Month: September 2008

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Grey Guns Against Addiction

Coronal section of brain through anterior comm...Image via Wikipedia

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Scientists have found that the brain tries to defend itself against drug abuse by making internal changes.

Researchers have received a rare insight into the brain’s fight against substance abuse, boosting the possibilities of developing better medicines to combat drug addiction.

The study, that involved many Indian scientists working in the US, has identified several brain mechanisms that underlie addiction-related structural changes. To their surprise, the researchers found that these mechanisms are an attempt by the brain to defend itself against excessive drug use.

The structural changes that appeared in the brain after chronic cocaine exposure, at critical points of communication between cells in the nucleus accumbens — the reward or pleasure centres — might actually function to limit addiction rather than support it, says Suprabha Pulipparacharuvil, the first author of the study. The findings appear in the August 28 issue of the journal Neuron. “They tend slow the process or minimise the long-lasting behavioural changes associated with addiction,” she told KnowHow.

Previous studies have shown that repeated use of drugs like cocaine, amphetamines and nicotine increases the number of anatomical structures called dendritic spines in the regions associated with pleasure and reward. These dendritic spines are the sites where brain cells communicate with one another. Many scientists believe that this long-lasting brain rewiring underlies similarly persistent drug taking and drug seeking behaviours associated with addiction and relapse. The mechanism that controls this brain rewiring and its relationship to addiction-related behaviour was not known previously.

In the new study involving laboratory mice, the researchers found that cocaine suppresses the activity of a set of proteins called MEF2 proteins. As MEF2 normally reduces the number of brain connections, suppressing it leads to an increase in dendritic spine density. The researchers also found that enhanced MEF2 activity in the brain blocked a drug-induced increase in dendritic spine density and increased addiction-related behavioural responses to cocaine.

This seems to be the brain’s natural response to cocaine. It may not be important for the process of addiction but may limit it, says Christopher Cowan, who led the research. The brain adapts to the effects of cocaine, and some of these changes may actually oppose addiction, he says.

Cowan is hopeful that by identifying a gene family that modulates the behavioural responses to cocaine, the study may generate new potential protein targets for drug companies to pursue.

“Relapse, or the resumption of active drug taking and drug seeking, is very common in drug addicts,” says Cowan. “Addiction-related brain changes and behaviours seem to be hardwired and semi-permanent, and treatment options are limited. Our data suggest that rather than trying to block the process of increasing dendritic spine density, we may actually want to look at treatments that try to enhance this.”

MEF2 is activated in response to brain activity. It actually tells the brain to eliminate the potential growth of too many communication sites between nerve cells. But repeated exposure to cocaine disrupts this function of MEF2, resulting in new brain connections.

To investigate the relationship between MEF2 and spine density changes, the researchers varied the level of the protein in the nucleus accumbens. Brain imaging done after administration of cocaine to the lab animals showed that it prevented MEF2 from limiting dendritic spine increase.

To test MEF2’s relationship to behaviour, the researchers monitored the movement of the mice after daily exposure to the same amount of cocaine. The same dose of cocaine produced a larger behavioural response after repeated days of drug injections, resulting in a “sensitised” response. This sensitised behavioural response to the drug is very stable, lasting for months after the drug is discontinued.

When the researchers manipulated the animals so that their MEF2 levels remained high in the presence of cocaine, they turned out to be more sensitive to the drug. This suggested that an increase in the number of communication sites might help combat the addiction process.

“This suggests the exciting possibility that MEF2 proteins may control the expression of key genes that modulate drug-related brain changes and behaviour,” says Cowan. “If we understand which genes are influenced by MEF2, we can intervene and try to help the system resist or reverse these sensitisation processes.”

Sources: The Telegraph (Kolkata, India)

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Suppliments our body needs

Creatine

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Other Names: creatine monohydrate, creatine phosphate, creatine citrate

Definition:Creatine is nitrogenous organic acid that occurs naturally in vertebrates and helps to supply energy to muscle and nerve cells. Creatine was identified in 1832 when Michel Eugène Chevreul discovered it as a component of skeletal muscle, which he later named creatine after the Greek word for flesh, Kreas.

It is a compound that’s involved in the production of energy in the body, in the form of adenosine triphosphate (ATP). Made in the liver, approximately 95% of the body’s creatine ends up being stored in skeletal muscles and the remaining 5% is found in the brain, heart and testes. Once it’s used, creatine is converted to a waste product called creatinine and excreted in urine.

Functions:
Creatine, by way of conversion to and from phosphocreatine, functions in all vertebrates and some invertebrates, in conjunction with the enzyme creatine kinase. A similar system based on arginine/phosphoarginine operates in many invertebrates via the action of Arginine Kinase. The presence of this energy buffer system keeps the ATP/ADP ratio high at subcellular places where ATP is needed, which ensures that the free energy of ATP remains high and minimizes the loss of adenosine nucleotides, which would cause cellular dysfunction. Such high-energy phosphate buffers in the form of phosphocreatine or phosphoarginine are known as phosphagens. In addition, due to the presence of subcompartmentalized Creatine Kinase Isoforms at specific sites of the cell, the phosphocreatine/creatine kinase system also acts as an intracellular energy transport system from those places where ATP is generated (mitochondria and glycolysis) to those places where energy is needed and used, e.g., at the myofibrils for muscle contraction, at the sarcoplasmic reticulum (SR) for calcium pumping, and at the sites of many more biological processes that depend on ATP.

Biosynthesis:
In humans, about half of the daily creatine is biosynthesized from three different amino acids – arginine, glycine, and methionine. The rest is taken in by alimentary sources. Ninety-five percent of creatine is later stored in the skeletal muscles.

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The enzyme GATM (L-arginine:glycine amidinotransferase (AGAT), EC 2.1.4.1) is a mitochondrial enzyme responsible for catalyzing the first rate-limiting step of creatine biosynthesis, and is primarily expressed in the kidneys and pancreas.

The second enzyme in the pathway (GAMT, guanidinoacetate N-methyltransferase, EC:2.1.1.2) is primarily expressed in the liver and pancreas.

Genetic deficiencies in the creatine biosynthetic pathway lead to various severe neurological defects.

Controversy:While creatine’s effectiveness in the treatment of many muscular, neuromuscular, and neuro-degenerative diseases is documented, its utility as a performance-enhancing food supplement in sports has been questioned (see creatine supplements for more information). Some have even proposed that its use as a performance enhancer should be banned. Despite this, creatine remains very popular.

Sources:
In humans, approximately half of stored creatine originates from food (mainly from fresh meat). Since vegetables do not contain creatine, vegetarians show lower levels of muscle creatine which, upon creatine supplementation, rise to a level higher than in meat-eaters.

Creatine is found in small amounts in red meat and fish. However, much of it is destroyed by cooking. It’s also made naturally in the body from L-arginine, L-glycine and L-methionine, amino acids that are principally found in animal protein. Insulin is needed for creatine to enter muscles, so consuming carbohydrates with creatine may increase the amount of creatine available to muscles.

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Creatine supplements are available in capsules or as a powder at health food stores, some drug stores and online. One of the most popular forms of creatine is creatine monohydrate.

Side effects:-

In the Cochrane Collaboration analysis of 12 trials, there were no notable adverse events reported, however, research on the side effects and safety of creatine supplements is still limited.

Possible side effects of creatine include:

*Stomach cramps

*Nausea
*Diarrhea

*Loss of appetite

*Muscle cramps

*Weight gain

Creatine may cause water to be drawn away from other areas of the body and into muscle tissue, which could increase the risk of dehydration.

High doses of creatine could potentially injure the kidneys, liver and heart. Theoretically, creatine may cause kidney damage because its by-product, creatinine, is filtered through the kidneys into urine. Although studies haven’t found adverse events in recommended doses, there have been a couple of case reports of people who have experienced kidney collapse and three deaths in people taking creatine, but there is no definitive evidence that creatine was the cause. People with kidney disease or liver disease should avoid creatine.

Creatine supplements may cause asthmatic symptoms, such as wheezing and coughing, in some people.

People with McArdle’s disease shouldn’t use high doses of creatine because it has been found to increase muscle pain.

There is some concern that oral creatine supplements are metabolized in the body to a toxic waste product formaldehyde, which could potentially damage cells, DNA molecules and blood vessels.

Pregnant or nursing women or children should not use creatine supplements.

One of the main safety concerns is that individuals using creatine to enhance athletic performance or muscle mass, particularly adolescents, may exceed recommended dosages and take it without supervision.

Short-term use of creatine in healthy individuals is generally considered safe (see Creatine supplements#Safety). , studies have not yet been able to demonstrate either long-term or short term creatine supplementation result in adverse health effects. Creatine supplementation utilizing proper cycling and dosages has not been linked with any adverse side effects beyond occasional dehydration due to increased muscular water uptake from the rest of the body.[13] In fact, an increase in body mass because of increased muscle hydration is the most widely accepted side effect of creatine supplementation[14][15].

According to the opinion statement of the European Food Safety Authorities (EFSA) published in 2004 it was concluded that “The safety and bioavailability of the requested source of creatine, creatine monohydrate in foods for particular nutritional uses, is not a matter of concern provided that there is adequate control of the purity of this source of creatine (minimum 99.95%) with respect to dicyandiamide and dihydro-1,3,5-triazine derivatives, as well as heavy metal contamination. The EFSA Panel endorses the previous opinion of the SCF that high loading doses (20 gram / day) of creatine should be avoided. Provided high purity creatine monohydrate is used in foods for particular nutritional uses, the Panel considers that the consumption of doses of up to 3g/day of supplemental creatine, similar to the daily turnover rate of creatine, is unlikely to pose any risk”.

This opinion is corroborated by the fact that creatine is a natural component in mothers’ milk and that creatine is absolutely necessary for brain development in the human embryo and the baby, as well as for optimal physiological functioning of the adult human body, especially the brain, nervous system, the muscles and other organs and cells of high energy expenditure, where the creatine kinase (CK) system is highly expressed and creatine levels are high.

Side effects that produce lower leg pain may be associated with the use of creatine. Creatine may be the cause of an increase in the anterior pressures of the lower leg. This is usually found in post-creatine use when at rest and after exercise. Normal at-rest pressures have been found to be highly elevated by subjects who used creatine within the prior 35 days when compared to no supplementation. This can produce an extreme amount of pain in the lower leg due to the rigidity of the anterior compartment of the lower leg and lack of fluid drainage out of the compartment. It may also be exasperated by the increase of water content in the muscle fibers, putting more pressure on the anterior compartment. If this condition persist, check with your doctor and inform them of your creatine use and dosage. Although this condition may and usually does subside, if left untreated complications may occur that require emergency medical attention. If the levels remain high for a long period of time, irreversible damage to tissue may occur, particularly to the peroneal nerve. These conditions can further be found under Chronic Compartment Syndrome.

Creatine and the treatment of muscular diseases:
Creatine supplementation has been, and continues to be, investigated as a possible therapeutic approach for the treatment of muscular, neuromuscular, neurological and neurodegenerative diseases (arthritis, congestive heart failure, Parkinson’s disease, disuse atrophy, gyrate atrophy, McArdle’s disease, Huntington’s disease, miscellaneous neuromuscular diseases, mitochondrial diseases, muscular dystrophy, neuroprotection, etc.).

Two studies have indicated that creatine may be beneficial for neuromuscular disorders. First, a study demonstrated that creatine is twice as effective as the prescription drug riluzole in extending the lives of mice with the degenerative neural disease amyotrophic lateral sclerosis (ALS, or Lou Gehrig’s disease). The neuroprotective effects of creatine in the mouse model of ALS may be due either to an increased availability of energy to injured nerve cells or to a blocking of the chemical pathway that leads to cell death.

Second, creatine has been demonstrated to cause modest increases in strength in people with a variety of neuromuscular disorders.

Third, creatine has been shown to be beneficial as an adjuvant treatment for several neuro-muscular and neuro-degenerative diseases and its potential is just beginning to be explored in several multi-center clinical studies in the USA and elsewhere.

Resources:
http://en.wikipedia.org/wiki/Creatine
http://altmedicine.about.com/od/creatine/a/creatine.htm

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Herbs & Plants

Twinleaf

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Botanical Name : Jeffersonia diphylla
Family: Berberidaceae
Genus: Jeffersonia
Kingdom: Plantae
Order: Ranunculales

Synonyms :  J. binata. Podophyllum diphyllum.

 Common  Names: —Jeffersonia, rheumatism root, helmetpod, ground-squirrel pea, yellowroot.
Habitat: .—Jeffersonia diphylla is native to Eastern N. America – New York and Ontario to Alabama and west to Wisconsin.   It is very rare in the wild, it is found in limestone soils and rich woods near rivers.


Description.—Twinleaf    is herbaceous perennial plants and only about 6 or 8 inches in height when in flower but reaches a height of 18 inches at the fruiting stage. The long-stemmed, smooth leaves are almost completely divided into two leaflets and arise directly from the base of the plant. The white flowers measuring about 1 inch across. which appear early in spring, are borne singly on a slender stalk arising from the root and are followed by a leathery, somewhat pear-shaped capsule containing many seeds. Twinleaf has a thick, knotty, yellowish-brown, horizontal root. stock with man fibrous, much-matted roots.
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The leaves and flowers of this plant are smooth and emerge directly from the base of the plant. Jeffersonia has showy white flowers with eight petals; the flower resembles Bloodroot, a small poppy. The flower last only a short time after blooming in April or May, and gives way to a green pear-shaped capsule with a hinged top. The characteristic leaves are large andnearly divided in half, giving rise to its common name, Twinleaf.

They are uncommon spring wildflowers, which grow in limestone soils of rich woodland. Jeffersonia was named for United States President Thomas Jefferson, by his contemporary Benjamin Smith Barton. This genus was formerly, incorrectly grouped in genus Podophyllum. Twinleaf is protected by state laws as a threatened or endangered plant in Georgia, Iowa, New York, and New Jersey.

Cultivation :
Landscape Uses:Rock garden, Woodland garden. Prefers a light sandy, peaty or humus-rich woodland soil and a rather shady situation. Suitable for a choice position in a cool leafy soil. Plants are hardy to at least -20°c. A slow-growing plant. Plants have an extensive root system and resent disturbance. They should be pot-grown and planted into their permanent positions as soon as possible. Special Features:North American native, Attractive flowers or blooms.
Propagation :
Seed – best sown as soon as it is ripe in a cold frame. Sow stored seed as soon as possible in late winter or the spring. When they are large enough to handle, prick the seedlings out into individual pots and grow them on in the greenhouse for at least their first winter. Plant them out into their permanent positions in late spring or early summer, after the last expected frosts. Division in early spring

Medicinal Uses:  

Though Twinleaf ( Jeffersonia) is a poisonous plant, it has had a variety of medical uses throughout history. One of those uses is hinted at by an archaic common name of Jeffersonia diphylla, Rheumatism root. The “roots” of both species contain berberine, a known anti-tumor alkaloid.

The whole plant is antispasmodic, diuretic, emetic, expectorant and tonic. An infusion is used in the treatment of diarrhoea, dropsy, gravel and urinary problems. The root is emetic in large doses and expectorant in smaller doses. The root contains berberine, which has been shown to have anti-tumor activity. A poultice of the plant is applied to sores, ulcers and inflamed parts.The Cherokee used a tea for dropsy, urinary problems and on sores and inflammation.

Known Hazards: Twinleaf ( Jeffersonia) is a poisonous plant.

America
Native Americans utilized Jeffersonia diphylla for a variety of medicines. The Cherokee reportedly used an infusion of this plant for treating dropsy and urinary tract problems, it was also used as a poultice for sores and inflammation. The Iroquois used a decoction of the plant to treat liver problems and diarrhea.

The whole plant was used in early American medicine as an antispasmodic, diuretic, emetic, expectorant and general tonic. The “root” was once also used as an emetic in large doses, and as an expectorant in small doses. Modern medicine does not currently utilize this plant.

China
Traditional Chinese medicine uses Jeffersonia dubia for strengthening the stomach and bringing

Disclaimer:The information presented herein is intended for educational purposes only. Individual results may vary, and before using any supplements, it is always advisable to consult with your own health care provider

Resources:
http://www.mounet.com/~jdye/twinleaf.html
http://www.hort.purdue.edu/newcrop/herbhunters/twinleaf.html
http://en.wikipedia.org/wiki/Jeffersonia
http://plant.gardenbed.com/gardening.how/species/3435/plant/seed
http://2bnthewild.com/plants/H296.htm

http://www.pfaf.org/user/Plant.aspx?LatinName=Jeffersonia+diphylla

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News on Health & Science

Prostrate Cancer – Myths And Facts

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Your prostate is “the size and shape of a walnut”
Misleading, though constantly trotted out by charities. Actually, an apricot is a far better analogy for this organ that sits in the pelvis below the bladder. Like fruits, the prostate has an indentation at the top, through which passes the tube carrying urine from the bladder. That’s why an enlarged prostate means problems peeing.

Hardly. All that most people know about the prostate is that it gets cancer. We chaps should rejoice in it a little more. Not only does it produce the fluids that carry and nourish our sperm; not only is it a complex chemical factory; it’s also the male G-spot and produces many of our sexual kicks.

Men who get prostate cancer are the unlucky minority:
Fiction. It’s not often said because it sounds frightening, but getting prostate cancer could be said to be the norm rather than the exception as men get older. Over the past 30 years prostate cancer rates in Britain have tripled, but this is because of increased detection through PSA tests and the fact that we are living longer. Knowing that you have prostate cancer in your fifties is certainly bad, but for a man aged 85 it’s largely a reflection of the fact that he has been lucky enough not to die of something else earlier. Of the millions of middle-aged men who are never tested, research suggests that a third may have sluggish, relatively harmless prostate cancer and most will never know it.

You cannot reduce your risk of prostate cancer:

Hogwash. Though it’s your genes, your ethnic background and age that are the main risk factors, eating healthily seems to have a preventive role. The Prostate Cancer Charity says that cutting down on animal fat and eating more fruit and veg may lower your chances of prostate cancer. Recent research has indicated that broccoli, in particular, protects because it changes the way our genes express themselves. The jury remains out on supplements based on extracts of tomato, pomegranate and red clover.

Masturbation prevents prostate cancer:
Probably true, according to credible recent research, which found that men who ejaculated more than five times a week (solo, or with a partner) between the ages of 20 and 50 were a third less likely to develop prostate cancer later in life – probably because their orgasms were flushing out cancer-causing chemicals in their prostate. Sex with a partner, however, may pose a small risk of infection transmission, which could cancel the benefits

Sources:Times On Line

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Ailmemts & Remedies

Seborrhea Dermatitis

An infant with Cradle CapImage via Wikipedia

Definition: Seborrhea (say: seb-uh-ree-uh) is a common skin problem. It causes a red, itchy rash and white scales. When it affects the scalp, it is called “dandruff.” It can be on parts of the face as well, including the folds around the nose and behind the ears, the forehead, and the eyebrows and eyelids. On the body, seborrhea often occurs in the middle part of the chest, around the navel and in the skin folds under the arm, below the breasts and in the groin and buttocks area.

Seborrhoeic eczema (also Seborrheic dermatitis AmE, seborrhea) is a skin disorder affecting the scalp, face, and trunk causing scaly, flaky, itchy, red skin. It particularly affects the sebum-gland rich areas of skin.

click to see the pictures…..(01)...…(1)..……..(2).…..…(3)....………………….

Who gets seborrhea?
Infants may get seborrhea. It’s known as “cradle cap.” Cradle cap goes away after about 6 months. It may also affect the diaper area and look like a diaper rash.

Seborrhea also affects adults and elderly persons, and is more common in men than in women. Seborrhea occurs more frequently in persons with oily skin.

It affects 3 percent of the general population. It occurs more commonly in older people who are bedridden or have neurologic conditions such as Parkinson’s disease. Seborrhea also affects almost 85 percent of people with AIDS.

Causes:The cause of seborrheic dermatitis is not fully understood, although many factors have been implicated.. It is likely that a number of factors, such as hormones and stress, can cause it.
The widely present yeast, Malassezia furfur (formerly known as Pityrosporum ovale), is involved, as well as genetic, environmental, hormonal, and immune-system factors. A theory that seborrhoeic dermatitis is an inflammatory response to the yeast has not been proven. Those afflicted with seborrhoeic dermatitis have an unfavourable epidermic response to the infection, with the skin becoming inflamed and flaking.

Acute form of seborrhoeic dermatitis on scalpIn children, excessive vitamin A intake can cause seborrhoeic dermatitis. Lack of biotin, pyridoxine (vitamin B6) and riboflavin (vitamin B2) may also be a cause.

It is a chronic inflammatory skin disorder that affects the areas of the head and trunk that have sebaceous glands. A type of yeast that has an affinity for these glands called Pityrosporum ovale may be the cause, but this has not been proven yet. It is believed that the build-up of yeast in these glands irritates the skin causing redness and flaking.

Seborrhea is more common in men than women and affects 3 percent of the general population. It occurs more commonly in older people who are bedridden or have neurologic conditions such as Parkinson’s disease. Seborrhea also affects almost 85 percent of people with AIDS.

Diagnosis:

Clinical Manifestations
Seborrheic dermatitis typically affects areas of the skin where sebaceous glands appear in high frequency and are most active. The distribution is classically symmetric, and common sites of involvement are the hairy areas of the head, including the scalp , the scalp margin , eyebrows, eyelashes, mustache and beard. Other common sites are the forehead , the nasolabial folds , the external ear canals and the postauricular creases. Seborrhea of the trunk may appear in the presternal area and in the body folds, including the axillae, navel, groin, and in the inframammary and anogenital areas. Figure 7 illustrates the typically symmetric distribution of seborrheic dermatitis.

More severe seborrheic dermatitis is characterized by erythematous plaques frequently associated with powdery or greasy scale in the scalp (Figure 8), behind the ears (Figure 9) and elsewhere in the distribution described above. Besides an itchy scalp, patients may complain of a burning sensation in facial areas affected by seborrhea. Seborrhea frequently becomes apparent when men grow mustaches or beards and disappears when the facial hair is removed. If left untreated, the scale may become thick, yellow and greasy and, occasionally, secondary bacterial infection may occur.

Seborrheic dermatitis is more common in men than in women, probably because sebaceous gland activity is under androgen control. Seborrhea usually first appears in persons in their teens and twenties and generally follows a waxing/waning course throughout adulthood.

UV-A and UV-B light inhibit the growth of P. ovale,9 and many patients report improvement in seborrhea during summer.

Treatment:
Soaps and detergents such as sodium laureth sulfate may precipitate a flare-up, as they strip moisture from the top layers of the skin, and the drying property of these can cause flare-ups and may worsen the condition. Accordingly a suitable alternative should be used instead.

Among dermatologist recommended treatments are shampoos containing coal tar, ciclopiroxolamine, ketoconazole, selenium sulfide, or zinc pyrithione. For severe disease, keratolytics such as salicylic acid or coal tar preparations may be used to remove dense scale. Topical terbinafine solution (1%) has also been shown to be effective in the treatment of scalp seborrhoea, as may lotions containing alpha hydroxy acids or corticosteroids (such as fluocinolone acetonide). Pimecrolimus topical lotion is also sometimes prescribed.

Chronic treatment with topical corticosteroids may lead to permanent skin changes, such as atrophy and telangiectasia.

UV-A and UV-B light inhibit the growth of M. furfur, although caution should be taken to avoid sun damage.

According to the American Academy of Family Physicians(AAFP), one treatment that has proven successful, especially when steroid topicals and shampoos aren’t working, and the patient continues to suffer from rapid hair loss and rashes, has been low doses(10mg-30mg daily) of the perscription drug Accutane,(Isotretinoin). The exact mechanism isn’t known, but it is thought to work by reducing sebum, which plays an important role in seborrhoeic dermatitis. Patients should be evaluated monthly, while examing the proper liver functions when putting a patient on accutane therapy. Special screening should be in place for women patients, because of the risk of birth defects. This therapy can last, when the condition is chronic and the isotretinoin does is low, for years. But, patients should be given a one to two month break off this particular therapy every 6 months to see if the condition still is affecting the patient

Adults who have seborrhea usually experience a waxing and waning course. In other words it can’t be “cured”. The good news is with proper maintenance, seborrhea can be controlled. Furthermore, most of the treatments can be found over-the-counter.

Treatment will help keep seborrhea under control. It’s important to keep your body clean.

Dandruff Shampoo
If you have dandruff, use medicated shampoos.

When using dandruff shampoo, first wet your hair. Rub some shampoo into your scalp and hair. Leave the shampoo on your scalp and hair for at least 5 minutes. Then rinse it out. Use the dandruff shampoo every day until your dandruff goes away. Then use the medicated shampoo 2 or 3 times a week to keep dandruff away. Having dandruff does not mean that your scalp is too dry! Dandruff comes because you need to wash your hair more often.

Medicated Shampoos should always be used.For black persons, daily shampooing may not be needed. Ask your doctor about a special steroid preparation in oil that can be used on the scalp like a pomade. Or you can use a steroid-containing shampoo.

Adults who have seborrhea usually experience a waxing and waning course. In other words it can’t be “cured”. The good news is with proper maintenance, seborrhea can be controlled. Furthermore, most of the treatments can be found over-the-counter.

Proper hygiene plays an important role in treatment. Frequent washing with soap gets rid of the oils in the affected areas and improves symptoms. Sunlight inhibits the growth of the yeast; therefore exposure of affected areas to sun is helpful, although caution should be exercised to avoid sun damage. The main medical treatments are antifungal shampoos and topical.

Cradle Cap:
Cradle cap in infants also gets better with daily shampooing. First try a mild, nonmedicated baby shampoo. If that doesn’t work, try an a dandruff shampoo. If the patch of cradle cap is large and thick, first try softening it by rubbing on warm mineral oil. Next, gently brush with a baby hairbrush. Then use shampoo.

Seborrhea Shampoos
There are several good antifungal shampoos on the market that can be purchased without a prescription. The main shampoos are selenium sulfide found in Selsun, pyrithione zinc found inHead & Shouldersulders and Sebulon, coal tar found in Sebutone and Tegrin, and finally ketoconazole found in Nizoral.

All of these shampoos have a medicated smell. The way to use them is to shampoo and leave on for at least 10 minutes then rinse off. The shampoos can be used on the face and other parts of the body as a lotion with the same instructions as long as precaution is used around the eyes. Do this daily until the redness and flaking is controlled then use 2-3 times a week as needed to keep symptoms from returning.

Topical Steroids For Seborrhea
Topical steroids reduce the inflammatory response and help control itching. You can buy hydrocortisone cream 1% over-the-counter, and it’s safe to use on the face. Apply twice a day to the affected area until the redness resolves. Save the hydrocortisone for flare-ups and use the antifungal shampoo for maintenance because long-term steroid use can cause side effects like acne and thinning of the skin.

Herbal Treatment:The World Health Organization mentions Aloe vera gel as a yet to be scientifically proven traditional medicine treatment for Seborrhoeic dermatitis.

*Arctium lappa (Burdock) oil
*Chelidonium majus (Celandine)
*Glycyrrhiza glabra (Licorice)
*Melaleuca (Tea tree) species
*Plantago (Plantain) species
*Symphytum officinale (Comfrey)
*Zingiber officinale (Ginger) root juice
*Ledebouriella Seseloides (Fang Feng)
*Smilax China (Smilax china)
*Trichosanthes Kirilowii (Snakegourd)
*Glycyrrhiza Uralensis
*Coptis Chinensis (Chinese goldthread)
*Phellodendron Amurense (Huang Bai)
*Sophora Flavescens
*Centella Asiatica (Gotu Kola)
*Evening primrose,
*dandelion root
*red clover Norwegian kelp
* berberine (from barberry, Oregon grape root or goldenseal).

Quik Tip: Evening primrose – anti-inflammatory herb of the first magnitude; it helps your

body balance itself hormonally, too.

Click to learn more about Seborrheic Dermatitis

 

Disclaimer: This information is not meant to be a substitute for professional medical advise or help. It is always best to consult with a Physician about serious health concerns. This information is in no way intended to diagnose or prescribe remedies.This is purely for educational purpose.

Resources:
http://en.wikipedia.org/wiki/Seborrheic_dermatitis
http://www.herbnews.org/seborrheadone.htm
http://dermatology.about.com/cs/seborrhea/a/sebderm.htm
http://www.aafp.org/afp/20000501/2703.html

http://en.wikipedia.org/wiki/Taraxacum

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