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Early Childhood Stress Can Have a Lingering Effect on Your Health

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Stressful experiences in early childhood can have long-lasting impacts on children‘s health that can persist well beyond the resolution of the situation.
……..CLICK & SEE
A study revealed impaired immune function in adolescents who experienced either physical abuse or time in an orphanage as youngsters. Even though their environments had changed, physiologically they were still responding to stress. How the immune system develops is very much influenced by early environment.

The researchers looked for high levels of antibodies against the common and usually latent herpes simplex virus type 1 (HSV-1). While roughly two-thirds of Americans carry this virus, which causes cold sores and fever blisters, people with healthy immune systems are able to keep the virus in check and rarely if ever have symptoms. However, people with weakened immune systems can have trouble suppressing HSV-1 and produce antibodies against the activated virus.

Adolescents who had experienced physical abuse or stressful home environments as children had higher levels of HSV-1 antibodies, showing their immune systems were compromised.

Resources:

Science Daily January 28, 2009

Proceedings of the National Academy of Sciences February 2, 2009

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Stress a Trigger for Skin Disease

Researchers from University of Medicine Berlin and McMaster University in Canada have found that stress may activate immune cells in the skin, leading to inflammatory skin disease.

..CLICK & SEE

This cross talk between stress perception, which involves the brain, and the skin is mediated through the “brain-skin connection”.

The immune cells in skin can over-react, resulting in inflammatory skin diseases like atopic dermatitis and psoriasis.

Study leader Petra Arck hypothesized that stress could exacerbate skin disease by increasing the number of immune cells in the skin.

The researcher said that the team exposed mice to sound stress, and found that the stress challenge resulted in higher numbers of mature white blood cells in the skin.

Moreover, blocking the function of two proteins that attract immune cells to the skin, LFA-1 and ICAM-1, prevented the stress-induced increase in white blood cells in the skin.

Based on their observations, the researchers came to the conclusion that stress activates immune cells, which in turn are central in initiating and perpetuating skin diseases. The study by Arck appears in the November issue of The American Journal of Pathology.

Sources: The Times Of India

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Yellow Fever

TEM micrograph: Multiple yellow fever virions ...
Image via Wikipedia

Definition:
Yellow fever (also called yellow jack, black vomit or sometimes American Plague) is an acute viral disease. It is an important cause of hemorrhagic illness in many African and South American countries despite existence of an effective vaccine. The yellow refers to the jaundice symptoms that affect some patients.It is a viral infection transmitted by mosquitoes.

Yellow fever is a viral hemorrhagic fever caused by the yellow fever virus. The yellow fever virus is a single-stranded enveloped virus that belongs to the flavivirus group. The disease can result in mild symptoms or severe illness and death (mortality rate 5-70%). Yellow fever derives its name from the yellowing of the skin and whites of the eyes (jaundice) that occur in some people infected with the virus. Jaundice is caused by the presence of bile pigment (bilirubin) in the bloodstream and results from damage to liver cells (hepatocytes) during severe infection.…click & see

The yellow fever virus infects mainly monkeys and humans: monkeys are the animal reservoir. Infection is transmitted from human to human, monkey to monkey, monkey to human, and human to monkey by daytime-biting mosquitos. Several species of Aedes and Haemoagogus mosquitos can serve as vectors, transmitting the virus during a blood meal.

Three types of transmission cycles exist for yellow fever: sylvatic (jungle), intermediate and urban. Although all three transmission cycles occur in Africa, only sylvatic and urban transmission cycles occur in South America.

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pictures

Sylvatic yellow fever (monkey to human)

* Occurs in monkeys infected by wild mosquitos in tropical rainforests

* Infected monkeys pass the virus to mosquitos during feeding
* Infected wild mosquitos bite humans entering the rainforest (accidental infection)

Intermediate yellow fever (monkey to human; human to monkey)
* Small-scale epidemics that occur in humid or semi-humid grasslands of Africa
* Separate villages experience simultaneous infections transmitted by semi-domestic mosquitos that infect both monkey and human hosts
* Most common type of outbreak in Africa

Urban yellow fever (human to human)

* Large epidemics occurring when the virus is introduced into high human population areas by migrants
* Domestic mosquitos of one species (Aedes aegypti) transmit the virus from person to person
* Monkeys are not involved in transmission
* Outbreaks spread from one source to cover a wide area

Yellow fever has been a source of several devastating epidemics. Yellow fever epidemics broke out in the 1700s in Italy, France, Spain, and England. 300,000 people are believed to have died from yellow fever in Spain during the 19th century. French soldiers were attacked by yellow fever during the 1802 Haitian Revolution; more than half of the army perished from the disease. Outbreaks followed by thousands of deaths occurred periodically in other Western Hemisphere locations until research, which included human volunteers (some of whom died), led to an understanding of the method of transmission to humans (primarily by mosquitos) and development of a vaccine and other preventive efforts in the early 20th century.

Despite the costly and sacrificial breakthrough research by Cuban physician Carlos Finlay, American physician Walter Reed, and many others over 100 years ago, unvaccinated populations in many developing nations in Africa and Central and South America continue to be at risk. As of 2001, the World Health Organization (WHO) estimates that yellow fever causes 200,000 illnesses and 30,000 deaths every year in unvaccinated populations.

Causes :-
Yellow fever is caused by a small virus that is spread by the bite of mosquitoes. This disease is common in South America and in sub-Saharan Africa.

……………You may click to see pictures of  mosquitoes  causing yellow fever.….

Anyone can get yellow fever, but the elderly have a higher risk of severe infection. If a person is bitten by an infected mosquito, symptoms usually develop 3 – 6 days later.

Yellow fever has three stages:
1.Early stage: Headache, muscle aches, fever, loss of appetite, vomiting, and jaundice are common. After approximately 3 – 4 days, often symptoms go away briefly (remission).

2.Period of remission: After 3 – 4 days, fever and other symptoms go away. Most people will recover at this stage, but others may move onto the third, most dangerous stage (intoxication stage) within 24 hours.

3.Period of intoxication: Multi-organ dysfunction occurs. This includes liver and kidney failure, bleeding disorders/hemorrhage, and brain dysfunction including delirium, seizures, coma, shock, and death.

Symptoms :-
*Arrhythmias, heart dysfunction
*Bleeding (may progress to hemorrhage)
*Coma
*Decreased urination
*Delirium
*Fever
*Headache
*Jaundice
*Muscle aches (myalgia)
*Red eyes, face, tongue
*Seizures
*Vomiting
*Vomiting blood

Although viral replication begins in cells at the site of the mosquito bite, symptoms of infection are not usually noted for a period of three to six days when the acute phase of infection presents. Acute yellow fever infection is characterized by high fever, muscle pain, backache, headache, shivers, loss of appetite, nausea and/or vomiting. Most people infected improve after three to four days.

However, within 24 hours of the disappearance of symptoms, up to 15% of those infected enter a toxic phase during which fever resumes, and the yellow fever virus quickly spreads to the kidneys, lymph nodes, spleen, bone marrow and liver. Liver invasion of one of the last stages to occur: as the liver is increasingly damaged, patients develop jaundice as bilirubin is released from damaged liver cells, experience abdominal pain and vomiting, and develop coagulopathies (inability of the blood to clot) characterized by bleeding from the mouth, nose, eyes and stomach, and presence of blood in vomit and stool. Up to 50% of people who enter the toxic phase die within two weeks of infection.

Diagnosis:-
Yellow fever may be difficult to diagnose, especially during the early stages, and may be confused with malaria, typhoid, other hemorrhagic fevers (dengue, Rift Valley, Venezuelan, Bolivian, Argentine, Lassa, Crimean-Congo, Marburg and Ebola), rickettsial infection, leptospirosis, viral hepatitis, other causes of liver failure and toxic hepatitis (e.g. carbon-tetrachloride poisoning).

Exams and Tests
A person with advanced yellow fever may show signs of liver failure, renal failure, and shock.

If you have symptoms of yellow fever, tell your doctor if you have traveled to areas where the disease is known to thrive. Blood tests can confirm the diagnosis.

Treatment :-

There is no specific treatment for yellow fever. Treatment for symptoms can include:

*Blood products for severe bleeding
*Dialysis for kidney failure
*Fluids through a vein (intravenous fluids)

The treatment for yellow fever is supportive: control of fever, fluids to treat dehydration, and intensive support related to organ damage.

The World Health Organization estimates 200,000 cases of yellow fever per year with approximately 30,000 deaths.

CLICK TO READ ..>: Early sign of yellow fever could lead to new treatment

Prognosis: :-

Yellow fever ranges in severity. Severe infections with internal bleeding and fever (hemorrhagic fever) are deadly in up to half of cases.

Historical reports have claimed a mortality rate of between 1 in 17 (5.8%) and 1 in 3 (33%). CDC has claimed that case-fatality rates from severe disease range from 15% to more than 50%. The WHO factsheet on yellow fever, updated in 2001, states that 15% of patients enter a “toxic phase” and that half of that number die within ten to fourteen days, with the other half recovering

Possible Complications :-

*Coma
*Death
*Disseminated intravascular coagulation (DIC)
*Kidney failure
*Liver failure
*Parotitis
*Secondary bacterial infections
*Shock

Prevention :-

If you will be traveling to an area where yellow fever is common:

*Sleep in screened housing
*Use mosquito repellents
*Wear clothing that fully covers your body
*There is an effective vaccine against yellow fever. Ask your doctor at least 10 – 14 days before traveling if you should be *vaccinated against yellow fever.

In 1937, Max Theiler, working at the Rockefeller Foundation, developed a safe and highly efficacious vaccine for yellow fever that gives a ten-year or more immunity from the virus. The vaccine consists of a live, but attenuated, virus called 17D. The 17D vaccine has been used commercially since the 1950s. The mechanisms of attenuation and immunogenicity for the 17D strain are not known. However, this vaccine is very safe, with few adverse reactions having been reported and millions of doses administered, and highly effective with over 90% of vaccinees developing a measurable immune response after the first dose.

click to see the picture

Although the vaccine is considered safe, there are risks involved. The majority of adverse reactions to the 17D vaccine result from allergic reaction to the eggs in which the vaccine is grown. Persons with a known egg allergy should discuss this with their physician prior to vaccination. In addition, there is a small risk of neurologic disease and encephalitis, particularly in individuals with compromised immune systems and very young children. The 17D vaccine is contraindicated in infants, pregnant women, and anyone with a diminished immune capacity, including those taking immunosuppressant drugs.

According to the travel clinic at the University of Utah Hospital, the vaccine presents an increased risk of adverse reaction in adults aged 60 and older, with the risk increasing again after age 65, and again after age 70. The reaction is capable of producing multiple organ failure and should be evaluated carefully by a qualified health professional before being administered to the elderly.

Finally, there is a very small risk of more severe yellow fever-like disease associated with the vaccine. This reaction occurs in 1~3 vaccinees per million doses administered. This reaction, called YEL-AVD, causes a fairly severe disease closely resembling yellow fever caused by virulent strains of the virus. The risk factor/s for YEL-AVD are not known, although it has been suggested that it may be genetic. The 2`-5` oligoadenylate synthetase (OAS) component of the innate immune response has been shown to be particularly important in protection from Flavivirus infection. In at least one case of YEL-AVD, the patient was found to have an allelic mutation in a single nucleotide polymorphism (SNP) of the OAS gene. People most at risk of contracting the virus should be vaccinated. Woodcutters working in tropical areas should be particularly targeted for vaccination. Insecticides, protective clothing, and screening of houses are helpful, but not always sufficient for mosquito control; people should always use an insecticide spray while in certain areas. In affected areas, mosquito control methods have proven effective in decreasing the number of cases.

Recent studies have noted the increase in the number of areas affected by a number of mosquito-borne viral infections and have called for further research and funding for vaccines

Current research:-
In the hamster model of yellow fever, early administration of the antiviral ribavirin is an effective early treatment of many pathological features of the disease. Ribavirin treatment during the first five days after virus infection improved survival rates, reduced tissue damage in target organs (liver and spleen), prevented hepatocellular steatosis, and normalized alanine aminotransferase (a liver damage marker) levels. The results of this study suggest that ribavirin may be effective in the early treatment of yellow fever, and that its mechanism of action in reducing liver pathology in yellow fever virus infection may be similar to that observed with ribavirin in the treatment of hepatitis C, a virus related to yellow fever. Because ribavirin had failed to improve survival in a virulent primate (rhesus) model of yellow fever infection, it had been previously discounted as a possible therapy.

In 2007, the World Community Grid launched a project whereby computer modelling of the yellow fever virus (and related viruses), thousands of small molecules are screened for their potential anti-viral properties in fighting yellow fever. This is the first project to utilize computer simulations in seeking out medicines to directly attack the virus once a person is infected. This is a distributed process project similar to SETI@Home where the general public downloads the World Community Grid agent and the program (along with thousands of other users) screens thousands of molecules while their computer would be otherwise idle. If the user needs to use the computer the program sleeps. There are several different projects running, including a similar one screening for anti-AIDS drugs. The project covering yellow fever is called “Discovering Dengue Drugs – Together.” The software and information about the project can be found at: World Community Grid web site

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.
Resources:
http://en.wikipedia.org/wiki/Yellow_fever
http://www.nlm.nih.gov/medlineplus/ency/article/001365.htm
http://microbiology.suite101.com/article.cfm/yellow_fever

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Down With a Cold ?

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At some time or another, everyone — even a robust fitness freak — gets felled by the common cold, developing sniffles, sneezing, puffy eyes, fever, body ache and malaise. Children start to develop colds during their first year, the frequency of which may increase to up to six times a year. This leaves the mothers with the feeling that the child is “always ill”. The average adult gets three to four colds a year.

Almost 40 per cent of outpatient medical consultations in a general practice deals with colds and their complications. This is not surprising, as colds are unavoidable infections. They are caused by viruses, 80 per cent of which belong to the rhinovirus family. Not only are there more than a hundred members in this group alone, but the types also mutate at a rapid rate. This makes immunity practically non-existent, or at best short lived. To make matters worse, there is no vaccine available, except for flu or influenza.

Colds are highly contagious. The spread is rapid as the virus, contained in nasal secretions, can be propelled forcefully into the environment by coughing and sneezing. It can also be transferred from the nose to the hands of infected people. Patients can then transfer the virus to door knobs, telephones, banisters, switches and other such objects. The virus can remain dormant but viable for 18 hours or more until it finds a susceptible host. Any person touching the contaminated surface has a 50 per cent chance of picking up the infection.

Infection increases during the rainy season and winter months. People tend to huddle together under umbrellas or shelters. Windows may be kept closed. The close contact and lack of ventilation provide ideal conditions for the spread of the cold virus. Contrary to popular myths, colds are not aggravated by washing the hair at night, eating ice cream or using air-conditioning.

The infection incubates for a day or two before symptoms appear. It may then last a variable period of time, usually 5-14 days. If there is no recovery within two weeks, there may be secondary bacterial infection and complications like sinusitis, ear infection, bronchitis and pneumonia may have set in.

Smokers develop colds more frequently than non-smokers do. Their colds are more severe, take longer to subside and are more likely to be complicated by secondary infection. This is because the cilia — fine protective hairs that line the respiratory passages — are paralysed by nicotine. They, therefore, clear accumulated mucous sluggishly and inefficiently. Also, smokers’ lungs are likely to be scarred, distorted, have a reduced blood supply and function sub-optimally, making elimination of the infection difficult.

Man has reached the moon but a cure for the common cold remains elusive. We still rely on “grandma’s recommendations” of hot drinks like ginger tea, lime juice with honey, rice gruel and chicken soup. These do soothe the irritated throat. Also, resting helps. It reduces the pain in the muscles and bones. Steam inhalations liquefy the secretions and help them to drain, providing relief.

Stuffed and blocked nasal passages can be cleared with saline (not chemical) nose drops. Aspirin and paracetamol reduce fever and pain. Anti histamines reduce itching in the nose and throat and dry up dripping nasal secretions. The older first-generation anti histamines (Avil, Benadryl) are very effective but they cause sedation. The second-generation non-sedating products (loratidine, cetrizine) are less effective.

Many health supplements are advocated to boost immunity and reduce the frequency and severity of attacks. Many are of doubtful efficacy and have not been studied scientifically. Zinc supplements, however, have been proven to be useful. They can be used as lozenges, syrups or tablets. Not more than 10-15 mg a day of elemental zinc should be taken.

Antibiotics do not work and administering them is futile and inappropriate. They do not shorten the course of the infection. Nor do they prevent complications. Antiviral medications used against the influenza and herpes viruses are ineffective against the rhinovirus. If the cold just refuses to go away and there are no bacterial complications, it may not be a cold at all. It may be an idiosyncratic allergic reaction to something inhaled or ingested from the environment. Mosquito coils, liquid repellents, room fresheners and incense sticks are particularly notorious.

The best advice for someone with a cold — “wait it out”.

Sources: The Telegraph (Kolkata, India)

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Chickenpox

Definition:

Chickenpox, sometimes called varicella, is a viral infection that used to be common among young children before routine immunization. the infection, with its characteristic rash of blisters, is caused by the varicella zoster virus, which also causes herpes zoster. The virus is transmitted in airborne droplets from the coughs and sneezes of infected people or by direct contact with the blisters. You can catch chickenpox from someone with chickenpox or herpes zoster if you are not immune.

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The illness is usually mild in children, but symptoms are more severe in young babies, older adolescents, and adults. chickenpox can also be more serious in people with reduced immunity, such as those with aids.

It is one of the five classical childhood exanthems or rashes, once a cause of significant morbidity and mortality, but now chiefly of historical importance. Formerly one of the childhood infectious diseases caught by and survived by almost every child, its incidence had been reduced since the introduction and use of a varicella vaccine in 1995 in the U.S. and Canada to inoculate against the disease. Areas such as England, where the vaccine is not mandated, have increasing prevalence rates for chickenpox. Chickenpox is caused by the varicella-zoster virus (VZV), also known as human herpes virus 3 (HHV-3), one of the eight herpes viruses known to affect humans. It starts with conjunctival and catarrhal symptoms and then characteristic spots appearing in two or three waves, mainly on the body and head rather than the hands and becoming itchy raw pox (pocks), small open sores which heal mostly without scarring.

Chickenpox has a 10-21 day incubation period and is highly contagious through physical contact two days before symptoms appear. Following primary infection there is usually lifelong protective immunity from further episodes of chickenpox.

Chickenpox is rarely fatal (usually from varicella pneumonia), with pregnant women and those with a suppressed immune system being more at risk. Pregnant women not known to be immune and who come into contact with chickenpox may need urgent treatment as the virus can cause serious problems for the fetus. This is less of an issue after 20 weeks.

The most common complication of chicken pox is shingles; this is most frequently a late effect.

Causes:

In a typical scenario, a young child is covered in pox and out of school for a week. The first half of the week the child feels miserable from intense itching; the second half from boredom. Since the introduction of the chickenpox vaccine, classic chickenpox is much less common.

Chickenpox is extremely contagious, and can be spread by direct contact, droplet transmission, and airborne transmission. Even those with mild illness after the vaccine may be contagious

Signs and symptoms:
The symptoms of chickenpox appear 1-3 weeks after infection. In children, the illness often starts with a mild fever or headache; in adults, there may be more pronounced flulike symptoms. as infection with the virus progresses, the following symptoms usually become apparent:

· Rash in the form of crops of tiny red spots that rapidly turn into itchy, fluid-filled blisters. within 24 hours the blisters dry out, forming scabs. successive crops occur for 1-6 days. The rash may be widespread or consist of only a few spots, and it can occur anywhere on the head or body.

· Sometimes, discomfort during eating caused by spots in the mouth that have developed into ulcers.

A person is contagious from about 2 days before the rash first appears until it crusts over it about 10-14 days.

Itis a highly contagious disease that spreads from person to person by direct contact or through the air from an infected person’s coughing or sneezing. Touching the fluid from a chickenpox blister can also spread the disease. A person with chickenpox is contagious from one to two days before the rash appears until all blisters have formed scabs. This may take five to 10 days. It takes from 10-21 days after contact with an infected person for someone to develop chickenpox.

The chickenpox lesions (blisters) start as a two to four millimeter red papule which develops an irregular outline (a rose petal). A thin-walled, clear vesicle (dew drop) develops on top of the area of redness. This “dew drop on a rose petal” lesion is very characteristic for chickenpox. After about eight to 12 hours the fluid in the vesicle gets cloudy and the vesicle breaks leaving a crust. The fluid is highly contagious, but once the lesion crusts over, it is not considered contagious. The crust usually falls off after seven days sometimes leaving a crater-like scar. Although one lesion goes through this complete cycle in about seven days, another hallmark of chickenpox is the fact that new lesions crop up every day for several days. Therefore, it may take about a week until new lesions stop appearing and existing lesions crust over. Children are not to be sent back to school until all lesions have crusted over.

Chickenpox is highly contagious and is spread through the air when infected people cough or sneeze, or through physical contact with fluid from lesions on the skin. Zoster, also known as shingles, is a reactivation of chickenpox and may also be a source of the virus for susceptible children and adults. It is not necessary to have physical contact with the infected person for the disease to spread. Those infected can spread chickenpox before they know they have the disease – even before any rash develops. In fact, people with chickenpox can infect others from about two days before the rash develops until all the sores have crusted over, usually four to five days after the rash starts.

Possible Complications:

*Women who get chickenpox during pregnancy are at risk for congenital infection of the fetus.

*Newborns are at risk for severe infection, if they are exposed and their mothers are not immune.

*A secondary infection of the blisters may occur.

*Encephalitis is a serious, but rare complication.

*Reye’s syndrome, pneumonia, myocarditis, and transient arthritis are other possible complications of chickenpox

*Cerebellar ataxia may appear during the recovery phase or later. This is characterized by a very unsteady walk.
The most common complication of chickenpox is bacterial infection of the blisters due to scratching. other complications include pneumonia, which is more common in adults, and rarely inflammation of the brain. newborn babies and people with reduced immunity are at higher risk of complications. Rarely, if a woman develops chickenpox in early pregnancy, the infection may result in fetal abnormalities.

Later in life, chickenpox viruses remaining dormant in the nerves can reactivate, causing shingles.

Secondary infections, such as inflammation of the brain, can occur in immunocompromised individuals. This is more dangerous with shingles.

Congenital defects in babies:
These may occur if the child’s mother was exposed to the zoster virus during pregnancy. Effects on the fetus may be minimal in nature but physical deformities range in severity from under developed toes and fingers, to severe anal and bladder malformation. Possible problems include:

*Damage to brain: encephalitis, microcephaly, hydrocephaly, aplasia of brain

*Damage to the eye (optic stalk, optic cap, and lens vesicles), microphthalmia, cataracts, chorioretinitis, optic atrophy

*Other neurological disorder: damage to cervical and lumbosacral spinal cord, motor/sensory deficits, absent deep tendon reflexes, anisocoria/Horner’s syndrome

*Damage to body: hypoplasia of upper/lower extremities, anal and bladder sphincter dysfunction

*Skin disorders: (cicatricial) skin lesions, hypopigmentation

Diagnosis:
Chickenpox can usually be diagnosed from the appearance of the rash. Children with mild infections do not need to see a doctor, and rest and simple measures to reduce fever are all that are needed for a full recovery. calamine lotion may help relieve itching. To prevent skin infections, keep fingernails short and avoid scratching. people at risk of severe attacks, such as babies, older adolescents, adults, and people with reduced immunity, should see their doctor immediately. An antiviral drug may be given to limit the effect of the infection, but it must be taken in the early stages of the illness in order to be effective.

Prognosis and treatment:
Children who are otherwise healthy usually recover within 10-14 days from the onset of the rash, but they may have permanent scars where blisters have become infected with bacteria and then been scratched. Adolescents, adults, and people with reduced immunity take longer to recover from chickenpox.

Chickenpox infection tends to be milder the younger a child is and symptomatic treatment, with a little sodium bicarbonate in baths or antihistamine medication to ease itching, and paracetamol (acetaminophen) to reduce fever, are widely used. Ibuprofen can also be used on advice of a doctor. However, aspirin or products containing aspirin must not be given to children with chickenpox (or any fever-causing illness), as this risks causing the serious and potentially fatal Reye’s Syndrome.

There is no evidence to support the effectiveness of topical application of calamine lotion, a topical barrier preparation containing zinc oxide in spite of its wide usage and excellent safety profile.

It is important to maintain good hygiene and daily cleaning of skin with warm water to avoid secondary bacterial infection. Infection in otherwise healthy adults tends to be more severe and active; treatment with antiviral drugs (e.g. acyclovir) is generally advised. Patients of any age with depressed immune systems or extensive eczema are at risk of more severe disease and should also be treated with antiviral medication. In the U.S., 55 percent of chickenpox deaths are in the over-20 age group, even though they are a tiny fraction of the cases.

In most cases, it is enough to keep children comfortable while their own bodies fight the illness. Oatmeal baths in lukewarm water provide a crusty, comforting coating on the skin. An oral antihistamine can help to ease the itching, as can topical lotions. Lotions containing antihistamines are not proven more effective. Trim the fingernails short to reduce secondary infections and scarring.

Safe antiviral medicines have been developed. To be effective, they usually must be started within the first 24 hours of the rash. For most otherwise healthy children, the benefits of these medicines may not outweigh the costs. Adults and teens, at risk for more severe symptoms, may benefit if the case is seen early in its course

In addition, for those with skin conditions (such as eczema or recent sunburn), lung conditions (such as asthma), or those who have recently taken steroids, the antiviral medicines may be very important. The same is also true for adolescents and children who must take aspirin on an ongoing basis.

Some doctors also give antiviral medicines to people in the same household who subsequently come down with chickenpox. Because of their increased exposure, they would normally experience a more severe case of chickenpox.

DO NOT USE ASPIRIN for someone who may have chickenpox. Use of aspirin has been associated with Reyes Syndrome. Ibuprofen has been associated with more severe secondary infections. Acetaminophen may be used.

Click for Ayurvedic medication of Chickenpox….…(1).…….(2)……..(3).…..(4)
Click for Homeopathic Remedies of Chicken Pox …….(1).…..(2)...(3)..…..(4)

Prevention:
Once you catch chickenpox, the virus usually stays in your body forever. You probably will not get chickenpox again, but the virus can cause shingles in adults. A chickenpox vaccine can help prevent most cases of chickenpox, or make it less severe if you do get it.

One attack of chickenpox gives lifelong immunity to the disease. However, the varicella zoster virus remains dormant within nerve cells and may reactivate years later, causing herpes zoster. Immunization against chickenpox is now routine for babies aged 12-18 months and is recommended for children aged 11-12 years who have neither had chickenpox nor been immunized.

Vaccination:

A varicella vaccine has been available since 1995 to inoculate against the disease. Some countries require the varicella vaccination or an exemption before entering elementary school. Protection is not lifelong and further vaccination is necessary five years after the initial immunization.

In the UK, varicella antibodies are measured as part of the routine of prenatal care, and by 2005 all NHS healthcare personnel had determined their immunity and been immunised if they were non-immune and have direct patient contact. Population-based immunization against varicella is not otherwise practiced in the UK, because of lack of evidence of lasting efficacy or public health benefit.

Vaccination reactions:
Common and mild reactions following vaccination may include:

*Fever of 101.9 (38.9 C) up to 42 days after injection

*Soreness, itching at the site of injection within 2 days

*Rash occurring at site of injection anywhere form 8 to 19 days after injection. If this happens you are considered contagious.

*Rash on other parts of body anywhere from 5 to 26 days after injection. If this happens you are considered contagious.

Fever and discomfort may be lessened by taking medication containing paracetamol (aka acetaminophen, such as Panadol, Tempra, Tylenol) or ibuprofen.

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Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://en.wikipedia.org/wiki/Chickenpox
http://www.nlm.nih.gov/medlineplus/ency/article/001592.htm
http://www.charak.com/DiseasePage.asp?thx=1&id=117

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