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Ailmemts & Remedies

“Eczema”- That Irritating Itch!

Eczema is a strange disease. If you have it, the frustrating itching and scratching starts and then just never seems to go away. The number of people with eczema is increasing. One in five now develops it in childhood. Many factors, such as early weaning, chemicals (preservatives and pesticides) in the human diet, perfumes and pollution, have been blamed. The fact is no one really knows the cause of eczema, but we do know that exclusively breast-fed infants are less prone to it.

You may click to see the pictures…...……Eczema

Eczema vaccinatum infant.

Eczema on face

Eczema on back

Eczema appears as red, itchy, dry and flaky skin, with or without small pustules. A few months of constant itching may make the skin permanently rough, thick and hyperpigmented.

The classification of eczema is not very clear. It can be due to atopy (tendency to develop allergy), allergic contact, infantile seborrhoea (dandruff-like condition of the head that occurs in infants) or varicose veins.

Atopic eczema is the commonest form. It usually occurs in childhood as an itchy rash distributed on the head and scalp, neck, inside of elbows, behind the knees and on the buttocks. It has a hereditary component and runs in families.

Infants can develop an eczema variant called cradle cap — with a greasy, itchy flaky scalp. This can extend to the eyebrows, face and the trunk. Although the condition is self limiting and harmless, the physical appearance can be distressing to the family.

Irritant contact eczema can be distinguished from the atopic form by the typical distribution and family history. It occurs as an immediate or delayed reaction to contact with an allergen. It may be nickel in the safety pins used on clothes, fashion jewellery containing unacceptable metals or colouring, plants in the garden or chemicals in the workplace. Makeup or face powder can cause an idiosyncratic reaction. Sometimes the contact eczema may be photosensitive and flare up only when the skin is exposed to sunlight. Eczema caused by a specific chemical or disease process can be cured if the underlying factor is removed.

Bacterial, viral or fungal infection of the skin, or infestations with skin parasites like scabies or body lice can cause secondary itching and eczema. Poor blood circulation to the legs as a result of varicose veins can lead to itching and discolouration, particularly near the ankles. These forms are totally curable and tend to disappear forever when the infection is treated or the blood circulation is improved.

Unfortunately most varieties of eczema do not fall into the curable category. They recur time and again, and sometimes become self perpetuating as scratching and picking the skin becomes a habit.

Dry skin aggravates eczema. Therefore it pays to keep the skin moist and oiled. Coconut oil can be applied to the skin half an hour before a bath. A teaspoon can also be added to the bath water. Mild and non-perfumed soaps should be used. The body should not be scrubbed with a loofah. The skin should be patted dry, and not wiped. After the bath a non-greasy oil or lotion can be applied.

There are several baby oils, aloe vera preparations, ceramide (a natural oil in the skin) and vaseline-based creams available in the market. If it says “non greasy” it means that it is unlikely to stain your clothes.
Traditional oils, like coconut, stain the clothes and that is why they need to be applied before a bath.

Salt water reduces eczema. People who immerse themselves regularly in the sea improve gradually over a period of time.

Detergents are widely used to wash clothes. Most contain chemicals like sodium lauryl sulphate, which remain behind in the clothes in small quantities. They can penetrate the skin when sweating occurs, aggravating eczema. Commercially available “hypoallergic” or “doctor tested and recommended” detergents have unsubstantiated claims and have not been proven safer. Since clothes have to be washed and kept clean, it is preferable to use non-allergic soaps made from neem oil. They are marketed in India by the government run chain of khadi stores.

Eczema disappears when steroid creams are applied. These should be used under medical supervision for a short period, till the symptoms subside. After that antihistamines (to control itching) and moisturisers should be continued. Patients, unfortunately, purchase and apply the ointments themselves. Prolonged use of topical corticosteroids causes side effects like thinning of the skin and secondary bacterial or fungal infection. Small quantities of the more potent steroid creams can become absorbed in the body through the skin. If they are inadvertently applied to the eye, cataracts and glaucoma can result.

Specific pinprick allergy testing can be done to identify allergens. Without this, drastic potentially harmful changes should not be made to the diet (particularly a child’s) presuming that the eczema is precipitated by allergies to milk, fish or certain vegetables.

Sources: The Telegraph (Kolkata, India)

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Amebic Liver Abscess

Alternative Names:Hepatic amebiasis; Extraintestinal amebiasis; Abscess – amebic liver
Definition :
Amebic liver abscess is a collection of pus in the liver caused by an intestinal parasite.Organisms that carry disease can travel through the blood stream into the liver and form an abscess, a collection of infected tissue and pus.

YOU MAY CLICK TO SEE THE PICTURES
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Amebic liver abscess is an extraintestinal manifestation caused by a protozoa Entamoeba histolytica. The disease spreads through the ingestion of cysts in fecally-contaminated food or water. The infection is worldwide, most common in tropical areas usually in developing countries where poor sanitation exist.

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This is an Abscess (walled area of infection/pus) in the liver that is caused by the organism Entamoeba histolytica.  It is common in tropical and subtropical areas (southeast Asia, Africa, India, Latin America).  In the U.S., it is most common in young Hispanic adults.  It is also seen in those with recent travels to tropical areas, homosexuals, and among the inhabitants of institutions for the mentally ill.

Causes :-
Travel to endemic areas (as above), where the cysts of the parasite may be ingested by consuming water or food contaminated by fecal matter.
Amebic liver abscess is caused by Entamoeba histolytica, the same organism that causes amebiasis, an intestinal infection. The organism is carried by the blood from the intestines to the liver.

The disease spreads through ingestion of cysts in fecally-contaminated food or water, use of human waste as fertilizer, and person-to-person contact.

The infection occurs worldwide, but is most common in tropical areas where crowded living conditions and poor sanitation exist. Africa, Latin America, Southeast Asia, and India have significant health problems associated with this disease.

Risk Factors :-
*Malnutrition
*Old age
*Pregnancy
*Steroid use
*Cancer
*Immunosuppression
*Alcoholism
*Recent travel to a tropical region
*Homosexuality, particularly in males

Symptoms :-
There may or may not be symptoms of intestinal infection. Symptoms may include:
*Right upper abdominal pain
*Right sided chest pain worse on a deep breath
*Intense, continuous, or stabbing pain
*Chills
*Diarrhea precedes infection in 20% of patients
*Fever
*General discomfort, uneasiness, or ill feeling (malaise)
*Jaundice
*Joint pain
*Loss of appetite
*Sweating
*Weight loss
*malaise

Nature of the disease:-
Most infected people, perhaps 90%, are asymptomatic, but this disease has the potential to make the sufferer dangerously ill. It is estimated by the World Health Organization that about 70,000 people die annually worldwide.

Infections can sometimes last for years. Symptoms take from a few days to a few weeks to develop and manifest themselves, but usually it is about two to four weeks. Symptoms can range from mild diarrhoea to dysentery with blood and mucus. The blood comes from amoebae invading the lining of the intestine. In about 10% of invasive cases the amoebae enter the bloodstream and may travel to other organs in the body. Most commonly this means the liver, as this is where blood from the intestine reaches first, but they can end up almost anywhere.

Onset time is highly variable and the average asymptomatic infection persists for over a year. It is theorised that the absence of symptoms or their intensity may vary with such factors as strain of amoeba, immune response of the host, and perhaps associated bacteria and viruses.

In asymptomatic infections the amoeba lives by eating and digesting bacteria and food particles in the gut, a part of the gastrointestinal tract. It does not usually come in contact with the intestine itself due to the protective layer of mucus that lines the gut. Disease occurs when amoeba comes in contact with the cells lining the intestine. It then secretes the same substances it uses to digest bacteria, which include enzymes that destroy cell membranes and proteins. This process can lead to penetration and digestion of human tissues, resulting first in flask-shaped ulcers in the intestine. Entamoeba histolytica ingests the destroyed cells by phagocytosis and is often seen with red blood cells inside when viewed in stool samples. Especially in Latin America,  a granulomatous mass (known as an amoeboma) may form in the wall of the ascending colon or rectum due to long-lasting immunological cellular response, and is sometimes confused with cancer.

Theoretically, the ingestion of one viable cyst can cause an infection.

Diagnosis:
Exams and Tests
Tests that may be done include:
*Abdominal ultrasound
*Abdominal CT scan or MRI
*Complete blood count
*Liver biopsy – rarely done due to high risk of complications
*Liver scan
*Liver function tests
*Serology for amebiasis

Treatment
A medicine called metronidazole (Flagyl) is the usual treatment for liver abscess. Medications such as paromomycin must also be taken to remove intestinal amebiasis to prevent recurrence of the disease.

In rare cases, the abscess may need to be drained to help relieve some of the abdominal pain.

Prognosis:
Without treatment, the abscess may rupture and spread into other organs, leading to death. Persons who receive treatment have a very high chance of a complete cure or having only minor complications.
Possible Complications :In the majority of cases, amoebas remain in the gastrointestinal tract of the hosts. Severe ulceration of the gastrointestinal mucosal surfaces occurs in less than 16% of cases. In fewer cases, the parasite invades the soft tissues, most commonly the liver. Only rarely are masses formed (amoebomas) that lead to intestinal obstruction.

The abscess may rupture into the abdominal cavity, the lining of the lungs, the lungs, or the sac around the heart. The infection can also spread to the brain.

When to Contact a Medical Professional
Call your health care provider if symptoms develop after travel to an area where the disease is known to occur.

Prevention:
To help prevent the spread of amoebiasis around the home :
*Wash hands thoroughly with soap and hot running water for at least 10 seconds after using the toilet or changing a baby’s diaper, and before handling food.
*Clean bathrooms and toilets often; pay particular attention to toilet seats and taps.
*Avoid sharing towels or face washers.

To help prevent infection:
*Avoid raw vegetables when in endemic areas, as they may have been fertilized using human feces.
*Boil water or treat with iodine tablets.
*When traveling in tropical countries where poor sanitation exists, drink purified water and do not eat uncooked vegetables or unpeeled fruit.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://www.nlm.nih.gov/medlineplus/ency/article/000211.htm
http://www.ecureme.com/emyhealth/data/Amebic_Liver_Abscess.asp
http://organizedwisdom.com/Amebic_Liver_Abscess
http://organizedwisdom.com/helpbar/index.html?return=http://organizedwisdom.com/Amebic_Liver_Abscess&url=en.wikipedia.org/wiki/Amoebiasis#Diagnosis_of_human_illness

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Acute Cholecystitis

Alternative Names: Cholecystitis – acute

Definition: Acute cholecystitis is a sudden inflammation of the gallbladder that causes severe abdominal pain.

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You may Click  See also: Chronic cholecystitis

Causes :-
In 90% of cases, acute cholecystitis is caused by gallstones in the gallbladder. Severe illness, alcohol abuse and, rarely, tumors of the gallbladder may also cause cholecystitis.

Acute cholecystitis causes bile to become trapped in the gallbladder. The build up of bile causes irritation and pressure in the gallbladder. This can lead to bacterial infection and perforation of the organ.

Gallstones occur more frequently in women than men. Gallstones become more common with age in both sexes. Native Americans have a higher rate of gallstones.

Symptoms :-

The main symptom is abdominal pain that is located on the upper right side or upper middle of the abdomen. The pain may:

*Be sharp, cramping, or dull
*Come and go
*Spread to the back or below the right shoulder blade
*Be worse after eating fatty or greasy foods
*Occur within minutes of a meal
*Shortness of breath due to pain when inhaling

Other symptoms that may occur include:
*Abdominal fullness
*Clay-colored stools
*Excess gas
*Fever
*Heartburn
*Indigestion
*Nausea and vomiting
*Yellowing of skin and whites of the eyes (jaundice)
*Stiff abdomen muscles, specially on the right side
*Chills

Diagnosis:
Because the symptoms of acute cholecystitis can resembles symptoms of other illness, it is sometimes difficult to diagnose. If doctor suspects Cholecystitis after a carefull physical examination, he or she may perform some of the following tests:-

Blood Test:-
*Amylase and lipase
*Bilirubin
*Complete blood count ( CBC) — may show a higher-than-normal white blood cell count
*Liver function tests

Imaging tests that can show gallstones or inflammation include:

*Abdominal ultrasound
*Abdominal CT scan
*Abdominal x-ray
*Oral cholecystogram
*Gallbladder radionuclide scan
.

Treatment:-

Seek immediate medical attention for severe abdominal pain.

In the emergency room, patients with acute cholecystitis are given fluids through a vein and antibiotics to fight infection.

Although cholecystitis may clear up on its own, surgery to remove the gallbladder (cholecystectomy) is usually needed when inflammation continues or recurs. Surgery is usually done as soon as possible, however some patients will not need surgery right away.

Nonsurgical treatment includes pain medicines, antibiotics to fight infection, and a low-fat diet (when food can be tolerated).

Emergency surgery may be necessary if gangrene (tissue death), perforation, pancreatitis, or inflammation of the common bile duct occurs.

Occasionally, in very ill patients, a tube may be placed through the skin to drain the gallbladder until the patient gets better and can have surgery.

For Alternative Medication  you may click to see:->
*Cholecystitis as related to Herbal Medicine :
*An alternative approach to acute cholecystitis :
*Acute cholecystitis – Traditional Chinese Medicine, Nature therapy :

Prognosis:-Patients who have surgery to remove the gallbladder are usually do very well.

Possible Complications:-
*Empyema (pus in the gallbladder)
*Gangrene (tissue death) of the gallbladder
*Injury to the bile ducts draining the liver (a rare complication of cholecystectomy)
*Pancreatitis
*Peritonitis (inflammation of the lining of the abdomen)

When to Contact a Medical Professional:

*Call your health care provider if severe abdominal pain persists.
*Call for an appointment with your health care provider if symptoms of cholecystitis recur after an acute episode.

Prevention :
Removal of the gallbladder and gallstones will prevent further attacks. Follow a low-fat diet if you are prone to gallstone attacks.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://www.nlm.nih.gov/medlineplus/ency/article/000264.htm

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Addison’s Disease

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Definition:
Addison’s disease is a disorder that results when your body produces insufficient amounts of certain hormones produced by your adrenal glands. In Addison’s disease, your adrenal glands produce too little cortisol, and often insufficient levels of aldosterone as well.

click & see the pictures

Also called adrenal insufficiency or hypocortisolism, Addison’s disease can occur at any age, but is most common in people ages 30 to 50. Addison’s disease can be life-threatening.

Symptoms:

Addison’s disease symptoms usually develop slowly, often over several months, and may include:
*Muscle weakness and fatigue
*Weight loss and decreased appetite
*Darkening of your skin (hyperpigmentation)
*Low blood pressure, even fainting
*Salt craving
*Low blood sugar (hypoglycemia)
*Nausea, diarrhea or vomiting
*Muscle or joint pains
*Irritability
*Depression

Acute adrenal failure (addisonian crisis)
Sometimes, however, the signs and symptoms of Addison’s disease may appear suddenly. In acute adrenal failure (addisonian crisis), the signs and symptoms may also include:

*Pain in your lower back, abdomen or legs
*Severe vomiting and diarrhea, leading to dehydration
*Low blood pressure
*Loss of consciousness
*High potassium (hyperkalemia)

Causes :
Your adrenal glands are located just above each of your two kidneys. These glands are part of your endocrine system, and they produce hormones that give instructions to virtually every organ and tissue in your body.

Your adrenal glands are composed of two sections. The interior (medulla) produces adrenaline-like hormones. The outer layer (cortex) produces a group of hormones called corticosteroids, which include glucocorticoids, mineralocorticoids and male sex hormones (androgens).

Some of the hormones the cortex produces are essential for life — the glucocorticoids and the mineralocorticoids.

*Glucocorticoids. These hormones, which include cortisol, influence your body’s ability to convert food fuels into energy, play a role in your immune system’s inflammatory response and help your body respond to stress.

*Mineralocorticoids. These hormones, which include aldosterone, maintain your body’s balance of sodium and potassium and water to keep your blood pressure normal.

*Primary adrenal insufficiency: Addison’s disease occurs when the cortex is damaged and doesn’t produce its hormones in adequate quantities. Doctors refer to the condition involving damage to the adrenal glands as primary adrenal insufficiency.

The failure of your adrenal glands to produce adrenocortical hormones is most commonly the result of the body attacking itself (autoimmune disease). For unknown reasons, your immune system views the adrenal cortex as foreign, something to attack and destroy.

Other causes of adrenal gland failure may include:
*Tuberculosis
*Other infections of the adrenal glands
*Spread of cancer to the adrenal glands
*Bleeding into the adrenal glands

Secondary adrenal insufficiency
Adrenal insufficiency can also occur if your pituitary gland is diseased. The pituitary gland makes a hormone called adrenocorticotropic hormone (ACTH), which stimulates the adrenal cortex to produce its hormones. Inadequate production of ACTH can lead to insufficient production of hormones normally produced by your adrenal glands, even though your adrenal glands aren’t damaged. Doctors call this condition secondary adrenal insufficiency.

Another more common possible cause of secondary adrenal insufficiency occurs when people who take corticosteroids for treatment of chronic conditions, such as asthma or arthritis, abruptly stop taking the corticosteroids.

Addisonian crisis
If you have untreated Addison’s disease, an addisonian crisis may be provoked by physical stress, such as an injury, infection or illness.

When to seek medical advice:
See your doctor if you have signs and symptoms that commonly occur in people with Addison’s disease. Most people with this condition experience darkening areas of skin (hyperpigmentation), severe fatigue, unintentional weight loss, and gastrointestinal problems, such as nausea, vomiting and abdominal pain. Dizziness or fainting, salt cravings, and muscle or joint pains also are common.

Your doctor can determine whether Addison’s disease or some other medical condition may be causing these problems.

Tests and diagnosis:-

Your doctor will talk to you first about your medical history and your signs and symptoms. If your doctor thinks that you may have Addison’s disease, you may undergo some of the following tests:

*Blood test. Measuring your blood levels of sodium, potassium, cortisol and ACTH gives your doctor an initial indication of whether adrenal insufficiency may be causing your signs and symptoms. A blood test can also measure antibodies associated with autoimmune Addison’s disease.

*ACTH stimulation test. This test involves measuring the level of cortisol in your blood before and after an injection of synthetic ACTH. ACTH signals your adrenal glands to produce cortisol. If your adrenal glands are damaged, the ACTH stimulation test shows that your output of cortisol in response to synthetic ACTH is blunted or nonexistent.

*Insulin-induced hypoglycemia test. Occasionally, doctors suggest this test if pituitary disease is a possible cause of adrenal insufficiency (secondary adrenal insufficiency). The test involves checking your blood sugar (blood glucose) and cortisol levels at various intervals after an injection of insulin. In healthy people, glucose levels fall and cortisol levels increase.

*Imaging tests. Your doctor may have you undergo a computerized tomography (CT) scan of your abdomen to check the size of your adrenal glands and look for other abnormalities that may give insight to the cause of the adrenal insufficiency. Your doctor may also suggest a CT scan or MRI scan of your pituitary gland if testing indicates you have secondary adrenal insufficiency.

Treatments and drugs:-
If you receive an early diagnosis of Addison’s disease, treatment may involve taking prescription corticosteroids. Because your body isn’t producing sufficient steroid hormones, your doctor may have you take one or more hormones to replace the deficiency. Cortisol is replaced using hydrocortisone (Cortef), prednisone or cortisone acetate. Fludrocortisone (Florinef) replaces aldosterone, which controls your body’s sodium and potassium needs and keeps your blood pressure normal.

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You take these hormones orally in daily doses that mimic the amount your body normally would make, thereby minimizing side effects. If you’re facing a stressful situation, such as an operation, an infection or a minor illness, your doctor will suggest a temporary increase in your dosage. If you’re ill with vomiting and can’t retain oral medications, you may need corticosteroid injections.

In addition, your doctor may recommend treating androgen deficiency with an androgen replacement called dehydroepiandrosterone. Some studies indicate that, for women with Addison’s disease, androgen replacement therapy may improve overall sense of well-being, libido and sexual satisfaction.

You may click to learn more about:-
-> Natural Addison’s Disease Treatment
->Natural Help for Addison’s Disease
->AN ALTERNATIVE AND COMPLEMENTARY MEDICINE RESOURCE GUIDE
->Treatment For Addison’s Disease – Herbal Remedies – Natural Cures

Addisonian crisis
An addisonian crisis is a life-threatening situation that results in low blood pressure, low blood levels of sugar and high blood levels of potassium. This situation requires immediate medical care. Treatment typically includes intravenous injections of:

*Hydrocortisone
*Saline solution
*Sugar (dextrose

Coping and support:-

These steps may help you cope better with a medical emergency if you have Addison’s disease:

Carry a medical alert card and bracelet at all times. In the event you’re incapacitated, emergency medical personnel know what kind of care you need.

Keep extra medication handy. Because missing even one day of therapy may be dangerous, it’s a good idea to keep a small supply of medication at work, at a vacation home and in your travel bag, in the event you forget to take your pills. Also, have your doctor prescribe a needle, syringe and injectable form of corticosteroids to have with you in case of an emergency.

Stay in contact with your doctor. Keep an ongoing relationship with your doctor to make sure that the doses or replacement hormones are adequate but not excessive. If you’re having persistent problems with your medications, you may need adjustments in the doses or timing of the medications.

Sources:http://www.mayoclinic.com/health/addisons-disease/DS00361/DSECTION=symptoms

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Acute Bilateral Obstructive Uropathy

Urinary system
Image via Wikipedia

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Alternative Names: Urethral obstruction; Acute urethral obstruction; Obstructive uropathy – bilateral – acute

Definition:Acute bilateral obstructive uropathy is a sudden blockage of the flow of urine from both kidneys. The kidneys continue to produce urine in the normal manner, but because urine does not drain properly, the kidneys start to swell. You may click to See also:

*Cronic unilateral obstructive uropathy

*Acute unilateral obstructive uropathy

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Causes: In men, acute bilateral obstructive uropathy is most often a result of an enlarged prostate. Other causes in men include: *Bladder cancer *Kidney stones *Prostate cancer Acute bilateral obstructive uropathy is much less common in women, but may be due to: *Bladder cystocele *Cervical cancer *Injury from surgery involving the reproductive organs *Pregnancy Other causes in men and women include: *Blood clots *Neurogenic bladder *Other rare retroperitoneal processes *Papillary necrosis *Posterior urethral valves in infant boys Acute bilateral obstructive uropathy occurs in about 5 out of 10,000 people. You may click to enlarge the pictures and see:-> *Female Bladder Catheterization..…..>. *Male Bladder catheterization…..……> *Female Urinary Tract…………………………..> *Male Urinary Tract………………………………>

 

Symptoms: *Abnormal urine flow — dribbling at the end of urination *Blood in the urine *Burning or stinging with urination *Decrease in the force of the urinary stream, stream small and weak *Decreased urine output (may be less than 10 mL per day) *Feeling of incomplete emptying of the bladder *Fever *Frequent strong urge to urinate *Recent increase in blood pressure *Leakage of urine (incontinence) *Nausea and vomiting *Need to urinate at night *Sudden flank pain or pain on both sides *Urinary hesitancy *Urine, abnormal color

 

.Diagnosis: Physical Exams : The doctor will perform a physical exam. The exam may show: *Large and full bladder *Swollen or tender kidneys *Enlarged prostate (men) *There may be signs of chronic kidney failure, high blood pressure, and infection. Fever is common with an infection. Tests that may be done include: *Arterial blood gas and blood chemistries *Basic metabolic panel — will reveal kidney function and electrolyte balance *Blood BUN *Creatinine clearance *Complete blood count *Potassium test *Serum creatinine test *Urinalysis and a urine culture (clean catch) *Ultrasound of the bladder *Uroflowmetry The following tests may show hydronephrosis (swelling of kidneys): *IVP *Renal scan *Ultrasound of the kidneys *Abdominal CT scan This disease may also alter the results of the following tests: *Creatinine – urine *Radionuclide cystogram Treatment: The goal of treatment is to relieve the blockage, which will allow urine to drain from the urinary tract. You may need to stay in a hospital for a short while. Short-term treatment may include: *Antibiotics and other medications to treat symptoms *Catheterization– the placement of a tube into the body to drain urine (See: Urinary catheters) Long-term treatment involves correcting the cause of the blockage and this may involve: *Surgery such as transurethral resection of the prostate (TURP) *Laser or heat therapy to shrink the prostate if the problem is due to an enlarged prostate Surgery may also be needed for other disorders that cause blockage of the urethra or bladder neck.

 

Prognosis: If the acute obstruction is quickly relieved, symptoms usually go away within hours to days. If untreated, the disorder causes progressive damage to the kidneys. It may eventually lead to high blood pressure or kidney failure.

Possible Complications : *Acute kidney failure *Chronic bilateral obstructive uropathy *High blood pressure *Reflux nephropathy *Urinary tract infection *Urinary retention or incontinence

When to Contact a Medical Professional : Call your health care provider if you have decreased urine output, difficulty urinating, flank pain, or other symptoms of acute bilateral obstructive uropathy.

Prevention You may not be able to prevent this condition. Routine annual physicals with a primary care doctor are recommended. If your doctor finds you have acute obstructive uropathy, you should be referred to the nearest emergency room and seen by a urologist.

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources: http://www.umm.edu/ency/article/000485.htm http://www.nlm.nih.gov/medlineplus/ency/article/000485.htm

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