Categories
Herbs & Plants

Bear’s Breeches(Acanthus mollis – L.)

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Botanical Name:Acanthus mollis – L.
Family : Acanthaceae
Synonyms: Acanthus latifolius – Hort. ex Goeze.
Common Name :Bear’s Breeches,
Genus: Acanthus
Kingdom: Plantae
Order: Lamiales
Family: Acanthaceae
Species: A. mollis

Habitat : South-western Europe – Portugal to the Balkans. Naturalized in Britain in W. Cornwall.  Woodland scrub and stony hillsides.Woodland Garden; Sunny Edge; Dappled Shade; Ground Cover; Meadow;

Description:
It is a herbaceous perennial plant .It grows to 2 m tall, with basal clusters of deeply lobed and cut, shining dark green leaves up to 1 m long and 20 cm broad. The flowers are tubular, whitish, lilac or rose with spiny green or purplish bracts, and produced on stout spikes which grow up to 2.5 m (8 ft) above the leaves. It flowers in late spring or early summer. It grows in dry areas, and is tolerant of drought and shade. The plants are propagated from tubers and tend to form large, localized clumps which can survive for several decades. The leaves of this plant are generally considered by historians[who?] to have been the design inspiration for the Corinthian column capitals of Roman architecture.
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It is hardy to zone 0. It is in flower from June to August. The flowers are hermaphrodite (have both male and female organs) and are pollinated by Bees.

Cultivation :
Prefers a deep loamy soil in a sheltered position in full sun but tolerates partial shade. Grows well in heavy clay soils if they are well-drained but dislikes heavy damp soils and will not overwinter in wet soils. Established plants are fairly drought tolerant. Hardy to about -15°c, though young plants may require protection in the winter and even older ones may need protection in cold winters. A very ornamental plant. The leaves can wilt on hot summer days when plants are grown in full sun. Plants can become invasive, spreading by suckers, and they are difficult to eradicate due to their deep roots. Does well in the lawn or wild garden. Plants can be grown in quite coarse grass, which can be cut in the autumn. Members of this genus are not usually browsed by deer.

Propagation:-
Seed – sow spring in a cold frame or outside as soon as the seed is ripe. It usually germinates in 3 – 4 weeks at 10°c. When they are large enough to handle, prick the seedlings out into individual pots and grow them on for two years before planting out in late spring or early summer. Division in spring or autumn. Very easy, they can be planted straight out into their permanent positions. Root cuttings – winter in a coldframe

Medicinal Actions & Uses:

Astringent; Detergent; Emollient; Vulnerary.
The leaves and roots are astringent, detergent, emollient and vulnerary. The plant contains appreciable quantities of mucilage and tannin. Traditionally it was used as a treatment for dislocated joints and for burns. A paste made from the plant, when applied to a dislocated joint, tends to normalize the affected muscles and ligaments, simultaneously relaxing and tightening them to encourage the joint back into its proper place. The crushed leaves have been used as a poultice to soothe burns and scalds. For internal use, the plant’s emollient properties are useful in treating irritated mucous membranes within the digestive and urinary tracts.

Disclaimer:The information presented herein ,  is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

Other Uses:-
Ground cover.

The sub-species A. mollis latifolia makes a good ground cover plant. Relatively slow to cover the ground at first but it can eventually become invasive.


Source:

http://www.pfaf.org/database/plants.php?Acanthus+mollis
http://www.floradecanarias.com/acanthus_mollis.html
http://en.wikipedia.org/wiki/Acanthus_mollis

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Categories
Diagnonistic Test

TB (Tuberculosis) Skin Test Or Mantoux Test

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Introduction :
The Mantoux skin test consists of an intradermal injection of exactly one tenth of a milliliter (mL) of PPD tuberculin. The size of induration is measured 48–72 hours later. Erythema (redness) should not be measured.The Mantoux test (also known as the Mantoux screening test, Tuberculin Sensitivity Test, Pirquet test, or PPD test for Purified Protein Derivative) is a diagnostic tool for tuberculosis. It is one of the two major tuberculin skin tests used in the world, largely replacing multiple-puncture tests such as the Tine test. Until 2005, the Heaf test was used in the United Kingdom, but the Mantoux test is now used. The Mantoux test is also used in Australia, Canada, Hungary, The Netherlands, Portugal, South Africa and the United States and is endorsed by the American Thoracic Society and Centers for Disease Control and Prevention (CDC). It was also used in the USSR and is now prevalent in most of the former Soviet states

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The Mantoux skin test consists of an intradermal injection of exactly one tenth of a milliliter (mL) of PPD tuberculin.

 

The size of induration is measured 48–72 hours later. Erythema (redness) should not be measured.
.Tuberculosis is a bacterial infection that most often involves the lungs, but can involve many other organs. Although antibiotics can treat most cases, TB remains one of the most common causes of death worldwide. The TB skin test, also called the purified protein derivative (PPD) test or Mantoux test, shows if you’ve ever been infected with the bacteria that cause tuberculosis. Infections with these bacteria can be active or inactive. In active infections, the bacteria are reproducing rapidly, and the person is contagious when he or she coughs. In people with inactive infections, the bacteria are alive deep within the lungs, but “asleep.” Because inactive infections can later “wake up” and become active, it is important to recognize and treat both types of TB infections.

History:
Tuberculin is a glycerine extract of the tubercule bacilli. Purified protein derivative (PPD) tuberculin is a precipitate of non-species-specific molecules obtained from filtrates of sterilized, concentrated cultures. It was first described by Robert Koch in 1890. The test is named after Charles Mantoux, a French physician who developed on the work of Koch and Clemens von Pirquet to create his test in 1907.

In 1939, M. A. Linnikova in the USSR created a modified version of PPD. In 1954, the Soviet Union started mass production of PPD-L, named after Linnikova.

Procedure:
A standard dose of 5 Tuberculin units (0.1 mL) (The standard Mantoux test in the UK consists of an intradermal injection of 2TU of Statens Serum Institute (SSI) tuberculin RT23 in 0.1ml solution for injection.) is injected intradermally (between the layers of dermis) and read 48 to 72 hours later. A person who has been exposed to the bacteria is expected to mount an immune response in the skin containing the bacterial proteins.

The reaction is read by measuring the diameter of induration (palpable raised hardened area) across the forearm (perpendicular to the long axis) in millimeters. If there is no induration, the result should be recorded as “0 mm”. Erythema (redness) should not be measured.

If a person has had a history of a positive tuberculin skin test, another skin test is not needed

How do you prepare for the test?
Because vaccinations and steroids can affect the results of the test, tell your doctor if you’ve recently been vaccinated for an infectious disease or if you’re taking a steroid medication.

Classification of tuberculin reaction:
The results of this test must be interpreted carefully. The person’s medical risk factors determine at which increment (5 mm, 10 mm, or 15 mm) of induration the result is considered positive.  A positive result indicates TB exposure.

*5 mm or more is positive in

*HIV-positive person

*Recent contacts of TB case

*Persons with nodular or fibrotic changes on chest x-ray consistent with old healed TB

*Patients with organ transplants and other immunosuppressed patients

*10 mm or more is positive in

*Recent arrivals (less than 5 years) from high-prevalence countries

*Injection drug users

*Residents and employees of high-risk congregate settings (e.g., prisons, nursing homes, hospitals, homeless shelters, etc.)

*Mycobacteriology lab personnel

*Persons with clinical conditions that place them at high risk (e.g., diabetes, prolonged corticosteroid therapy, leukemia, end-stage renal disease, chronic malabsorption syndromes, low body weight, etc)

*Children less than 4 years of age, or children and adolescents exposed to adults in high-risk categories

*15 mm or more is positive in

*Persons with no known risk factors for TB

*(Note: Targeted skin testing programs should only be conducted among high-risk groups)

A tuberculin test conversion is defined as an increase of 10 mm or more within a 2-year period, regardless of age.

False positive result:
A false positive result may be caused by nontuberculous mycobacteria or previous administration of BCG vaccine. Prior vaccination with BCG may result in a false-positive result for many years afterwards

BCG vaccine and the Mantoux test:
There is disagreement about the role of Mantoux testing in people who have been vaccinated. The US recommendation is that tuberculin skin testing is not contraindicated for BCG-vaccinated persons and that prior BCG vaccination should not influence the interpretation of the test. The UK recommendation is that interferon-? testing should be used to help interpret positive Mantoux tests, and that serial tuberculin skin testing must not be done in people who have had prior BCG vaccination. Please refer to the chapter on latent tuberculosis for a discussion of the two approaches. In general, the US recommendation results in a much larger number of people being falsely diagnosed with latent tuberculosis, while the UK approach probably misses patients with latent tuberculosis who should be treated.

According to the U. S. guidelines, latent TB infection (LTBI) diagnosis and treatment for LTBI is considered for any BCG-vaccinated person whose skin test is 10 mm or greater, if any of these circumstances are present:

*Was in contact with another person with infectious TB

*Was born or has lived in a high TB prevalence country

*Is continually exposed to populations where TB prevalence is high.

Anergy testing:
In cases of anergy, a lack of reaction by the body’s defence mechanisms when it comes into contact with foreign substances, the tuberculin reaction will occur weakly, thus compromising the value of Mantoux testing. For example, anergy is present in AIDS, a disease which strongly depresses the immune system. Therefore, anergy testing is advised in cases where suspicion is warranted that it is present. However, routine anergy skin testing is not recommended.

Two-step testing:
Some people who were previously infected with TB may have a negative reaction when tested years after infection, as the immune system response may gradually wane. This initial skin test, though negative, may stimulate (boost) the body’s ability to react to tuberculin in future tests. Thus, a positive reaction to a subsequent test may be misinterpreted as a new infection, when in fact it is the result of the boosted reaction to an old infection.

Use two-step testing for initial skin testing of adults who will be retested periodically (e.g., health care workers). This ensures that any future positive tests can be interpreted as being caused by a new infection, rather than simply a reaction to an old infection.

*Return to have first test read 48–72 hours after injection

*If first test is positive, consider the person infected.

*If first test is negative, give second test 1–3 weeks after first injection

*Return to have second test read 48–72 hours after injection

*If second test is positive, consider person previously infected

*If second test is negative, consider person uninfected [6]

A person who is diagnosed as “infected” on two-step testing is called a “tuberculin converter”. The US recommendation that prior BCG-vaccination be ignored results in almost universal false diagnosis of tuberculosis infection in people who have had BCG (mostly foreign nationals). Please refer to the chapter on BCG for a discussion of boosting. The UK guidelines avoid this error

Recent developments:
As a replacement for the Mantoux test, several other tests are being developed. QuantiFERON-TB Gold is a blood test that measures the patient’s immune reactivity to the TB bacteria and is useful for initial and serial testing of persons with an increased risk of latent or active tuberculosis infection. Guidelines for the use of QuantiFERON-TB Gold were released by the CDC in December 2005. QuantiFERON-TB Gold is FDA approved in the United States, has CE Mark approval in Europe and has been approved by the MHLW in Japan.

Heaf Test:
The Heaf test is a tuberculin skin test formerly used in the United Kingdom, but discontinued in 2005.

The equivalent Mantoux test positive levels done with 10 TU (0.1 mL 100 TU/mL, 1:1000) are

*<5 mm induration (Heaf 0-1)

*5-15 mm induration (Heaf 2)

*>15 mm induration (Heaf 3-4)

Risk Factors:There are no risks.

Results:The result is known two to three days later when the skin is examined. If the test is positive, your doctor may do blood or urine tests and x-rays of the chest and possibly other parts of the body to look for evidence of an active infection. If you do not have an active infection, your doctor might prescribe an antibiotic given over several months, to help prevent you from developing active tuberculosis. If you do have an active infection, a much more intensive treatment involving multiple antibiotics is required.

Sources:
https://www.health.harvard.edu/fhg/diagnostics/tb-skin-test.shtml
http://en.wikipedia.org/wiki/Mantoux_test

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Categories
Suppliments our body needs

Kefir

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Alternative Names:keefir, kephir, kewra, talai, mudu kekiya, milkkefir, búlgaros

Definition:
Kefir is a fermented milk drink that originated in the Caucasus region. It is prepared by inoculating cow, goat, or sheep’s milk with kefir grains. Traditional kefir was made in skin bags that were hung near a doorway; the bag would be knocked by anyone passing through the doorway to help keep the milk and kefir grains well mixed.

....CLICK & SEE

Overview:
Kefir grains are a combination of bacteria and yeasts in a matrix of proteins, lipids, and sugars. This symbiotic matrix forms grains that resemble cauliflower. Today, kefir is becoming increasingly popular due to new research into its health benefits. Many different bacteria and yeasts are found in the kefir grains, which are a complex and highly variable community of micro-organisms.

Traditional kefir is fermented at ambient temperatures, generally overnight. Fermentation of the lactose yields a sour, carbonated, slightly alcoholic beverage, with a consistency similar to thin yogurt. Kefir fermented by small-scale dairies early in the 20th century achieved alcohol levels between 1% and 2%, but kefir made commercially with modern methods of production has less than 1% alcohol, possibly due to reduced fermentation time.

Variations that thrive in various other liquids exist. They may vary markedly from kefir in both appearance and microbial composition. Water kefir (or kefir d’acqua) is grown in water with sugar (sometimes with added dry fruit such as figs, and lemon juice) for a day or more at room temperature.

Making Kefir;
Production of traditional kefir requires kefir grains which are a gelatinous community of bacteria and yeasts. Kefir grains contain a water soluble polysaccharide known as kefiran that imparts a rope-like texture and feeling in one’s mouth. Kefir grains cannot be produced from scratch, but the grains grow during fermentation, and additional grains are produced. Kefir grains can be purchased or acquired from other hobbyists, see below. Kefir grains appear white to yellow and are usually the size of a walnut, but may be as small as a grain of rice.

Health and nutrition
One can change the nutrient content by simply fermenting for shorter or longer periods. Both stages have different healthful benefits. For instance, kefir over-ripened (increases sour taste) significantly increases folic acid content. Kefir also aids in lactose digestion as a catalyst, making it more suitable than other dairy products for those who are lactose intolerant. The kefiran in kefir has been shown to suppress an increase in blood pressure and reduce serum cholesterol levels in rats.

Drinking kefir
While some drink kefir straight, many find it too sour on its own and prefer to add fruits, honey, maple syrup or other flavors or sweeteners. Frozen bananas, strawberries, blueberries or other fruits can be mixed with kefir in a blender to make a smoothie. Vanilla, agave nectar and other flavorings may also be added. It is a breakfast, lunch and dinner drink popular across all areas of the former Soviet Union and Finland, where it it is known as an affordable health drink

Different milk types
Kefir grains will successfully ferment the milk from most mammals, and will continue to grow in such milk. Typical milks used include cow, goat, and sheep, each with varying organoleptic and nutritional qualities.

In addition, kefir grains will ferment milk substitutes such as soy milk, rice milk, and coconut milk, as well as other sugary liquids including fruit juice, coconut water, beer wort and ginger beer. However, the kefir grains may cease growing if the medium used does not contain all the growth factors required by the bacteria (which are all present in mammalian milk), so it is best to only use excess kefir grains for trying alternative fermentation media.

Milk sugar is, however, not essential for the synthesis of the polysaccharide that makes up the grains (kefiran), and scientific studies have demonstrated that rice hydrolysate is a suitable alternative medium. Additionally, it has been shown that kefir grains will reproduce when fermenting soy milk, although they will change in appearance and size due to the differing proteins available to them.

Culinary uses & benefits
Kefir is one of the main ingredients in Lithuanian cold beet soup (šaltibarš?iai, commonly known as cold borscht) and Russian summer soup (okroshka). Other variations of kefir soups and foods prepared with kefir are popular across the former Soviet Union.

Others enjoy kefir, in lieu of milk, on cereal or granola.

Kefir is a cultured, enzyme-rich food filled with friendly micro-organisms that help balance your “inner ecosystem.” More nutritious and therapeutic than yogurt, it supplies complete protein, essential minerals, and valuable B vitamins.

*Kefir is simple and inexpensive to make at home.

*Kefir is used to restore the inner eco-system after antibiotic therapy.

*Kefir can be made into a delicious smoothie that kids love.

*Kefir is excellent nourishment for pregnant and nursing women, the elderly, and those with compromised immunity.


What if I’m lactose intolerant, don’t do dairy or don’t digest milk products well – is kefir right for me?

The beneficial yeast and friendly bacteria in the kefir culture consume most of the lactose (or milk sugar). Eat kefir on an empty stomach first thing in the morning before (or for) breakfast and you’ll be delighted to find it can be easily digested — as numerous people who have been lactose intolerant for years have discovered.

You may click to see:->

Answers to Frequently Asked Questions about Kefir

Both kefir and yogurt are cultured milk products

The Body Ecology Diet and Kefir

Discover the Incredible Health-Promoting Benefits of Kefir
Kefir recipes

Resources:
http://en.wikipedia.org/wiki/Kefir

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Categories
Psychiatry

Delusion

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Definition:
A delusion is commonly defined as a fixed false belief and is used in everyday language to describe a belief that is either false, fanciful or derived from deception. In psychiatry, the definition is necessarily more precise and implies that the belief is pathological (the result of an illness or illness process). As a pathology it is distinct from a belief based on false or incomplete information or certain effects of perception which would more properly be termed an apperception or illusion.

Delusions typically occur in the context of neurological or mental illness, although they are not tied to any particular disease and have been found to occur in the context of many pathological states (both physical and mental). However, they are of particular diagnostic importance in psychotic disorders and particularly in schizophrenia and bipolar disorder.

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Psychiatric definition
Although non-specific concepts of madness have been around for several thousand years, the psychiatrist and philosopher Karl Jaspers was the first to define the three main criteria for a belief to be considered delusional in his book General Psychopathology. These criteria are:

*certainty (held with absolute conviction)

*incorrigibility (not changeable by compelling counterargument or proof to the contrary)

*impossibility or falsity of content (implausible, bizarre or patently untrue)

These criteria still continue in modern psychiatric diagnosis. In the most recent Diagnostic and Statistical Manual of Mental Disorders, a delusion is defined as:

A false belief based on incorrect inference about external reality that is firmly sustained despite what almost everybody else believes and despite what constitutes incontrovertible and obvious proof or evidence to the contrary. The belief is not one ordinarily accepted by other members of the person’s culture or subculture (e.g., it is not an article of religious faith).

Symptoms:
The criteria that define delusional disorder are furnished in the Diagnostic and Statistical Manual of Mental Disorders Fourth Edition Text Revision, or DSM-IV-TR, published by the American Psychiatric Association. The criteria for delusional disorder are as follows:

*non-bizarre delusions which have been present for at least one month

*absence of obviously odd or bizarre behavior

*absence of hallucinations, or hallucinations that only occur infrequently in comparison to other psychotic disorders

*no memory loss, medical illness or drug or alcohol-related effects are associated with the development of delusions

Diagnostic issues:
The modern definition and Jaspers’ original criteria have been criticised, as counter-examples can be shown for every defining feature.

Studies on psychiatric patients have shown that delusions can be seen to vary in intensity and conviction over time which suggests that certainty and incorrigibility are not necessary components of a delusional belief.

Delusions do not necessarily have to be false or ‘incorrect inferences about external reality’. Some religious or spiritual beliefs by their nature may not be falsifiable, and hence cannot be described as false or incorrect, no matter whether the person holding these beliefs was diagnosed as delusional or not.

In other situations the delusion may turn out to be true belief. For example, delusional jealousy, where a person believes that their partner is being unfaithful (and may even follow them into the bathroom believing them to be seeing their lover even during the briefest of partings) may result in the faithful partner being driven to infidelity by the constant and unreasonable strain put on them by their delusional spouse. In this case the delusion does not cease to be a delusion because the content later turns out to be true.

In other cases, the delusion may be assumed to be false by a doctor or psychiatrist assessing the belief, because it seems to be unlikely, bizarre or held with excessive conviction. Psychiatrists rarely have the time or resources to check the validity of a person’s claims leading to some true beliefs to be erroneously classified as delusional.This is known as the Martha Mitchell effect, after the wife of the attorney general who alleged that illegal activity was taking place in the White House. At the time her claims were thought to be signs of mental illness, and only after the Watergate scandal broke was she proved right (and hence sane).

Similar factors have led to criticisms of Jaspers’ definition of true delusions as being ultimately ‘un-understandable’. Critics (such as R. D. Laing) have argued that this leads to the diagnosis of delusions being based on the subjective understanding of a particular psychiatrist, who may not have access to all the information which might make a belief otherwise interpretable.

Another difficulty with the diagnosis of delusions is that almost all of these features can be found in “normal” beliefs. Many religious beliefs hold exactly the same features, yet are not universally considered delusional. Similarly, Thomas Kuhn argued in The Structure of Scientific Revolutions that scientists can hold strong beliefs in scientific theories despite considerable apparent discrepancies with experimental evidence.

These factors have led the psychiatrist Anthony David to note that “there is no acceptable (rather than accepted) definition of a delusion.” In practice psychiatrists tend to diagnose a belief as delusional if it is either patently bizarre, causing significant distress, or excessively pre-occupies the patient, especially if the person is subsequently unswayed in belief by counter-evidence or reasonable arguments.

Click for Delusions Description, Types

Diagnosis:

Client interviews focused on obtaining information about the sufferer’s life situation and past history aid in identification of delusional disorder. With the client’s permission, the clinician obtains details from earlier medical records, and engages in thorough discussion with the client’s immediate family—helpful measures in determining whether delusions are present. The clinician may use a semi-structured interview called a mental status examination to assess the patient’s concentration, memory, understanding the individual’s situation and logical thinking. The mental status examination is intended to reveal peculiar thought processes in the patient. The Peters Delusion Inventory (PDI) is a psychological test that focuses on identifying and understanding delusional thinking; but its use is more common in research than in clinical practice.

Even using the DSM-IV-TRcriteria listed above, classification of delusional disorder is relatively subjective. The criteria “non-bizarre” and “resistant to change” and “not culturally accepted” are all subject to very individual interpretations. They create variability in how professionals diagnose the illness. The utility of diagnosing the syndrome rather than focusing on successful treatment of delusion in any form of illness is debated in the medical community. Some researchers further contend that delusional disorder, currently classified as a psychotic disorder, is actually a variation of depression and might respond better to antidepressants or therapy more similar to that utilized for depression. Also, the meaning and implications of “culturally accepted” can create problems. The cultural relativity of “delusions,”—most evident where the beliefs shown are typical of the person’s subculture or religion yet would be viewed as strange or delusional by the dominant culture—can force complex choices to be made in diagnosis and treatment. An example could be that of a Haitian immigrant to the United States who believed in voodoo. If that person became aggressive toward neighbors issuing curses or hexes, believing that death is imminent at the hands of those neighbors, a question arises. The belief is typical of the individual’s subculture, so the issue is whether it should be diagnosed or treated. If it were to be treated, whether the remedy should come through Western medicine, or be conducted through voodoo shamanistic treatment is the problem to be solved.

Treatments:
Delusional disorder treatment often involves atypical(also callednovelornewer-generation) antipsychotic medications, which can be effective in some patients. Risperidone(Risperdal), quetiapine(Seroquel), and olanzapine(Zyprexa) are all examples of atypical or novel antipsychotic medications. If agitationoccurs, a number of different antipsychotics can be used to conclude the outbreak of acute agitation. Agitation, a state of frantic activity experienced concurrently with anger or exaggerated fearfulness, increases the risk that the client will endanger self or others. To decrease anxiety and slow behavior in emergency situations where agitation is a factor, an injection of haloperidol(Haldol) is often given usually in combination with other medications (often lorazepam, also known as Ativan). Agitation in delusional disorder is a typical response to severe or harsh confrontation when dealing with the existence of the delusions. It can also be a result of blocking the individual from performing inappropriate actions the client views as urgent in light of the delusional reality. A novel antipsychotic is generally given orally on a daily basis for ongoing treatment meant for long-term effect on the symptoms. Response to antipsychotics in delusional disorder seems to follow the “rule of thirds,” in which about one-third of patients respond somewhat positively, one-third show little change, and one-third worsen or are unable to comply.

Cognitive therapy has shown promise as an emerging treatment for delusions. The cognitive therapist tries to capitalize on any doubt the individual has about the delusions; then attempts to develop a joint effort with the sufferer to generate alternative explanations, assisting the client in checking the evidence. This examination proceeds in favor of the various explanations. Much of the work is done by use of empathy, asking hypothetical questions in a form of therapeutic Socratic dialogue—a process that follows a basic question and answer format, figuring out what is known and unknown before reaching a logical conclusion. Combining pharmacotherapy with cognitive therapy integrates both treating the possible underlying biological problems and decreasing the symptoms with psychotherapy.

Prognosis:
Evidence collected to date indicates about 10% of cases will show some improvement of delusional symptoms though irrational beliefs may remain; 33–50% may show complete remission; and, in 30–40% of cases there will be persistent non-improving symptoms. The prognosis for clients with delusional disorder is largely related to the level of conviction regarding the delusions and the openness the person has for allowing information that contradicts the delusion.

Prevention:
Little work has been done thus far regarding prevention of the disorder. Effective means of prevention have not been identified.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://en.wikipedia.org/wiki/Delusions
http://www.minddisorders.com/Br-Del/Delusional-disorder.html

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