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Hair Transplantation

Definition:
Hair transplantation is a surgical technique that involves moving skin containing hair follicles from one part of the body (the donor site) to bald or balding parts (the recipient site). It is primarily used to treat male pattern baldness, whereby grafts containing hair follicles that are genetically resistant to balding are transplanted to bald scalp. However, it is also used to restore eye lashes, eye brows, beard hair, and to fill in scars caused by accidents and surgery such as face lifts and previous hair transplants. Hair transplantation differs from skin grafting in that grafts contain almost all of the epidermis and dermis surrounding the hair follicle, and many tiny grafts are transplanted rather than a single strip of skin.

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Since hair naturally grows in follicles that contain groupings of 1 to 4 hairs, today’s most advanced techniques transplant these naturally occurring 1 – 4 hair “follicular units” in their natural groupings. Thus modern hair transplantation can achieve a natural appearance by mimicking nature hair for hair. This recent hair transplant procedure is called “Follicular Unit Transplantation.”..

History:
The use of both scalp flaps, in which a band of tissue with its original blood supply is shifted to the bald area, and free grafts dates back to the 19th century. Modern transplant techniques began in Japan in the 1930s, where surgeons used small grafts, and even “follicular unit grafts” to replace damaged areas of eyebrows or lashes. They did not attempt to treat baldness per se. Their efforts did not receive worldwide attention at the time, and the traumas of World War II kept their advances isolated for another two decades.

The modern era of hair transplantation in the western world was ushered in the late 1950s, when New York dermatologist Norman Orentreich began to experiment with free donor grafts to balding areas in patients with male pattern baldness. Previously it had been thought that transplanted hair would thrive no more than the original hair at the “recipient” site. Dr. Orentreich demonstrated that such grafts were “donor dominant,” as the new hairs grew and lasted just as they would have at their original home. Today Dr. Orentreich’s practice still performs hair transplants.

For the next twenty years, surgeons worked on transplanting smaller grafts, but results were only minimally successful, with 2-4 mm “plugs” leading to a doll’s head-like appearance. In the 1980s, Uebel in Brazil popularized using large numbers of small grafts, while in the United States Rassman began using thousands of “micrografts” in a single session.

In the late 1980s, Limmer introduced the use of the stereo-microscope to dissect a single donor strip into small micrografts. In 1995, Bernstein and Rassman published the first paper on “Follicular Unit Transplantation,” where hair is transplanted exclusively in naturally occurring groups of 1-4 hairs. With microscopic dissection of donor pieces from an excised portion of scalp, individual follicular units containing but 1-4 hairs could be prepared and individually relocated into needle punctures in the recipient areas. Since the transplanted hair mimics the way hair grows in nature, close to natural results were attainable.

The follicular unit hair transplant procedure has continued to evolve, becoming more refined and minimally invasive as the size of the graft incisions have become smaller. These smaller and less invasive incisions enable surgeons to place a larger number of follicular unit grafts into a given area. With the new “gold standard” of ultra refined follicular unit hair transplantation, over 50 grafts can be placed per square centimeter, when appropriate for the patient.

Surgeons have also devoted more attention to the angle and orientation of the transplanted grafts. The adoption of the “lateral slit” technique in the early 2000s, enabled hair transplant surgeons to orient 2 to 4 hair follicular unit grafts so that they splay out across the scalp’s surface. This enabled the transplanted hair to lie better on the scalp and provide better coverage to the bald areas. One disadvantage however, is that lateral incisions also tend to disrupt the scalp’s vascularity more than sagitals. Thus sagital incisions transect less hairs and blood vessels assuming the cutting instruments are of the same size. One of the big advantages of sagitals is that they do a much better job of sliding in and around existing hairs to avoid follicle transection. This certainly makes a strong case for physicians who do not require shaving of the recipient area. The lateral incisions bisect existing hairs perpendicular (horizontal) like a T while sagital incisions run parallel (vertical) along side and in between existing hairs. The use of perpendicular (lateral/coronal) slits verses parallel (sagital) slits however, has been heavily debated on patient based hair transplant communities. Many elite hair transplant surgeons typically adopt a combination of both methods depending on what is best for the patient.

The procedure:
At an initial consultation, the surgeon analyzes the patient’s scalp, discusses his preferences and expectations, and advises him/her on the best approach (e.g.,single vs. multiple sessions) and what results might reasonably be expected.

Click to see on pictures : CORRECTIVE HAIR TRANSPLANT PROCEDURE

For several days prior to surgery the patient refrains from using any medicines, or alcohol, which might result in intraoperative bleeding and resultant poor “take” of the grafts. Pre-operative antibiotics are commonly prescribed to prevent wound or graft infections.

Hair transplantation is a surgical technique in which a physician redistributes hairs from an area of thick growth to bald areas.

This Procedure is Performed because:
In patients who are concerned about their balding, hair transplantation can significantly improve their appearance and self confidence. Realistic expectations are important, however. It is important to remember that hair still cannot be created; it can only be redistributed from the back of the scalp to the front.

Most patients undergoing hair transplantation have traditional male or female pattern baldness, with hair loss on the front or top of the scalp. Patients must still have thick hair on the back or sides of the scalp, or there may not be enough hair follicles to move. In some cases, patients with hair loss from lupus, injuries, or other medical problems may be treated with hair transplantation.

Patients undergoing hair transplantation should be otherwise relatively healthy, or surgery is less likely to be safe and successful. Always discuss your risks and options with your physician before undergoing any elective surgery.

Surgery:
Transplant operations are performed on an outpatient basis, with mild sedation (optional) and injected topical anesthesia, and typically last about four hours. The scalp is shampooed and then treated with an antibacterial chemical prior to the donor scalp being harvested.

In the usual follicular unit procedure, the surgeon harvests a strip of skin from the posterior scalp, in an area of good hair growth. The excised strip is about 1-1.5 x 15-30 cm in size. While closing the resulting wound, assistants begin to dissect individual follicular unit grafts from the strip. Working with binocular microscopes, they carefully remove excess fibrous and fatty tissue while trying to avoid damage to the follicular cells that will be used for grafting.

The surgeon then uses a fine needle to puncture the sites for receiving the grafts, placing them in a predetermined density and pattern, and angling the wounds in a consistent fashion to promote a realistic hair pattern. The assistants generally do the final part of the procedure, inserting the individual grafts in place.

Risk Factor:As with any surgical procedure, risks exist. The most common complications of hair transplantation are bleeding, infection, and scarring.

Though less dangerous, it is also possible that the transplanted hair won’t look as good as you had desired. Older techniques often resulted in unnatural appearing tufts of new hair growth. With modern techniques, this complication is infrequent.

Post-operative care:
Advances in wound care allow for semi-permeable dressings, which allow seepage of blood and tissue fluid, to be applied and changed at least daily. The vulnerable recipient area must be shielded from the sun, and shampooing is started two days after the surgery. Some surgeons will have you shampoo the day after surgery. Shampooing is important to prevent scabs from occurring around the hair shaft. Scabs adhere to the hair shaft and increase the risk of losing newly transplanted hair follicles during the first 7 to 10 days post-op.

During the first ten days, virtually all of the transplanted hairs, inevitably traumatized by their relocation, will fall out (“shock loss”). After two to three months new hair will begin to grow from the moved follicles. The patient’s hair will grow normally, and continue to thicken through the next six to nine months. Any subsequent hair loss is likely to be only from untreated areas. Some patients elect to use medications to retard such loss, while others plan a subsequent transplant procedure to deal with this eventuality.

Modern techniques:
There are two main ways in which donor grafts are extracted today. These are the Strip Harvesting Technique and the Follicular Unit Extraction (FUE) Technique.

The Strip Harvesting Technique involves removing a strip containing a large group of follicular units from the donor area – almost always from the back and sides of the scalp. The strip is then divided into grafts (or follicular units) containing 1 to 4 follicles.

The Follicular Unit Extraction (FUE) Technique involves removing one follicular unit at a time directly from the donor area – usually the back and sides, but also sometimes from the chest, legs or face (beard hair) – using a small punch usually of between 0.5mm and 1mm in diameter.

Side effects:
Hair thinning, known as “shock loss”, is a common side effect that is usually temporary. Bald patches are also common, as fifty to a hundred hairs can be lost each day.

Other side effects include swelling of areas such as the scalp and forehead. If this becomes uncomfortable, medication may ease the swelling. Additionally, the patient must be careful if his scalp starts itching, as scratching will make it worse and cause scabs to form. A moisturizer or massage shampoo may be used in order to relieve the itching.

Relevant Anatomy
The scalp is divided into 5 layers, which are easily remembered by the mnemonic SCALP, which represents, in order from outermost to innermost layer, the skin, connective subcutaneous tissue, galea aponeurosis, loose connective tissue, and periosteum over the cranium.
The skin contains all the epidermal appendages, including hair follicles, which extend into the connective subcutaneous layer. In areas that have undergone hair loss, thinning of the outer 2 layers usually occurs. This situation can be appreciated when one compares the thickness of the scalp in recipient areas to that in donor areas.

The subcutaneous layer is well vascularized and contains the main penetrating branches of the named main arteries that travel primarily along the external surface of the galea. The importance of staying superficial along the connective subcutaneous tissue layer (when one makes slit recipient sites to avoid compromising circulation) has only recently become apparent. The scalp has an excellent blood supply. The supraorbital, supratrochlear, superficial temporal, postauricular, and occipital arteries are the primary vessels, and they typically travel with the veins.

The galea aponeurotica is a nonelastic layer that connects the frontalis muscles anteriorly with the occipitalis muscle at its posterior aspect. The temporoparietal fascia, in which the superficial temporal artery travels, is also connected to the galea. The galea sliding over the loose connective tissue layer allows for most scalp mobility. This loose connective tissue layer and the periosteum below have minimal sensory innervation.

The sensory innervation of the scalp closely follows the vascular supply. At the anterior aspect, the supraorbital and supratrochlear nerves provide sensation to the anterior half of the scalp. On occasion, sensation to the frontal scalp can diminish for several weeks when a large number of graft recipient sites are made along the hairline. The occipital nerve serves the posterior half of the scalp, whereas the supraauricular and superficial temporal nerves contribute innervation from the sides.

Perhaps no anatomic feature of the scalp is more important with regard to hair transplantation than the microscopic distribution of hair. Scalp hairs usually do not grow individually; they most often grow in tiny follicular-unit bundles, which usually contain 2-3 hairs and occasionally 1 or 4 hairs. A follicular-unit contains these 1-4 terminal hairs, a sebaceous gland element, and insertions of the arrector pili muscles, all wrapped in an adventitial tissue sheath. These follicular units are dispersed throughout the scalp, where non–hair-bearing skin constitutes up to 50% of the total tissue. By transplanting only these follicular units and dissecting away the 50% of unnecessary non–hair-bearing tissue, the most natural-appearing results can be attained.
Prognosis
Most modern hair transplants result in excellent hair growth within several months after the procedure. Often, however, more than one treatment session is needed to create the best-looking results. The replaced hairs are usually permanent, and no long-term care is necessary.

Recovery
During the recovery period after surgery, the scalp is often very tender. Strong pain medications taken by mouth may be necessary for several days. A bulky surgical dressing, or sometimes a smaller dressing protected by a baseball cap, must be worn for at least a day or two. Some surgeons may also recommend several days of antibiotics or anti-inflammatory drugs following surgery. After this very brief recovery period, no special treatment is needed.

Cost
In recent years hair transplants have become less expensive. Prices typically range from $3.00 to $7.00 per graft, with $4 to $5 per graft being about average. Normally the price per graft also drops as the size of the surgical session increases. Depending on the needs of the patient a typical surgical session can range from 1,500 to over 4,000 grafts, resulting in a total cost of approximately $6000 to $15,000. A few clinics offer larger sessions of up to 6000 grafts in one sitting.

Contraindications:
Perhaps the most difficult part of being a surgeon is knowing when not to operate. In elective cosmetic surgery, sound judgment must certainly be exercised.

Individuals must be motivated to undergo hair transplantation. Although the author does not conduct a formal psychological evaluation by means of lengthy questionnaires and examinations, some surgeons use this method. During the consultation, the present author generally reads to the individual to ensure that he or she is mature enough to decide to undergo the planned procedure. A prospective patient who has realistic motivations and expectations before the procedure is likely to be happy after the procedure. Honest and thorough preprocedural consultation is perhaps the most important part of the process.

Poor medical health is a potential contraindication for elective surgery of any kind. Individuals cannot be taking anticoagulants (eg, Coumadin, aspirin) before the procedure. Good surgical judgment must be exercised when one considers surgery in individuals with potentially complicating medical conditions. Age is not a medical contraindication. The author has performed procedures on men in their late 70s. Ensure that such patients provide medical clearance from their internist.

Perhaps no single hair-loss condition calls for more conservatism in judgment than premature MPB. Teenagers and men in their early 20s are particularly self-conscious about hair loss because most of their peers still have full heads of hair. These young men often hold unrealistic expectations, desiring a youthful hairline that will not be appropriate as they age. Worse, early surgical correction uses a large number of donor hairs, which will be sparse in the future, potentially resulting in an unnatural look and a disappointed patient.

In general, attempt to delay the procedure in individuals in their 20s or younger, though the author has performed procedures in select individuals as young as 20 years. When counseling young men about hair loss, the author advises a conservative approach to give patients time to consider hair transplantation. If the patient and surgeon agree on transplantation, restore a relatively high hairline and instruct the patient to use minoxidil for the crown region. Perhaps in the future, as effective medical therapies that end or substantially slow MPB progression become available, a less conservative approach can be taken.

For a number of medical conditions that are associated with or that can cause hair loss, treatment with hair transplantation is not appropriate. Examples are the active phases of alopecia areata, lupus, and infections. Scalp conditions, such as vitiligo and psoriasis, must be evaluated because hair transplantation can aggravate them.

Resources:
http://en.wikipedia.org/wiki/Hair_transplantation
http://www.nlm.nih.gov/medlineplus/ency/article/007205.htm

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Diagnonistic Test

Carotid Ultrasound (Carotid Doppler)

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Definition:
Ultrasound uses sound waves instead of radiation to generate snapshots or moving pictures of structures inside the body. This imaging technique works in a manner similar to radar and sonar, developed in World War II to detect airplanes, missiles, and submarines that were otherwise invisible. After coating your skin with a lubricant to reduce friction, a radiologist or ultrasound technician places an ultrasound transducer, which looks like a microphone, on your skin and may rub it back and forth to get the right view. The transducer sends sound waves into your body and picks up the echoes of the sound waves as they bounce off internal organs and tissue. A computer transforms these echoes into an image that is displayed on a monitor…….....CLICK & SEE

Doppler ultrasound is a variation of this technique that not only shows internal structures but also examines the flow of blood through blood vessels. Using the Doppler effect—the change in the frequency of sound or light waves as they bounce off a moving object—this kind of ultrasound produces an image of blood in motion…..CLICK & SEE

A Carotid ultrasound shows the amount of blood flow in the carotid arteries, the major blood vessels to the brain located on either side of your neck. With this imaging technique, your doctor can see if there is any narrowing of your carotid arteries because of cholesterol deposits or some other problem. This test is often used to evaluate people who have had a stroke or who might be at high risk for one because of reduced blood flow in the carotid arteries…......CLICK & SEE.

Who Needs Carotid Ultrasound?
Carotid ultrasound checks for plaque buildup in the carotid arteries. This buildup can narrow or block your carotid arteries. You may need a carotid ultrasound if you:

*Had a stroke or ministroke recently.
*Have an abnormal sound in your carotid artery called a carotid bruit (broo-E).

Your doctor can hear a carotid bruit with the help of a stethoscope put on your neck over the carotid artery. A bruit can mean that there’s a partial blockage in your carotid artery that could lead to a stroke.
Your doctor also may order a carotid ultrasound if he or she suspects you may have:

*Blood clots that can slow blood flow in your carotid artery
*A split between the layers of your carotid artery wall that weakens the wall or reduces the blood flow to your brain
A carotid ultrasound also may be done to see whether carotid artery surgery has restored normal blood flow. If you had a procedure called carotid stenting, your doctor may order a carotid ultrasound afterward to check the position of the stent put in your carotid artery. (The stent, a small mesh tube, helps prevent the artery from becoming narrowed or blocked again.)

Sometimes carotid ultrasound is used as a preventive screening test in people who have medical conditions that increase their risk of stroke, including high blood pressure and diabetes. People with these conditions may benefit from having their carotid arteries checked regularly even if they show no signs of plaque buildup.

What To Expect Before Carotid Ultrasound
Carotid ultrasound is a painless test, and typically there is little to do in advance. Your doctor will tell you how to prepare for your carotid ultrasound.

Process of Performing the Test.:
After squirting some clear jelly onto one side of your neck to help the ultrasound sensor slide around easily, a technician places the sensor against your skin. An image then appears on a video screen . As the technician moves the sensor back and forth on your neck, different views of the carotid artery appear on the screen. As the equipment measures the blood flow through the artery, you hear a noise that sounds like your heartbeat. The other side of your neck is checked in the same way. This test usually takes 15–30 minutes.

The ultrasound machine includes a computer, a video screen, and a transducer, which is a hand-held device that sends and receives ultrasound waves into and from the body.

You will lie down on your back on an exam table for the test. Your technician or doctor will put a gel on your neck where your carotid arteries are located. This gel helps the ultrasound waves reach the arteries better. Your technician or doctor will put the transducer against different spots on your neck and move it back and forth.

FIG-A

 

 

 
……….Fig->A.
The above Figure shows how the ultrasound probe is placed over the carotid artery. Figure B is a color ultrasound image showing blood flow (the red color in the image) in the carotid artery. Figure C is a waveform image showing the sound of flowing blood in the carotid artery.

The transducer gives off ultrasound waves and detects their echoes after they bounce off the artery walls and blood cells. Ultrasound waves can’t be heard by the human ear.

A computer uses the echoes of the ultrasound waves bouncing off the carotid arteries to create and record images of the insides of the arteries (usually in black and white) and your blood flowing through them (usually in color; this is the Doppler ultrasound). A video screen displays these live images for your doctor to review.

Risk Factor:
There are no risks linked to having a carotid ultrasound, because the test uses harmless sound waves. These are the same type of sound waves that doctors use to record pictures of fetuses in pregnant women.

What one must do after the test is over?
Carotid ultrasound is usually done in a doctor’s office or hospital. The test is painless and usually doesn’t take more than 30 minutes.
Usually there is nothing special you have to do after a carotid ultrasound, and you should be able to return to normal activities immediately.

Often your doctor will be able to tell you the results of the carotid ultrasound when it occurs or soon afterward.

What Does a Carotid Ultrasound Show?
A carotid ultrasound can show whether buildup of a fatty material called plaque has narrowed one or both of your carotid arteries and reduced blood flow to your brain.

.Fig->B………………….
The illustration shows a normal artery with normal blood flow (figure A) and an artery containing plaque buildup ( figure B).

If your carotid arteries are narrowed by plaque, you may be at risk for having a stroke. That risk depends on how much of your artery is blocked and how much blood flow is restricted. To reduce your risk for stroke, your doctor may recommend medical or surgical treatments to reduce or remove the plaque buildup in your carotid arteries.

How long is it before the result of the test is known?
The technician records the test on videotape for review by a radiologist. The radiologist then makes measurements from the video picture and submits a report to your doctor. Your doctor should have the results within a few days.

Key Points to Note:
*Carotid ultrasound is a test that uses high-frequency sound waves to create images of the insides of the two large arteries in your neck. These arteries, called carotid arteries, supply your brain with blood.

*A carotid ultrasound can show whether buildup of a fatty material called plaque has narrowed one or both of your carotid arteries and reduced blood flow to your brain.

*If your carotid arteries are narrowed by plaque, you may be at risk for having a stroke, depending on how much of your artery is blocked and how much blood flow is restricted.

*You may need a carotid ultrasound if you had a stroke or ministroke recently or are at high risk for having a stroke.

*Carotid ultrasound is a painless test done in a doctor’s office or hospital. It usually doesn’t take more than 30 minutes and requires no preparation or followup.

*There are no risks linked to having a carotid ultrasound, because the test uses harmless sound waves.

Resources:
http://www.health.harvard.edu/diagnostic-tests/carotid-ultrasound.htm
http://www.nhlbi.nih.gov/health/dci/Diseases/cu/cu_all.html

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Herbs & Plants Suppliments our body needs

Kava

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Botanical Name: Piper methysticum
Family:Pepper/ Piperaceae
Kingdom: Plantae
Order: Piperales
Genus: Piper
Species: P. methysticum
Other names: kava kava, kawa, kew, yagona, sakau .awa (Hawaii), ‘ava (Samoa), yaqona (Fiji), and sakau (Pohnpei).
Parts Used:The part of the plant used medicinally is the root. Although the root was traditionally chewed or made into a beverage, kava is now available in capsule, tablet, beverage, tea, and liquid extract forms.

Habitat:South Pacific Isands.

Description:
Kava is a tall shrub in the pepper family that grows in the South Pacific islands.Kava kava belongs to the pepper family (Piperaceae) and is also known as kava, asava pepper, or intoxicating pepper. It grows to an average height of 6 ft (1.83 m) and has large heart-shaped leaves that can grow to 10 in (25.4 cm) wide. A related species is Piper sanctum, a native plant of Mexico that is used as a stimulant. It has been used there for thousands of years as a folk remedy and as a social and ceremonial beverage.

click to see the pictures…..(01)…..(1)…....(2)………(3)………..(4)..

The part of the plant used medicinally is the root. Although the root was traditionally chewed or made into a beverage, kava is now available in capsule, tablet, beverage, tea, and liquid extract forms.

Botany and agronomy
There are several cultivars of kava, with varying concentrations of primary and secondary psychoactive substances. The largest number are grown in the Republic of Vanuatu, and so it is recognised as the “home” of kava. Kava was historically grown only in the Pacific islands of Hawaii, Federated States of Micronesia, Vanuatu, Fiji, the Samoas and Tonga. Some is grown in the Solomon Islands since World War II, but most is imported. Kava is a cash crop in Vanuatu and Fiji.

The kava shrub thrives in loose, well-drained soils where plenty of air reaches the roots. It grows naturally where rainfall is plentiful (over 2,000 mm/yr). Ideal growing conditions are 20 to 35 degrees Celsius (70 to 95 °F), and 70–100% relative humidity. Too much sunlight is harmful, especially in early growth, so kava is an understory crop.

Kava cannot reproduce sexually. Female flowers are especially rare and do not produce fruit even when hand-pollinated. Its propagation is entirely due to human efforts by the method of striking.

Traditionally, plants are harvested around 4 years of age, as older plants have higher concentrations of kavalactones. But in the past two decades farmers have been harvesting younger and younger plants, as young as 18 months. After reaching about 2 m height, plants grow a wider stalk and additional stalks, but not much taller. The roots can reach 60 cm depth.

Composition
Fresh kava root contains on average 80% water. Dried root contains approximately 43% starch, 20% fibers, 15% kavalactones, 12% water, 3.2% sugars, 3.6% proteins, and 3.2% minerals. Kavalactone content is greatest in the roots and decreases higher up the plant. Relative concentrations of 15%, 10% and 5% have been observed in the root, stump, and basal stems, respectively.

The mature roots of the kava plant are harvested after a minimum of 4 years (at least five years ideally) for peak kavalactone content. Most kava plants produce around 50 kgs (110 lbs) of root when they are harvested. Kava root is classified into two categories: crown root (or chips) and lateral root. Crown roots are the large diameter pieces that look like big (1.5 inch to 5 inches diameter) wooden poker chips. Most kava plants consist of approximately 80% crown root upon harvesting. Lateral roots are smaller diameter roots that look more like a typical root. A mature kava plant is approximately 20% lateral roots. Kava lateral roots have the highest content of kavalactones in the kava plant. “Waka” grade kava is kava that is made of lateral roots only.
General use
Kava kava has been prescribed by healthcare providers to treat a wide range of ailments, including insomnia, nervousness, and stress-related anxiety and anxiety disorders. It is also reported to relieve urinary infections, vaginitis, fatigue, asthma, rheumatism, and pain.

The active ingredients in kava kava are called kavalactones and are found in the root of the plant. Kavalactones cause reactions in the brain similar to pharmaceutical drugs prescribed for depression and anxiety. Research has shown that kavalactones have a calming, sedative effect that relaxes muscles, relieves spasms, and prevents convulsions. Kavalactones also have analgesic (pain-relieving) properties that may bring relief to sore throats, sore gums, canker sores, and toothaches.

Kava kava is a strong diuretic that is reportedly beneficial in the treatment of gout, rheumatism, and arthritis. The diuretic effect of the herb relieves pain and helps remove waste products from the afflicted joints. Antispasmodic properties have shown to help ease menstrual cramps by relaxing the muscles of the uterus. Kava kava’s antiseptic and anti-inflammatory agents may help relieve an irritable bladder, urinary tract infections, and inflammation of the prostate gland.

Kava root...……………….....Kava root power…………….Cava in bottle as medicine

Medicinal Uses:
Kava kava has been used as a medicinal herb for hundreds of years and used by Pacific Islanders to treat rheumatism, asthma, worms, obesity, headaches, fungal infections,leprosy, gonorrhea, vaginal infections, urinary infections, menstrual problems, migraine headaches, and insomnia. It was also used as a diuretic, an aphrodisiac, to promote energy, and to bring about sweating during colds and fevers. Pacific Islanders consume a kava kava drink at social, ritual, and ceremonial functions. It is drunk at ceremonies to commemorate marriages, births, and deaths; in meetings of village elders; as an offering to the gods; to cure illness; and to welcome honored guests. Pope John Paul II, Queen Elizabeth II, and Hillary Rodham Clinton have all drunk kava kava during their island visits.

The drink is prepared by grinding, grating, or pounding the roots of the plant, then soaking the pulp in cold water or coconut milk. Traditionally the root was chewed, spit into a bowl, and mixed with coconut milk or water. That practice is no longer the standard.

Captain James Cook has been credited with the Western discovery of kava kava during his journey to the South Pacific in the late 1700s. The first herbal products made from kava kava appeared in Europe in the 1860s. Pharmaceutical preparations became available in Germany in the 1920s. Currently, kava kava has received widespread attention because of its reputation to promote relaxation and reduce stress.

Preparation & Consumption:
Traditional preparation
Kava is consumed in various ways throughout the Pacific Ocean cultures of Polynesia, Vanuatu, Melanesia and some parts of Micronesia and Australia. Traditionally it is prepared by either chewing, grinding or pounding the roots of the kava plant. Grinding is done by hand against a cone-shaped block of dead coral; the hand forms a mortar and the coral a pestle. The ground root/bark is combined with only a little water, as the fresh root releases moisture during grinding. Pounding is done in a large stone with a small log. The product is then added to cold water and consumed as quickly as possible.

Kava root drying in Lovoni village, Ovalau

The extract is an emulsion of kavalactone droplets in starch. The taste is slightly pungent, while the distinctive aroma depends on whether it was prepared from dry or fresh plant, and on the variety. The colour is grey to tan to opaque greenish.

Kava prepared as described above is much more potent than processed kava. Chewing produces the strongest effect because it produces the finest particles. Fresh, undried kava produces a stronger beverage than dry kava. The strength also depends on the species and techniques of cultivation. Many find mixing powdered kava with hot water makes the drink stronger. However the active ingredients of kava, such as Kavalactone, are ruined at 140 degrees. Most tea steeps at 180 degrees for at least a couple minutes which will reduce the potency of the kava.

In Vanuatu, a strong kava drink is normally followed by a hot meal or tea. The meal traditionally follows some time after the drink so that the psychoactives are absorbed into the bloodstream quicker. Traditionally no flavoring is added.

Fijians commonly share a drink called “grog”, made by pounding sun-dried kava root into a fine powder and mixing it with cold water. Traditionally, grog is drunk from the shorn half-shell of a coconut, called a “bilo.” Despite tasting very much like dirty water, grog is very popular in Fiji, especially among young men, and often brings people together for storytelling and socializing.

Modern preparation
In modernized countries Kava beverage is usually made from Kava root powder. The root is dried and then finely ground into powder before being exported. Generally one tablespoon of powder is added per cup of water, but sometimes as much as a half a cup of powder (eight tablespoons) is added per cup of water to increase potency. The powder is then soaked in water for approximately 30 minutes to allow the water to completely soak through the powdered fibers. Lecithin is often added to aid in the process of emulsifying the kavalactones with water. The Kava powder, water, and lecithin are blended in a blender for several minutes then strained into a straining cloth. Nylon, cheesecloth, and silk screen are common materials for straining. The remaining liquid is squeezed from the pulp and the pulp is discarded. As an alternative to the blender method, with the powdered pulp enclosed within the straining material, the pulp is massaged for five to ten minutes in water, then the liquid is wrung out. The more pressure that is applied to the wet powdered pulp while wringing it out, the more kavalactones will be released from it. Finally the pulp resin is discarded and the beverage is enjoyed. Often coconut water, coconut milk, lemongrass, cocoa, sugar, or soy milk is added to improve flavor.

Pharmacology
Kava’s active principal ingredients are the kavalactones, of which at least 15 have been identified and are all considered psychoactive. Only six of them produce noticeable effects, and their concentrations in kava plants vary. Different ratios can produce different effects. Kava has some abuse potential and some experts recommend cycling use over 1 to 3 months.

Pills
Pharmaceutical companies and herbal supplement companies extract kavalactones from the kava plant using solvents such as acetone and ethanol and produce pills standardized with between 30% and 90% kavalactones. Some kava herbal supplements have been accused of contributing to very rare but severe hepatotoxic reactions (see section on safety) such may have been due to the use of plant parts other than the root, such as stems or peelings that are known to have been exported to European manufacturers. A kava pill usually has anywhere from 60 mg to 150 mg of kavalactones. By comparison the typical bowl of traditionally prepared kava beverage has around 250 mg of kavalactones.

Uses: In some parts of the Western World, kava extract is marketed as herbal medicine against stress, insomnia, and anxiety. A Cochrane Collaboration systematic review of its evidence concluded that it was likely to be more effective than placebo at treating short-term social anxiety. Safety concerns have been raised over liver toxicity, although research indicates that this may be largely due to the use of stems and leaves in supplements, which were not used indigeneously

The word kava is used to refer both to the plant and the beverage produced from its roots. Kava is a tranquilizer primarily consumed to relax without disrupting mental clarity. Its active ingredients are called kavalactones.

Because kava can cause sedation, and in high amounts, intoxication, kava drinks are consumed in some parts of the world in much the same way as alcohol.

Although it’s not clear exactly how kava works, kavalactones may affect the levels of neurotransmitters (chemicals that carry messages from nerve cells to other cells) in the blood. Kava has been found to affect the levels of specific neurotransmitters, including norepinephrine, gamma aminobutyric acid (GABA) and dopamine

A number of well-designed studies have examined kava’s ability to relieve anxiety compared to anxiety medication or a placebo. The results have been promising.

In 2003, a review by the Cochrane Collaboration examined the existing research to see how kava fared compared to a placebo in treating anxiety. After analyzing the 11 studies (involving a total of 645 people) that met the criteria, the researchers concluded that kava “appears to be an effective symptomatic treatment option for anxiety.” However, they added that it seemed to be a small effect.

Effects:
A moderately potent kava drink causes effects within 20–30 minutes that last for about two and a half hours, but can be felt for up to eight hours. Because of this, it is recommended to space out servings about fifteen minutes apart. Some report longer term effects up to two days after ingestion, including mental clarity, patience, and an ease of acceptance. The effects of kava are most often compared to alcohol, or a large dose of Valium.

The sensations, in order of appearance, are slight tongue and lip numbing (the lips and skin surrounding may appear unusually pale); mildly talkative and sociable behavior; clear thinking; anxiolytic (calming) effects; relaxed muscles; and a very euphoric sense of well-being. The numbing of the mouth is caused by the two kavalactones kavain and dihydrokavain which cause the contraction of the blood vessels in these areas acting as a local topical anesthetic. These anesthetics can also make one’s stomach feel numb. Sometimes this feeling has been mistaken for nausea. Some report that caffeine, consumed in moderation in conjunction with kava can significantly increase mental alertness.

A potent drink results in a faster onset with a lack of stimulation, the user’s eyes become sensitive to light, they soon become somnolent and then have deep, dreamless sleep within 30 minutes. Sleep is often restful and there are pronounced periods of sleepiness correlating to the amount and potency of kava consumed. Unlike alcohol-induced sleep, after wakening the drinker does not experience any mental or physical after effects. However, this sleep has been reported as extremely restful and the user often wakes up more stimulated than he or she normally would. Although excessive consumption of exceptionally potent brew has been known to cause pronounced sleepiness into the next day. Although heavy doses can cause deep dreamless sleep, it is reported that many people experience lighter sleep and rather vivid dreams after drinking moderate amounts of kava.

After thousands of years of use by the Polynesians and decades of research in Europe and the U.S., the traditional use of kava root has never been found to have any addictive or permanent adverse effects. Users do not develop a tolerance. While small doses of kava have been shown to slightly improve memory and cognition, large amounts at one time have been shown to cause intoxication. In Utah, California, and Hawaii there have been cases where people were charged with driving under the influence of alcohol after drinking a significant amount of kava (eight cups or more) although some of them were acquitted due to the laws not being broad enough to cover kava consumption.

Kava Culture:
Kava is used for medicinal, religious, political, cultural and social purposes throughout the Pacific. These cultures have a great respect for the plant and place a high importance on it. Correspondingly, the paraphernalia surrounding the traditional kava ceremony are expertly crafted. Traditionally designed kava bowls are bowls made from a single piece of wood, with multiple legs. More modern examples are also highly decorated, often carved and inlayed with mother of pearl and shell.…CLICK & SEE

Kava root being prepared in Asanvari, a village on Maewo Island in the Pacific nation of Vanuatu. Photo taken in September, 2002 by Jordan Bigel.

Kava is used primarily at social gatherings to increase amiability and to relax after work. It has great religious significance, being used to obtain inspiration. Among some fundamentalist Christian sects in the Western Pacific, the drink has been demonized and seen as a vice, and young members of these religions often reject its traditional use. However, among most mainline Christian denominations, i.e. the Roman Catholic, Methodist, and Anglican churches, kava drinking is encouraged where it replaces the greater danger of alcohol.

Basic research on anti-cancer potential
On 15 February 2006, the Fiji Times and Fiji Live reported that researchers at the University of Aberdeen in Scotland and the Laboratoire de Biologie Moleculaire du Cancer in Luxembourg had discovered that kava may treat ovarian cancer and leukemia. Kava compounds inhibited the activation of a nuclear factor that led to the growth of cancer cells. The Aberdeen University researchers published in the journal The South Pacific Journal of Natural Science that kava methanol extracts had been shown to kill leukemia and ovarian cancer cells in test tubes.The kava compounds were shown to target only cancerous cells; no healthy cells were harmed. This may help explain why kava consumption is correlated with decreased incidence of cancer.

Fiji Kava Council Chairman Ratu Josateki Nawalowalo welcomed the findings, saying that they would boost the kava industry. For his part, Agriculture Minister Ilaitia Tuisese called on the researchers to help persuade members of European Union to lift their ban on kava imports.

Liver damage incidents and regulation
In 2001 concerns were raised about the safety of commercial kava products. There have been allegations of severe liver toxicity, including liver failure in some people who had used dietary supplements containing kava extract (but not in anyone who had drunk kava the traditional way). Out of the 50 people worldwide taking kava pills and extracts that have had some type of problem, almost all of them had been mixing them with alcohol and pills that could have effects on the liver.[16] The fact that different kava strains have slightly different chemical composition made testing for toxicity difficult as well.

The possibility of liver damage consequently prompted action of many regulatory agencies in European countries where the legal precautionary principle so mandated. In the UK, the Medicines for Human Use (Kava-kava) (Prohibition) Order 2002 prohibits the sale, supply or import of most derivative medicinal products. Kava is banned in Switzerland, France, Germany and The Netherlands[17]. The health agency of Canada issued a stop-sale order for kava in 2002. But legislation in 2004 made the legal status of kava uncertain. The United States CDC has released a report[18] expressing reservations about the use of kava and its possibly adverse side effects (specifically severe liver toxicity), as has the Food and Drug Administration (FDA). The Australian Therapeutic Goods Administration has recommended that no more than 250 mg of kavalactones be taken in a 24 hour period.[20] According to the Medicines Control Agency in the U.K., there is no safe dose of kava, as there is no way to predict which individuals would have adverse reactions.

Potential Side Effects of Kava:
Side effects include indigestion, mouth numbness, skin rash, headache, drowsiness and visual disturbances. Chronic or heavy use of kava has linked to pulmonary hypertension, skin scaling, loss of muscle control, kidney damage, and blood abnormalities.

Kava may lower blood pressure and it also may interfere with blood clotting, so it shouldn’t be used by people with bleeding disorders. People with Parkinson’s disease shouldn’t use kava because it may worsen symptoms.

Kava should not be taken within 2 weeks of surgery. Pregnant and nursing women, children, and people with liver or kidney disease shouldn’t use kava.

Possible Drug Interactions:
Kava shouldn’t be taken by people who are taking Parkinson’s disease medications, antipsychotic drugs, or any medication that influences dopamine levels.

Kava shouldn’t be combined with alcohol or medications for anxiety or insomnia, including benzodiazepines such as Valium (diazepam) or Ativan (lorazepam). It may have an additive effect if taken with drugs that cause drowsiness.

Kava may have an additive effect if combined with antidepressant drugs called monoamine oxidase inhibitors (MAOI).

Kava shouldn’t be taken with any drug or herb that impairs liver function. Kava also may interfere with blood clotting, so people taking Coumadin (warfarin) or any drug that influences blood clotting should avoid it unless under a doctor’s supervision.

Kava is a diuretic, so it may have an additive effect if combined with drugs or herbs that have diuretic properties.

Allergy
Literature suggests that <0.5% of people that take kava have an allergic reaction to it. Allergic reactions are usually mild and include itchy skin or itchy throat, and hives on the skin usually prevalent on the user’s belly region. If someone has an allergy to any relative of the pepper family, such as black pepper, they have a higher chance of having a kava allergy.

Click to learn more about Kava.:-.>………(1)….(2)……(3).…..(4)….(5)

Disclaimer:The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

Resources:
http://altmedicine.about.com/od/kava/p/kava.htm
http://en.wikipedia.org/wiki/Kava

http://www.healthline.com/galecontent/kava-kava

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Herbs & Plants

TIENCHI GINSENG /GINSENG

Panax quinquefolius foliage and fruitImage via Wikipedia

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Botanical Name :Panax notoginseng, Panax pseudoginseng San qi.
Family:Araliaceae
Subfamily:Aralioideae
Genus:Panax
Kingdom:    Plantae
Order:Apiales
Common Name:Ginseng, San Qi,Tan Qi,Teinchi Ginseng
Part Used: Tuberous root.
Collection and habitat: Origin –China, An Asian herb used primarily in Korea, China, and Japan; the root is gathered in the spring or fall. The older the roots, the better.

Description:Tien-chi Ginseng (Panax pseudoginseng) is a unique type of ginseng plant that grows in southwestern China; mainly cultivated in Yunnan and Guangxi Provinces. The main part of the tien-chi plant used as a health product is the root, which is valued for regulating blood circulation, as detailed in a 1979 report (cover pictured here). The flower is used somewhat differently, as a “heat clearing and toxin cleansing” herb, given to reduce inflammation, feverish feeling, skin eruptions, and sore throat.

…click to see the pictures>…....(01)…….(1).…(2)..…..…(3)..…..…(4)..…….

All parts of the ginseng plants contain the saponins-known as ginsenosides-that have been shown to be responsible for most of ginseng’s beneficial effects. Additionally, the flowers contain flavonoids that contribute to the cooling and detoxifying action. The flowers have a mild, pleasant taste, and subtle aroma. In China, the flower tea is a favorite summer drink, used to compensate for the hot weather of the central and southern regions. The flowers are collected in early summer and then extracted and concentrated onto cane sugar to yield an instant tea granule, manufactured by Shenbao Corporation of Guanxi Province.

WHAT IT DOES: Tien chi root is sweet and slightly bitter in taste, and warming in action. It stops bleeding while simultaneously reducing blood congestion and clotting. It also relaxes, detoxifies and repairs blood vessels, and speeds wound healing. It is a mild tonic.

Medicinal Uses: Immune tonic and stimulant, adaptogenic, hepatoprotective, antiviral, cardiotonic, anti-inflammatory, anticomplement, antihyperglycemic, antiulcer, antioxidant, hemostatic, analgesic; promotes blood circulation.

Functions in liver disease: Antiviral, hepatoprotective, strong stimulant and tonic for the immune system. Directly active against hepatitis viruses.

Properties: Warming, both hemostatic and anticoagulant (depending on the condition), disperses blood stasis, anti-swelling, analgesic, anti-inflammatory, immunomodulating, hypolipemic (raw sanqi), hyperlipemic (cured sanqi), anti-atherosclerotic, antioxidant.

Most Common Traditional Uses: Hemorrhages of various kinds (e.g., coughing blood, vomiting blood, nosebleeds, hematochezia, and metrorrhagia), traumatic injuries with bleeding and pain, stabbing pain in chest and abdomen.

By Chinese standards, tienchi is not an ancient herb, being first described only about 400 years ago, in Li Shi-Zhen’s Ben Cao Gang Mu (circa 1590 A.D.). It is cultivated mainly in southern China, in the provinces of Yunnan, Guangxi, and Guangdong, at altitudes between 800 and 1,000 m. Tienchi is closely related to Asian ginseng and American ginseng.Like ginseng, it also contains ginsenosides (esp. Rb1 and Rg1) as its major active components. However, unlike ginseng, tienchi’s most well-known traditional use is not as a tonic but as a hemostatic, and is a common ingredient in many hemostatic formulas both for internal and external applications. Perhaps the most famous formula of this kind is Yunnan Baoyao (White Drug of Yunnan Province) which contains tienchi as a major component. This medicine was carried by both Chinese and American airmen (the Flying Tigers) during World War II to stop bleeding due to wounds and injuries.

After modern chemical and pharmacological studies have shown tienchi to contain ginsenosides and to exhibit broad biological activities that are typical of tonics (cardiovascular, immunomodulating, anti-inflammatory, antioxidant, normalizing blood lipids and blood pressure, etc.), it is now also used in tonic formulas.

Research Highlights:-

• Studies from China show that it speeds recovery from wounds by over 50% (reported in Dharmananda, 1994).

• Studies have shown that this action is strengthened by repeated administration and tends to be dose-dependent (Gong YH et al., 1991).

• In mouse studies, Tien chi root extract has shown significant anti-tumor activity on skin tumors induced by chemical toxins (Konoshima et al., 1999).

• In a study of patients with essential hypertension, tien chi root saponins, were shown to precipitate remarkable improvement in left ventricular diastolic function. The researchers concluded that the herb could improve heart muscle relaxation by enhancing calcium pump activity, inhibiting intracellular calcium overload, and lightening left ventricular muscle mass (Feng et al., 1997). In spite of this positive effect, however, the herb is not a reliable blood pressure-lowering agent by itself, though it may be a useful addition to a treatment protocol (Lei XL et al., 1986).

•The development of cardiac dysfunction and weakness immediately following traumatic burns is a serious problem, and one that is very difficult to treat. In a placebo controlled trial performed on rats at the Institute of Burn Research in Chongqing, China, researchers determined that tien chi root was effective in improving early post-burn cardiac function (Huang et al., 1999).

• The actions of this herb on the cardiovascular system are complex, involving multiple mechanisms. Studies done at the Chinese Academy of Medical Science in Beijing have shown that the saponins in tien chi root act as calcium channel blockers in neurons (Ma et al., 1997).

• The protection the whole root affords against hypoxic damage is attributed to the improvement of energy metabolism, preserving the structural integrity of neurons (Jiang KY et al., 1995).

• Other effects include lipid-lowering activity (Xu et al., 1993), increased outflow of coronary vessels and relaxed constriction of ileum smooth muscles (Hu Y et al., 1992), and anti-arrhythmic activity (Gao BY et al., 1992).

• A study on rabbits suffering from hemorrhagic shock examined the effects of various combinations of salvia root, tien chi root and chuan xiong rhizome (Ligusticum wallichii). Blood tests showed that all three herbs were effective for relieving blood pressure and heart rate reduction, but that the combination of any two herbs was superior to
using a single herb, improving results and lowering the required dosage (Wang et al., 1997)

Disclaimer:
The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider.

Resources:
http://eatmoreherbs.com/zine/ginseng_tienchi.html
http://www.globalherbalsupplies.com/herb_information/ginseng_tienchi.htm#top
http://www.health-report.co.uk/phytochi_tienchi_ginseng.html
http://oneearthherbs.squarespace.com/important-herbs/tien-chi-root-panax-notoginseng.html
http://www.itmonline.org/jintu/tienchi.htm

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Positive thinking

Money Doesnot Always Buy Happiness

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Economists and psychologists and the rest of us have long wondered if more money would make us happier. Here’s the answer :-

click & see

All in all, it was probably a mistake to look for the answer to the eternal question. Does money buy happiness?This is  from people who practice what’s called the dismal science. For when economists tackled the question, they started from the observation that when people put something up for sale they try to get as much for it as they can, and when people buy something they try to pay as little for it as they can. Both sides in the transaction, the economists noticed, are therefore behaving as if they would be more satisfied (happier, dare we say) if they wound up receiving more money (the seller) or holding on to more money (the buyer). Hence, more money must be better than less, and the only way more of something can be better than less of it is if it brings you greater contentment. The economists’ conclusion: the more money you have, the happier you must be.

 

Depressed debutantes, suicidal CEOs, miserable magnates and other unhappy rich folks aren’t the only ones giving the lie to this. “Psychologists have spent decades studying the relation between wealth and happiness,” writes Harvard University psychologist Daniel Gilbert in his best-selling “Stumbling on Happiness,” “and they have generally concluded that wealth increases human happiness when it lifts people out of abject poverty and into the middle class but that it does little to increase happiness thereafter.”

That flies in the face of intuition, not to mention economic theory. According to standard economics, the most important commodity you can buy with additional wealth is choice. If you have $20 in your pocket, you can decide between steak and peanut butter for dinner, but if you have only $1 you’d better hope you already have a jar of jelly at home. Additional wealth also lets you satisfy additional needs and wants, and the more of those you satisfy the happier you are supposed to be.

The trouble is, choice is not all it’s cracked up to be. Studies show that people like selecting from among maybe half a dozen kinds of pasta at the grocery store but find 27 choices overwhelming, leaving them chronically on edge that they could have chosen a better one than they did. And wants, which are nice to be able to afford, have a bad habit of becoming needs (iPod, anyone?), of which an advertising- and media-saturated culture create endless numbers. Satisfying needs brings less emotional well-being than satisfying wants.

The nonlinear nature of how much happiness money can buy—lots more happiness when it moves you out of penury and into middle-class comfort, hardly any more when it lifts you from millionaire to decamillionaire—comes through clearly in global surveys that ask people how content they feel with their lives. In a typical survey people are asked to rank their sense of well-being or happiness on a scale of 1 to 7, where 1 means “not at all satisfied with my life” and 7 means “completely satisfied.” Of the American multimillionaires who responded, the average happiness score was 5.8. Homeless people in Calcutta came in at 2.9. But before you assume that money does buy happiness after all, consider who else rated themselves around 5.8: the Inuit of northern Greenland, who do not exactly lead a life of luxury, and the cattle-herding Masai of Kenya, whose dung huts have no electricity or running water. And proving Gilbert’s point about money buying happiness only when it lifts you out of abject poverty, slum dwellers in Calcutta—one economic rung above the homeless—rate themselves at 4.6.

Studies tracking changes in a population’s reported level of happiness over time have also dealt a death blow to the money-buys-happiness claim. Since World War II the gross domestic product per capita has tripled in the United States. But people’s sense of well-being, as measured by surveys asking some variation of “Overall, how satisfied are you with your life?,” has barely budged. Japan has had an even more meteoric rise in GDP per capita since its postwar misery, but measures of national happiness have been flat, as they have also been in Western Europe during its long postwar boom, according to social psychologist Ruut Veenhoven of Erasmus University in Rotterdam. A 2004 analysis of more than 150 studies on wealth and happiness concluded that “economic indicators have glaring shortcomings” as approximations of well-being across nations, wrote Ed Diener of the University of Illinois, Urbana-Champaign, and Martin E. P. Seligman of the University of Pennsylvania. “Although economic output has risen steeply over the past decades, there has been no rise in life satisfaction … and there has been a substantial increase in depression and distrust.”

That’s partly because in an expanding economy, in which former luxuries such as washing machines become necessities, the newly affluent don’t feel the same joy in having a machine do the laundry that their grandparents, suddenly freed from washboards, did. They just take the Maytag for granted. “Americans who earn $50,000 per year are much happier than those who earn $10,000 per year,” writes Gilbert, “but Americans who earn $5 million per year are not much happier than those who earn $100,000 per year.” Another reason is that an expanding paycheck, especially in an expanding economy, produces expanding aspirations and a sense that there is always one more cool thing out there that you absolutely have to have. “Economic success falls short as a measure of well-being, in part because materialism can negatively influence well-being,” Diener and Seligman conclude.

If money doesn’t buy happiness, what does? Grandma was right when she told you to value health and friends, not money and stuff. Or as Diener and Seligman put it, once your basic needs are met “differences in well-being are less frequently due to income, and are more frequently due to factors such as social relationships and enjoyment at work.” Other researchers add fulfillment, a sense that life has meaning, belonging to civic and other groups, and living in a democracy that respects individual rights and the rule of law. If a nation wants to increase its population’s sense of well-being, says Veenhoven, it should make “less investment in economic growth and more in policies that promote good governance, liberties, democracy, trust and public safety.”

(Curiously, although money doesn’t buy happiness, happiness can buy money. Young people who describe themselves as happy typically earn higher incomes, years later, than those who said they were unhappy. It seems that a sense of well-being can make you more productive and more likely to show initiative and other traits that lead to a higher income. Contented people are also more likely to marry and stay married, as well as to be healthy, both of which increase happiness.)

If more money doesn’t buy more happiness, then the behavior of most Americans looks downright insane, as we work harder and longer, decade after decade, to fatten our W-2s. But what is insane for an individual is crucial for a national economy that is, ever more growth and consumption. Gilbert again: “Economies can blossom and grow only if people are deluded into believing that the production of wealth will make them happy … Economies thrive when individuals strive, but because individuals will strive only for their own happiness, it is essential that they mistakenly believe that producing and consuming are routes to personal well-being.” In other words, if you want to do your part for your country’s economy, forget all of the above about money not buying happiness.

The best things in life are free

Source: Newsweek

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