Categories
Ailmemts & Remedies

Hair Loss in Women

Introduction:
One of the commonest forms of hair loss in women (and men) is a condition called telogen effluvium, in which there is a diffuse (or widely spread out) shedding of hairs around the scalp and elsewhere on the body.

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This is usually a reaction to intense stress on the body’s physical or hormonal systems, or as a reaction to medication.

The condition, which can occur at any age, generally begins fairly suddenly and gets better on its own within about six months, although for a few people it can become a chronic problem.

Because telogen effluvium develops a while after its trigger, and causes generalised thinning of hair density rather than a bald patch, women with the condition can easily be diagnosed as overanxious or neurotic.

Fortunately, it often gets better with time. Telogen effluvium is a phenomenon related to the growth cycles of hair.

Hair growth cycles alternate between a growth phase (called anagen, it lasts about three years) and a resting phase (telogen, which lasts about three months). During telogen, the hair remains in the follicle until it is pushed out by the growth of a new hair in the anagen phase.

At any one time, up to about 15 per cent of hairs are in telogen. But a sudden stress on the body can trigger large numbers of hairs to enter the telogen phase at the same time. Then, about three months later, this large number of hairs will be shed. As the new hairs start to grow out, so the density of hair may thicken again.

Many adults have had an episode of telogen effluvium at some point in their lives, reflecting episodes of illness or stress.

Another common type of hair loss in women is androgenetic alopecia, which is related to hormone levels in the body. There’s a large genetic predisposition, which may be inherited from the father or mother.

Androgenetic alopecia affects roughly 50 per cent of men (this is the main cause of the usual pattern of balding seen as men age) and perhaps as many women over the age of 40.

Research shows that up to 13 per cent of women have some degree of this sort of hair loss before the menopause, and afterwards it becomes far more common – one piece of research suggests that over the age of 65 as many as 75 per cent of women are affected.

The cause of hair loss in androgentic alopecia is a chemical called dihydrotestosterone, or DHT, which is made from androgens (male hormones that all men and women produce) by the action of an enzyme called 5-alpha reductase.

People with a lot of this enzyme make more DHT, which in excess can cause the hair follicles to make thinner and thinner hair, until eventually they pack up completely.

Women’s pattern of hair loss is different to the typical receding hairline and crown loss in men. Instead, androgenetic alopecia causes a general thinning of women’s hair, with loss predominantly over the top and sides of the head.

Another important cause of hair loss in women is a condition called alopecia areata, an autoimmune disease that affects more than two per cent of the population. In this, the hair follicles are attacked by white blood cells. The follicles then become very small and hair production slows down dramatically, so there may be no visible hair growth for months and years.

After some time, hair may regrow as before, come back in patchy areas, or not regrow at all. The good news is that in every case the hair follicles remain alive and can be switched on again; the bad news is that we don’t yet know how to do this.

TOP MYTHS ABOUT FEMALE HAIR LOSS:-
•It means you’re not a proper women with two X chromosomes.
•It’s caused by washing your hair too often.
•It’s caused by too much brushing or combing.
•Hair dyes and perms can cause permanent loss.
•It may result from wearing hats and wigs.
•Shaving your hair will make it regrow thicker.
•Standing on your head will help it grow back.
•It’s a sign of an overactive brain.
•There’s a miracle cure out there waiting for you.
•Scan the internet and you’ll see all sorts of miracle cures for baldness on offer, from strange herbal lotions to mechanical devices. Perhaps the most useful first step you can take is to avoid the myths.
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After this there are several options. You can find some way to accept the change and live with it (let’s face it, this is a tall order – most men struggle to come to terms with their baldness and for them at least society equates it with maturity and power).

You can try cosmetic treatments such as wigs or hair thickeners, or you can try medical therapies. The last option is hair-replacement surgery.

The drug minoxidil was first developed for treating high blood pressure, which was found to have the side effect of thickening hair growth in some people. It’s now available as a lotion to apply directly to the scalp.

No one really knows how it works, however, and it’s not effective for everyone. Studies show that only about 20 per cent of women between 18 and 45 have moderate regrowth using the drug, while another 40 per cent experience minimal regrowth.

It works best on younger people with early hair loss. A big disadvantage is that you have to carry on using minoxidil indefinitely or the new hair will fall out.

Another drug, finasteride, which was developed for treating prostate cancer, has also been found to be effective but is only available for men.

Surgical techniques for restoring hair have improved greatly in the past couple of decades, but this is still an option that requires careful consideration.

There are two main options:
•Hair transplantation – tiny punch-holes of skin containing a few follicles of hair are taken from elsewhere in the body (such as the back of the head, if this is still well covered) and implanted into the thinning areas. Some surgeons use a needle to sew in just one or two hairs. However, as women are more likely to have diffuse loss of hair all over the scalp, this technique may not be possible. There has been little success with implanting artificial fibres.
•Scalp reduction – devices are inserted under the skin to stretch areas of scalp that still have hair, then the redundant bald areas are removed. Alternatively, flaps of hairy scalp can be moved around the head.
Key points
•Many causes of female hair loss are temporary – check your general health and be patient.
•Take a look at your family for an idea of your risk of female pattern baldness.
•Don’t be taken in by claims for wonder products – there’s no cure for female pattern hair loss.
•Many women cope well by using cosmetic products, hats and wigs, so persevere until you find your own style.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources :

http://www.bbc.co.uk/health/physical_health/conditions/hair_loss_women.shtml

http://www.prevention.com/health/beauty/unsure/hair-loss-in-women/article/1aebd08f88803110VgnVCM20000012281eac____/

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Ailmemts & Remedies

Hair Loss in Men

Definition:
Each hair grows in cycles?it grows, rests, and then falls out. Usually, this cycle repeats approximately yearly. At any time, about ninety percent of a person’s scalp hair is growing, a phase that lasts between two and six years. Ten percent of the scalp hair is in a resting phase that lasts between two and three months. At the end of its resting stage, the hair goes through a shedding phase.

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Shedding 50 to 100 hairs a day is considered normal. When a hair is shed, it is replaced by a new hair from the same follicle located just beneath the skin surface. Scalp hair grows about one-half inch a month.

Hair is made up of a form of protein, the same material that is found in fingernails and toenails. Everyone, regardless of age, should eat an adequate amount of protein to maintain normal hair production. Protein is found in meat, chicken, fish, eggs, some cheese, dried beans, tofu, grains, and nuts.

Male pattern baldness occurs in a distinct way (and sometimes can affect women too). Hair may start Hair may start to disappear from the temples and the crown of the head at any time. For a few men this process starts as early as the later teenage years, but for most it happens in their late 20s and early 30s. A little thinning of the hair may be noticeable first, followed by wider hair loss allowing more of the scalp to become visible.

Some men aren’t troubled by this process at all. Others, however, suffer great emotional distress associated with a lack of self-confidence and sometimes depression.

Abnormal hair loss can be due to many different causes, but about 50 percent of the population experience normal hair loss by the time they reach 50. People who notice their hair shedding in large amounts after combing or brushing, or whose hair becomes thinner or falls out should consult a dermatologist.

Causes:
By far the most common cause of hair loss in men is androgenetic alopecia, also referred to as “male pattern” or “common” baldness. It is caused by the effects of the male hormone dihydrotestosterone (DHT) on genetically susceptible scalp hair follicles. This sensitivity to DHT is present mainly in hair follicles that reside in the front, top, and crown of the scalp (rather than the back and sides) producing a characteristic and easily identifiable pattern described by Norwood (see Norwood Classification).

It is frequently stated that “hair loss comes from the mother’s side of the family.” The truth is that baldness can be inherited from either parent. However, recent research suggests that the reasons for hair loss and balding may be a bit more complex than originally thought. Factors on the x-chromosome have been shown to influence hair loss, making the inheritance from the maternal side of the family slightly more important than the paternal one (Markus Nothen, 2005).

The identification of an androgen receptor gene (AR) on the x-chromosome helps to explain why the hair loss pattern of a man resembles his maternal grandfather more often than his father. However, this is clearly not the whole story since a direct inheritance of baldness from the father is observed as well. An autosomal (non-sex) linked gene would explain this type of transmission – but this gene has not yet been found.

DHT is formed by the action of the enzyme 5-alpha reductase on testosterone, the hormone that causes sex characteristics in men. DHT causes male hair loss by shortening the growth, or anagen, phase of the hair cycle, causing miniaturization (decreased size) of the follicles, and producing progressively shorter, finer hairs. Eventually these hairs totally disappear.

In the patient below, we see a close-up of the side of his scalp where the hair is not affected by DHT. We see mostly groups of full thickness hairs (called terminal hairs) and a few scattered fine, vellus hairs. This is normal.
In the area of thinning (see circle below), we see that most of the hair has been miniaturized, although all of the hair is still present.

In the region that is balding (second circle in the center), there is extensive miniaturization and some, but not all of the hair has disappeared.

What this shows is that the initial appearance of balding is due to the progressive decrease in hair shaft size, rather than the actual loss of hair – in early hair loss, all the hair is still present. This is the reason why hair loss medications, such as finasteride (Propecia) work in early hair loss (since they are able to partly reverse the miniaturization process) but don’t work in areas that are totally bald. It is also the reason why men’s hair restoration surgery, if not planned properly, can result in hair loss due to the shedding of surrounding miniaturized hair.
Androgenetic hair loss is caused by three interdependent factors: genes, hormones, and age:

Other causes of hair loss, which may not follow this pattern, include:

•Iron-deficiency anaemia
•Under active thyroid
•Fungal scalp infection
•Some prescribed medicines
•Stress

Other medical conditions that can produce diffuse hair loss in men include thyroid disease . Certain medications, including some drugs used for high blood pressure and depression, and the use of anabolic steroids, can also cause male hair loss.

How the problem can be solved:
If there’s a reversible cause, it’s normally possible to stop hair loss. For instance, if it’s caused by iron deficiency you can stop hair loss by replenishing the body’s iron stores.

A huge number of treatments have been tried to slow down and even reverse the process of male pattern hair loss – some are successful, others aren’t. But many men find their hair loss slows down or stops for no apparent reason at a certain age anyway.

It’s a good idea to ensure an illness isn’t responsible, particularly if the hair loss is patchy rather than being in the typical male pattern distribution. Moreover, if the hair loss is accompanied by other symptoms (such as tiredness) then blood tests may be necessary.

Treatments
•Wigs, weaves and hair transplants are, obviously, the most direct form of treatment, while some advocate shaving or close cutting which simply makes the hair loss less obvious. Different hairstyles can create the appearance of a fuller head of hair, or a close shave cut can make baldness less apparent.
•Herbal preparations that contain zinc, magnesium, iron, vitamin E and other substances in various combinations can help.
•Minoxidil is a lotion available from the pharmacist that you rub on to the scalp. It slows down the process of hair loss and can cause new hair growth but you have to keep on using it or it will stop being effective.
•Finasteride (Propecia) is the latest drug treatment. It comes in tablet form and works by slowing down hair loss; it’s also reported to cause new hair growth. In the UK it’s only available on private prescription from your GP and is only effective while you take it.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://www.bbc.co.uk/health/physical_health/conditions/hair_loss_male.shtml

Causes


http://beatpsoriasis.com/baldness-definition.htm

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Herbs & Plants

Beach Pea

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Botanical Name :Lathyrus japonicus
Family: Fabaceae
Subfamily: Faboideae
Tribe: Vicieae
Genus: Lathyrus
Species: L. japonicus
Kingdom: Plantae
Order: Fabales
Common Name :Sea Pea, Beach Pea, Circumpolar Pea, Sea Vetchling

Habitat :Native to temperate coastal areas of Asia, Europe, North and South America.

Description:
It is a herbaceous perennial plant growing trailing stems to 50–80 cm long, typically on sand and gravel storm beaches. The leaves are waxy glaucous green, 5–10 cm long, pinnate, with 2-5 pairs of leaflets, the terminal leaflet usually replaced by a twining tendril. The flowers are 14–22 mm broad, with a dark purple standard petal and paler purple wing and keel petals; they are produced in racemes of 2-7 together.

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The unusually extensive native range is explained by the ability of the seeds to remain viable while floating in sea water for up to 5 years, enabling the seeds to drift nearly worldwide. Germination occurs when the hard outer seed coat is abraded by waves on sand and gravel.

Medicinal Uses:
The leaves of the plant are used in Chinese traditional medicine.
Chinese used this Pacific Rim wild food as a tonic for the urinary organs and intestinal tract.  Eskimo considered the peas poisonous…Iroquois treated rheumatism with cooked whole young plant.

Known Hazards :The pods can be eaten but like many members of the genus Lathyrus they contain -diaminopropionic acid, which can cause paralysis called lathyrism.

Disclaimer:
The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

Resources:
http://en.wikipedia.org/wiki/Lathyrus_japonicus
http://www.herbnet.com/Herb%20Uses_AB.htm
http://calphotos.berkeley.edu/cgi/img_query?enlarge=6040+1631+1307+0099

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Categories
Ailmemts & Remedies

Haemospermia (Blood in the Semen)

Definition:
When semen changes from its usual cream-white colour to white with ‘a hint of pink’, or is bloodstained, it’s called haemospermia, which simply means blood in the semen. The semen can also appear brownish-red in colour. Whatever the shade, it isn’t normal and means something is not right and should be checked out.

One problem with haemospermia is that it invariably causes men great anxiety. Another problem is that the cause often remains unknown.

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click to see the pictures

Hematospermia (haematospermia), (or Hemospermia, haemospermia) or the presence of blood in semen, is most often a benign and idiopathic symptom, but can sometimes result from medical problems such as a urethral stricture, infection of the prostate, or a congenital bleeding disorder, and can occur transiently after surgical procedures such as a prostate biopsy. It is present in less than 2% of urology referrals, although prevalence in the overall population is unknown.

Patients with hematospermia should be evaluated by a urologist to identify or rule out medical causes. Idiopathic hematospermia is sometimes treated with tetracycline and prostatic massage.

Causes/Risk Factors
Haemospermia most commonly affects men in their 30s, although it’s by no means exclusive to this age group.

It’s not always possible to prevent it from occurring, but if the cause can be found then taking care to avoid such triggers can result in fewer sleepless nights.

Most commonly, haemospermia is a consequence of non-specific inflammation of the urethra (the tube urine passes through), prostate and/or seminal vesicles. That is, inflammation anywhere along the path semen follows when leaving the body.

Other possible causes include:
•Minor trauma – the result of vigorous sexual activity, for example, or a trouser-zip accident.
•Infections of the genital tract ­ this is usually accompanied by other symptoms, including pain on urination, scrotal tenderness or swelling, groin tenderness or aching, pain on ejaculation, low back pain, fever or chills.
•Biopsy of the prostate gland, where seminal fluid is manufactured.
•In rare instances, haemospermia is secondary to cancer.
For most men, haemospermia is a one-off event. For some, though, the problem is a recurrent one for which a cause cannot be identified, despite full and thorough investigation.

Diagnosis:
This disease generally affects men after their 30s though it can not be confirmed that men of other age group are not at risk. It cannot be prevented from happening always but if the cause is detected  preventive measures can be taken easily.

If the underline cause is found by the doctor,such as an inflamation or an infection,he can prescribe proper medicine

Treatment/Recovery
It may necessary to refer a man with haemospermia to the local hospital urology service where a number of tests such as laboratory examination of urine and semen, ultrasound or CT scans, or even a cystourethroscopy (a telescopic examination of the inside of the urinary tract under anaesthetic) may be recommended in order to check the diagnosis.

Fortunately, in the majority of cases haemospermia is benign and self-limiting, so no specific treatment is required other than a large dose of reassurance and advice about safer practices.

Where an underlying cause is identified – for example, infection or inflammation – specific treatment can be provided. This may involve a course of antibiotics or anti-inflammatory medication.

Advice :
The treatment is not always gaurantee  that the condition will not reappear.If the ailment is caused by some blood related problems then treating that may give better results. It is always advicible for men who have suffered from this disease  to monitor their semen  for any further occurance of bleeding.They should remember various factors as time of blood appear in their semen and they should keep count of times  they had sex  recently. They also need to be cautioned about STD and make their urine routine check.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:

http://en.wikipedia.org/wiki/Hematospermia
http://www.ayushveda.com/healthcare/haemospermia-blood-in-the-semen.htm
http://www.bbc.co.uk/health/physical_health/conditions/haemospermia1.shtml

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Herbs & Plants

Myrica cerifera

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Botanical Name : Myrica cerifera
Family: Myricaceae
Genus: Myrica
Species: M. cerifera
Kingdom: Plantae
Order: Fagales

Synonyms : Cerothamnus arborescens, Cerothamnus cerifer, Morella cerifera, Myrica mexicana, Myrica pumila.

Common Names :Wax Myrtle – Bayberry Wild Cinnamon, Southern Bayberry, Wax Myrtle, Southern Wax Myrtle (Southern)  Bayberry, Candleberry, Bayberry tree, and Tallow shrub.

Habitat : Myrica cerifera  is native to South-eastern N. America. Possibly naturalized in S. England. It grows in the  thickets on sandy soil near swamps and marshes, also on dry arid hills in which situation it is often only a few centimetres tall..

Description:
Myrica cerifera is a small tree or large shrub, and is adaptable to many habitats. It grows naturally in wetlands, near flowing bodies of water, sand dunes, fields, hillsides, pine barrens, and in both needleleaf and mixed-broadleaf forests. Specimens in drier and sandier areas are shrub-like, have rhizomes and smaller leaves than usual. Specimens in wetter areas are more tree-like with bigger leaves. However, these two forms are not clear-cut, with many intermediate forms. It is found in various habitats ranging from Central America to Delaware and Maryland in the United States. However, the plant can be successfully cultivated as far north as southern Connecticut and Long Island on the U.S. east coast. It also grows in Bermuda and the Caribbean. In terms of succession, M. cerifera is often one of the first plants to colonize an area.

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M. cerifera is an evergreen. The leaves are long, and have leathery textures and serrated edges. They contain aromatic compounds. The leaves are glandular.

This plant is dioecious. Male flowers have three or four stamens, and are surrounded by short bracts. The flowers are borne on catkins. The female flowers develop into fruit, which are globular and surrounded by a natural wax-like coating. All flowers are borne in inflorescences. The species flowers in late winter to spring, and female specimens bear fruit in late summer or fall. No endosperm is present on the seeds. M. cerifera can also reproduce clonally through runners.

The fruit is a source of food for a lot of bird species, including the Northern Bobwhite Quail and the Wild Turkey. In winter, the seeds are important foods for the Carolina Wren and species of Tree Sparrow. To a point, M. cerifera will also provide habitat for the Northern Bobwhite Quail. Birds digestive systems’ remove the wax from the fruit, which a prerequisite for germination.

This plant’s roots possess root nodules. These are home to a symbiotic species of actinomycotal fungus, which fixes nitrogen at a faster rate than legumes.

M. cerifera, or rather its shoot, cannot handle wildfires well. Indeed, since the leaves, stem, and branches contain flammable aromatic compounds, a specimen of M. cerifera is a fire hazard. For that reason, a wildfire will often kill the shoot. Only a very small or transient fire will do less. In that case, only the most recent primary growth may be incinerated. In contrast to the weakness of its shoot, M. cerifera’s root system is fire-resistant. As of 1991, no known fire has killed this plant’s roots. However, this plant will not survive shoot destruction indefinitely. Three consecutive years of shoot destruction may kill all plants affected. If this does not happen, this species will regrow a shoot. This is most rapid in the first season after a fire.

Cultivation:
Landscape Uses:Border, Erosion control, Screen, Seashore, Specimen. Prefers a moist soil. Grows well in an open position in a well-drained soil in sun or light shade. Thrives in any ordinary garden soil according to one report whilst another says that it thrives in an acid soil. Prefers a lime-free loamy or peaty soil. Plants can be evergreen in areas with warmer winters than in Britain. Some reports say that the plant is dioecious whilst others say it is monoecious. It is most likely that both forms exist. A polymorphic species, there are some named forms. ‘Myda’ is a large-fruited female form of low growth. The fruit is covered with a deposit of wax that has a balsamic odour. The fruits can hang on the plant for several years. Closely related to M. pensylvanica, with which it hybridizes. Plants in this genus are notably resistant to honey fungus. Many species in this genus have a symbiotic relationship with certain soil micro-organisms, these form nodules on the roots of the plants and fix atmospheric nitrogen. Some of this nitrogen is utilized by the growing plant but some can also be used by other plants growing nearby. Special Features:Attracts birds, North American native, Fragrant foliage, Naturalizing, Wetlands plant, Attracts butterflies, Inconspicuous flowers or blooms.

Propagation: 
Seed – best sown as soon as it is ripe in the autumn in a cold frame. Stored seed germinates more freely if given a 3 month cold stratification and then sown in a cold frame. Germination is usually good. Prick out the seedlings into individual pots when they are large enough to handle and grow them on in the cold frame for the first winter. Plant out in late spring or early summer. Cuttings of half-ripe wood, 5 – 8cm with a heel, July/August in a frame. Pot up and overwinter in a cold frame then plant out in late spring or early summer. Fair to good percentage. Layering in spring

Edible Uses:
Edible Parts: Fruit.
Edible Uses: Condiment; Tea.

Fruit – raw or cooked. The fruit is about 3mm in diameter with a large seed. There is very little edible flesh and the quality is poor. Leaves and berries are used as a food flavouring. They make an aromatic, attractive and agreeable substitute for bay leaves, and can be used in flavouring soups, stews etc. The dried leaves are brewed into a robust tea

Medicinal Uses:
Bayberry root bark is the part used in herbalism. The plant contains several organic compounds, including: triterpenes such as myricadiol, taraxerol, and taraxerone, as well as chemicals such as different flavonoids, tannins, resins, gums, and phenols. These compounds have varying effects. Myricadiol has a slight impact on levels of potassium and sodium, while a substance called myricitrin has antibiotic properties.

A key herb in the Thomsonian system of medicine, being the main astringent used for “any stomach or bowel derangement, particularly after fevers.”   Internally used for fevers, colds, influenza, excess mucus, diarrhea, colitis, excessive menstruation, and vaginal discharge.  Externally for sore throat, ulcers, sores, itching skin conditions, dandruff and hair loss.  Bayberry is commonly used to increase circulation, stimulate perspiration, and keep bacterial infections in check. Colds, flu, coughs, and sore throats benefit from treatment with this herb as a hot decoction.  It helps to strengthen local resistance to infection and to tighten and dry mucous membranes.  An infusion is helpful for strengthening spongy gums, and a gargle is used for sore throat.  Bayberry’s astringency helps intestinal disorders such as irritable bowel syndrome and mucous colitis. It increases circulation to the area while acting to tone tissues involved. An infusion can also help treat excess vaginal discharge.  A paste of the powdered root bark may be applied onto ulcers and sores.  The powdered bark has been used as a snuff for congested nasal passages.  It has been used to treat post-partum hemorrhage and taken internally and used as a douche is recommended for excessive menstruation and leucorrhea.  It is used as a poultice to soothe varicose veins. Myricadiol has a mild effect on potassium and sodium levels.  Myricitrin is antibacterial and encourages the flow of bile.  The powder is strongly sternutatory and excites coughing. Water in which the wax has been ‘tried,’ when boiled to an extract, is regarded as a certain cure for dysentery, and the wax itself, being astringent and slightly narcotic, is valuable in severe dysentery and internal ulcerations. The leaves have provided vitamin C for curing scurvy.

Bayberry has a history of medicinal use. The Choctaw boiled and used the result as a treatment for fevers. Bayberry was eventually adopted as a medicinal plant, but only in the South. In 1722, it was reported that colonists in Louisiana drank a mixture of wax and hot water to treat severe dysentery.   Bayberry was reported in an account from 1737 as being used to treat convulsions, colic, palsy, and seizures. Starting in the early 19th century, a herbalist called Samuel Thompson recommended this plant for producing “heat” within the body and as a treatment for infectious diseases and diarrhea. That use of bayberry waned later in the 19th century, in favor of using it for a variety of ailments, including a topical use for bleeding gums. For twenty years starting in 1916, bayberry root bark was listed in the American National Formulary.

Medicinal use of Bayberry has declined since its peak in popularity in the 19th century. The plant is still used today in the treatment of fever, diarrhea, and a few other ailments. The chemical myricitrin has anti-fever properties. In addition, that chemical, along with the tannins, has anti-diarrheal properties. The myricitrin works as an antibiotic, while the tannins have astringent properties.

In general, either a decoction or a tincture is used. Infusions and a topical paste have also been used.

Pregnant women should not use Bayberry. In addition, tannin action relating to cancer is unclear, with studies indicating both pro and anti-cancer effects. Bayberry, just like any other medicinal plant, should only be used under the supervision of a physician

Other Uses:
Ornamental

Myrica cerifera finds use in gardening and horticulture. It has been commonly purported to grow in American hardiness zones of 11 to 7b. However, this is an old, conservative estimate; in recent years, plants have performed well along the east coast as far north as zone 6b in northern Rhode Island. M. pensylvanica substitutes for M. cerifera in areas colder than zone 6. Since the species is adaptable, it will tolerate many conditions, although it has a need for frequent pruning. It can handle abuse from bad pruning, however. The species has at least four cultivars. Those dubbed Fairfax, Jamaica Road, and Don’s Dwarf differ from the “typical” specimen in habit and form. The latter two are also resistant to leaf spot. Var. pumila is a dwarf cultivar

Candles;

Southern Bayberry’s fruits are a traditional source of the wax for those old-fashioned Christmas decorations called bayberry candles.  The wax was extracted by boiling the berries, and skimming off the floating hydrocarbons. The fats were then boiled again and then strained. After that the liquid was usable in candle making, whether through dipping or molding. Southern Bayberry is not the only plant usable for making bayberry candles, however. Its close relatives are also usable.

Southern Bayberry and its relatives have largely been supplanted in candlemaking by substitutes made from paraffin. The substitute candles have artificial colors and scents that create candles that look and smell similar to natural ones

Disclaimer:
The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

Resources:
http://en.wikipedia.org/wiki/Myrica_cerifera
http://www.herbnet.com/Herb%20Uses_AB.htm

http://www.ag.auburn.edu/hort/landscape/dbpages/233.html

http://www.sbs.utexas.edu/bio406d/images/pics/myr/myrica_cerifera.htm

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