangos); tagatose; mushrooms; chilis.

Medium GI…..(56–69 Examples: white sugar or sucrose, not intact whole wheat or enriched wheat, pita bread, basmati rice, unpeeled boiled potato, grape juice, raisins, prunes, pumpernickel bread, cranberry juice,[10] regular ice cream, banana.

High GI….….(70 and above) Examples: glucose (dextrose, grape sugar), high fructose corn syrup, white bread (only wheat endosperm), most white rice (only rice endosperm), corn flakes, extruded breakfast cereals, maltose, maltodextrins, sweet potato , white potato , pretzels, bagels.

A low-GI food will release glucose more slowly and steadily, which leads to more suitable postprandial (after meal) blood glucose readings. A high-GI food causes a more rapid rise in blood glucose levels and is suitable for energy recovery after exercise or for a person experiencing hypoglycemia.

The glycemic effect of foods depends on a number of factors, such as the type of starch (amylose versus amylopectin), physical entrapment of the starch molecules within the food, fat and protein content of the food and organic acids or their salts in the meal — adding vinegar, for example, will lower the GI. The presence of fat or soluble dietary fiber can slow the gastric emptying rate, thus lowering the GI. In general, coarse, grainy breads with higher amounts of fiber have a lower GI value than white breads.  However, most breads made with 100% whole wheat or wholemeal flour have a GI not very different from endosperm only (white) bread.  Many brown breads are treated with enzymes to soften the crust, which makes the starch more accessible (high GI).

While adding fat or protein will lower the glycemic response to a meal, the relative differences remain. That is, with or without additions, there is still a higher blood glucose curve after a high-GI bread than after a low-GI bread such as pumpernickel.

Fruits and vegetables tend to have a low glycemic index. The glycemic index can be applied only to foods where the test relies on subjects consuming an amount of food containing 50 g of available carbohydrate.[citation needed] But many fruits and vegetables (not potatoes, sweet potatoes, corn) contain less than 50 g of available carbohydrate per typical serving. Carrots were originally and incorrectly reported as having a high GI.  Alcoholic beverages have been reported to have low GI values; however, beer was initially reported to have a moderate GI due to the presence of maltose. This has been refuted by brewing industry professionals, who say that all maltose sugar is consumed in the brewing process and that packaged beer has little to no maltose present. Recent studies have shown that the consumption of an alcoholic drink prior to a meal reduces the GI of the meal by approximately 15%.  Moderate alcohol consumption more than 12 hours prior to a test does not affect the GI.

Many modern diets rely on the glycemic index, including the South Beach Diet, Transitions by Market America and NutriSystem Nourish Diet. However, others have pointed out that foods generally considered to be unhealthy can have a low glycemic index, for instance, chocolate cake (GI 38), ice cream (37), or pure fructose (19), whereas foods like potatoes and rice have GIs around 100 but are commonly eaten in some countries with low rates of diabetes.

The GI Symbol Program is an independent worldwide GI certification program that helps consumers identify low-GI foods and drinks. The symbol is only on foods or beverages that have had their GI values tested according to standard and meet the GI Foundation’s certification criteria as a healthy choice within their food group, so they are also lower in kilojoules, fat and/or salt.

Weight control:
Recent animal research provides compelling evidence that high-GI carbohydrate is associated with increased risk of obesity. In one study,  male rats were split into high- and low-GI groups over 18 weeks while mean body weight was maintained. Rats fed the high-GI diet were 71% fatter and had 8% less lean body mass than the low-GI group. Postmeal glycemia and insulin levels were significantly higher, and plasma triglycerides were threefold greater in the high-GI-fed rats. Furthermore, pancreatic islet cells suffered “severely disorganized architecture and extensive fibrosis.” However, the GI of these diets was not experimentally determined. In a well controlled feeding study no improvement in weight loss was observed with a low glycemic index diet over calorie restriction.  Because high-amylose cornstarch (the major component of the assumed low-GI diet) contains large amounts of resistant starch, which is not digested and absorbed as glucose, the lower glycemic response and possibly the beneficial effects can be attributed to lower energy density and fermentation products of the resistant starch, rather than the GI.

In humans, a 2012 study shows that, after weight loss, the energy expenditure is higher on a low-glycemic index diet than on a low-fat diet (but lower than on the Atkins diet).

 Prevention of Diseases:
Several lines of recent [1999] scientific evidence have shown that individuals who followed a low-GI diet over many years were at a significantly lower risk for developing both type 2 diabetes, coronary heart disease, and age-related macular degeneration than others.  High blood glucose levels or repeated glycemic “spikes” following a meal may promote these diseases by increasing systemic glycative stress, other oxidative stress to the vasculature, and also by the direct increase in insulin levels.  The glycative stress sets up a vicious cycle of systemic protein glycation, compromised protein editing capacity involving the ubiquitin proteolytic pathway and autophagic pathways, leading to enhanced accumulation of glycated and other obsolete proteins.

In the past, postprandial hyperglycemia has been considered a risk factor associated mainly with diabetes. However, more recent evidence shows that it also presents an increased risk for atherosclerosis in the non-diabetic population   and that high GI diets,  high blood-sugar levels more generally,  and diabetes  are related to kidney disease as well.

Conversely, there are areas such as Peru and Asia where people eat high-glycemic index foods such as potatoes and high-GI rice without a high level of obesity or diabetes.  The high consumption of legumes in South America and fresh fruit and vegetables in Asia likely lowers the glycemic effect in these individuals. The mixing of high- and low-GI carbohydrates produces moderate GI values.

A study from the University of Sydney in Australia suggests that having a breakfast of white bread and sugar-rich cereals, over time, may make a person susceptible to diabetes, heart disease, and even cancer.

A study published in the American Journal of Clinical Nutrition found that age-related adult macular degeneration (AMD), which leads to blindness, is 42% higher among people with a high-GI diet, and concluded that eating a lower-GI diet would eliminate 20% of AMD cases.

The American Diabetes Association supports glycemic index but warns that the total amount of carbohydrate in the food is still the strongest and most important indicator, and that everyone should make their own custom method that works best for them.

The International Life Sciences Institute concluded in 2011 that because there are many different ways of lowering glycemic response, not all of which have the same effects on health, “It is becoming evident that modifying the glycemic response of the diet should not be seen as a stand-alone strategy but rather as an element of an overall balanced diet and lifestyle.”

A systematic review of few human trials examined the potential of low GI diet to improve pregnancy outcomes. Potential benefits were still seen despite no ground breaking findings in maternal glycemia or pregnancy outcomes. In this regard, more women under low GI diet achieved the target treatment goal for the postprandial glycemic level and reduced their need for insulin treatment. A low GI diet may also provide greater benefits to overweight and obese women. Interestingly, intervention at an early stage of pregnancy has shown a tendency to lower birth weight and birth centile in infants born to women with GDM.

Other factors:
The number of grams of carbohydrate can have a bigger impact than glycemic index on blood sugar levels, depending on quantities. Consuming fewer calories, losing weight, and carbohydrate counting can be better for lowering the blood sugar level. Carbohydrates impact glucose levels most profoundly,  and two foods with the same carbohydrate content are, in general, comparable in their effects on blood sugar.  A food with a low glycemic index may have a high carbohydrate content or vice versa; this can be accounted for with the glycemic load (GL). Consuming carbohydrates with a low glycemic index and calculating carbohydrate intake would produce the most stable blood sugar levels.

Criticism and alternatives:
The glycemic index does not take into account other factors besides glycemic response, such as insulin response, which is measured by the insulin index and can be more appropriate in representing the effects from some food contents other than carbohydrates. In particular, since it is based on the area under the curve of the glucose response over time from ingesting a subject food, the shape of the curve has no bearing on the corresponding GI value. The glucose response can rise to a high level and fall quickly, or rise less high but remain there for a longer time, and have the same area under the curve. For subjects with type 1 diabetes who do not have an insulin response, the rate of appearance of glucose after ingestion represents the absorption of the food itself. This glycemic response has been modeled, where the model parameters for the food enable prediction of the continuous effect of the food over time on glucose values, and not merely the ultimate effect that the GI represents.

Although the glycemic index provides some insights into the relative diabetic risk within specific food groups, it contains many counter-intuitive ratings. These include suggestions that bread generally has a higher glycemic ranking than sugar and that some potatoes are more glycemic than glucose. More significantly, studies such as that by Bazzano et al.  demonstrate a significant beneficial diabetic effect for fruit compared to a substantial detrimental impact for fruit juice despite these having similar “low GI” ratings.

From blood glucose curves presented by Brand-Miller et al.  the main distinguishing feature between average fruit and fruit juice blood glucose curves is the maximum slope of the leading edge of 4.38 mmol·L-1·h-1 for fruit and 6.71 mmol·L-1·h-1 for fruit juice. This raises the concept that the rate of increase in blood glucose may be a significant determinant particularly when comparing liquids to solids which release carbohydrates over time and therefore have an inherently greater area under the blood glucose curve.

If you were to restrict yourself to eating only low GI foods, your diet is likely to be unbalanced and may be high in fat and calories, leading to weight gain and increasing your risk of heart disease. It is important not to focus exclusively on GI and to think about the balance of your meals, which should be low in fat, salt and sugar and contain plenty of fruit and vegetables.

There are books that give a long list of GI values for many different foods. This kind of list does have its limitations. The GI value relates to the food eaten on its own and in practice we usually eat foods in combination as meals. Bread, for example is usually eaten with butter or margarine, and potatoes could be eaten with meat and vegetables.

An additional problem is that GI compares the glycaemic effect of an amount of food containing 50g of carbohydrate but in real life we eat different amounts of food containing different amounts of carbohydrate.

Note: The amount of carbohydrate you eat has a bigger effect on blood glucose levels than GI alone.

How to have lower GI?
*Choose basmati or easy cook rice, pasta or noodles.
*Switch baked or mashed potato for sweet potato or boiled new potatoes.
*Instead of white and wholemeal bread, choose granary, pumpernickel or rye bread.
*Swap frozen microwaveable French fries for pasta or noodles.
*Try porridge, natural muesli or wholegrain breakfast cereals.
*You can maximise the benefit of GI by switching to a low GI option food with each meal or snack

Resources:
http://en.wikipedia.org/wiki/Glycemic_index
https://www.diabetes.org.uk/Guide-to-diabetes/Managing-your-diabetes/Glycaemic-Index-GI/
http://www.whfoods.com/genpage.php?tname=faq&dbid=32

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